Matthew E. Cockman
Impact in
- Cancer Research top 0.1%
- Cancer, Hypoxia, and Metabolism
- Molecular Biology top 1%
- RNA modifications and cancer
- Epigenetics and DNA Methylation
- Mitochondrial Function and Pathology
- Ubiquitin and proteasome pathways
Papers in
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- Cancer, Hypoxia, and Metabolism 21
-
- Epigenetics and DNA Methylation 7
- RNA modifications and cancer 7
- Fibroblast Growth Factor Research 4
- Mitochondrial Function and Pathology 4
- Ubiquitin and proteasome pathways 4
- Co-authors
- Peter J. RatcliffeChristopher W. PughPatrick H. MaxwellGin-Wen ChangEamonn R. MaherSteven C. CliffordCharles C. WykoffMichael S. Wiesener
- Journals
- Journal of Biological Chemistry (7 papers)Proceedings of the National Academy of Sciences (5 papers)Oncogene (2 papers)Chemistry - A European Journal (1 paper)British Journal of Cancer (1 paper)
- Partner nations
- United KingdomUnited StatesGermany
In The Last Decade
Matthew E. Cockman
34 papers receiving 7.0k citations
Hit Papers
Peers
Comparison fields: 5 of 127
- Cancer Research 4.8k
- Molecular Biology 4.8k
- Biochemistry 369
- Cell Biology 602
- Genetics 987
Countries citing papers authored by Matthew E. Cockman
This map shows the geographic impact of Matthew E. Cockman's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Matthew E. Cockman with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Matthew E. Cockman more than expected).
Fields of papers citing papers by Matthew E. Cockman
This network shows the impact of papers produced by Matthew E. Cockman. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Matthew E. Cockman. The network helps show where Matthew E. Cockman may publish in the future.
Co-authors
The 25 scholars most cited alongside Matthew E. Cockman, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2025 | 0 | |
| 2 | 2022 | 23 | |
| 3 | 2021 | 5 | |
| 4 | 2019 | 75 | |
| 5 | 2018 | 140 | |
| 6 | 2017 | 62 | |
| 7 | 2013 | 206 | |
| 8 | 2012 | 45 | |
| 9 | 2011 | 66 | |
| 10 | 2011 | 20 | |
| 11 | 2009 | 69 | |
| 12 | 2007 | 75 | |
| 13 | 2007 | 180 | |
| 14 | 2007 | 56 | |
| 15 | 2004 | 85 | |
| 16 | 2001 | 32 | |
| 17 | Hypoxia Inducible Factor-α Binding and Ubiquitylation by the von Hippel-Lindau Tumor Suppressor Protein Hit paper breakdown → | 2000 | 893 |
| 18 | The tumour suppressor protein VHL targets hypoxia-inducible factors for oxygen-dependent proteolysis Hit paper breakdown → | 1999 | 4162 |
| 19 | 1999 | 34 | |
| 20 | 1998 | 1 |
About Matthew E. Cockman
Matthew E. Cockman is a scholar working on Cancer Research, Molecular Biology, Cell Biology, Genetics and Physiology, having authored 35 papers that have together received 7.1k indexed citations. Recurring topics across this work include Cancer, Hypoxia, and Metabolism (21 papers), Epigenetics and DNA Methylation (7 papers), RNA modifications and cancer (7 papers), High Altitude and Hypoxia (5 papers), Fibroblast Growth Factor Research (4 papers), Mitochondrial Function and Pathology (4 papers), Endoplasmic Reticulum Stress and Disease (4 papers) and Ubiquitin and proteasome pathways (4 papers). The work is most often cited by research in Cancer Research (4.8k citations), Molecular Biology (4.8k citations), Biochemistry (369 citations), Cell Biology (602 citations) and Genetics (987 citations). Matthew E. Cockman has collaborated with scholars based in United Kingdom, United States and Germany. Frequent co-authors include Peter J. Ratcliffe, Christopher W. Pugh, Patrick H. Maxwell, Gin-Wen Chang, Eamonn R. Maher, Steven C. Clifford, Charles C. Wykoff, Michael S. Wiesener, Norma Masson and David R. Mole. Their work appears in journals such as Journal of Biological Chemistry, Proceedings of the National Academy of Sciences, Oncogene, Chemistry - A European Journal and British Journal of Cancer.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.