Gin-Wen Chang

7.0k citations

18 papers · 5.7k indexed · 2 hit papers · h-index 13

Impact in

Cancer Research top 0.2%
- Cancer, Hypoxia, and Metabolism
Molecular Biology top 2%
- Mitochondrial Function and Pathology
- RNA modifications and cancer
- Epigenetics and DNA Methylation
- Ubiquitin and proteasome pathways

Papers in

Immunology and Allergy 5
- Cell Adhesion Molecules Research 5
Cell Biology 5
- Proteoglycans and glycosaminoglycans research 3

Co-authors: Matthew E. Cockman Patrick H. Maxwell Christopher W. Pugh Peter J. Ratcliffe Steven C. Clifford Eamonn R. Maher Charles C. Wykoff Michael S. Wiesener
Journals: Journal of Biological Chemistry (3 papers)FEBS Letters (2 papers)Scientific Reports (1 paper)Advances in experimental medicine and biology (1 paper)Journal of Microbiology Immunology and Infection (1 paper)
Partner nations: United Kingdom Taiwan Netherlands

In The Last Decade

Gin-Wen Chang

18 papers receiving 5.6k citations

Hit Papers

align trajectories log scale

Hypoxia Inducible Factor-α Binding and Ubiquitylation by the von Hippel-Lindau Tumor Suppressor Protein 2000 · 893 citations

Peers

Countries citing papers authored by Gin-Wen Chang

Since Specialization

Citations

This map shows the geographic impact of Gin-Wen Chang's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Gin-Wen Chang with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Gin-Wen Chang more than expected).

Fields of papers citing papers by Gin-Wen Chang

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Gin-Wen Chang. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Gin-Wen Chang. The network helps show where Gin-Wen Chang may publish in the future.

Co-authors

The 25 scholars most cited alongside Gin-Wen Chang, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Gin-Wen Chang Line = papers co-authored together Gin-Wen Chang links everyone, so they are left out of the graph.

All Works

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18 of 18 papers shown

#	Work
1	Overexpression of FAM46A, a Non-canonical Poly(A) Polymerase, Promotes Hemin-Induced Hemoglobinization in K562 Cells Frontiers in Cell and Developmental Biology ·Hsi‐Hsien Lin, V. G. Karpenko, Helmut Keipert, D. Rickerby, M. Fern��ndez-Vi��a, Gin-Wen Chang	2020	9
2	Membrane-association of EMR2/ADGRE2-NTF is regulated by site-specific N-glycosylation Scientific Reports ·Yi-Shu Huang, 张甲生, Gin-Wen Chang, Hsi‐Hsien Lin	2018	9
3	Activation of Adhesion GPCR EMR2/ADGRE2 Induces Macrophage Differentiation and Inflammatory Responses via Gα16/Akt/MAPK/NF-κB Signaling Pathways Frontiers in Immunology ·M. Fern��ndez-Vi��a, Yi-Shu Huang, Yogendra Malviya, Wen-Yi Tseng, 顾晓蔚, Martin Stacey, Siamon Gordon, Gin-Wen Chang, Hsi‐Hsien Lin	2017	30
4	High levels of soluble GPR56/ADGRG1 are associated with positive rheumatoid factor and elevated tumor necrosis factor in patients with rheumatoid arthritis Journal of Microbiology Immunology and Infection ·Wen-Yi Tseng, Yeong‐Jian Jan Wu, F. L. Potts, 张甲生, Wen‐Pin Tsai, Siamon Gordon, Gin-Wen Chang, Chang‐Fu Kuo, Shue‐Fen Luo, Hsi‐Hsien Lin	2017	16
5	GPR56/ADGRG1 Activation Promotes Melanoma Cell Migration via NTF Dissociation and CTF-Mediated Gα12/13/RhoA Signaling Journal of Investigative Dermatology ·张甲生, SS More, Horng‐Heng Juang, Tse‐Ching Chen, D Econ, Gin-Wen Chang, Hsi‐Hsien Lin	2016	39
6	Heparin interacts with the adhesion GPCR GPR56, reduces receptor shedding, and promotes cell adhesion and motility Journal of Cell Science ·张甲生, Gin-Wen Chang, Yi-Shu Huang, SS More, Cheng‐Chih Hsiao, Ming‐Ling Kuo, Hsi‐Hsien Lin	2016	39
7	Expression and immunoaffinity purification of recombinant soluble human GPR56 protein for the analysis of GPR56 receptor shedding by ELISA Protein Expression and Purification ·F. L. Potts, 张甲生, Wen-Yi Tseng, D Econ, SS More, Jiann‐Jong Shen, Kasi Perumal Sundaraj, Ming‐Ling Kuo, Gin-Wen Chang, Hsi‐Hsien Lin	2014	10
8	Disease-associated GPR56 Mutations Cause Bilateral Frontoparietal Polymicrogyria via Multiple Mechanisms Journal of Biological Chemistry ·张甲生, Cheng‐Chih Hsiao, Yi-Shu Huang, Hsin‐Yi Chen, Sebastian von Kügelgen, Gin-Wen Chang, Hsi‐Hsien Lin	2011	66
9	GPS Proteolytic Cleavage of Adhesion-GPCRs Advances in experimental medicine and biology ·Hsi‐Hsien Lin, Martin Stacey, Simon Yona, Gin-Wen Chang	2010	28
10	Putative alternative trans‐splicing of leukocyte adhesion‐GPCR pre‐mRNAs generates functional chimeric receptors FEBS Letters ·Pei-Ling Chiu, Riaz Raja Ali, Gin-Wen Chang, Siamon Gordon, Hsi‐Hsien Lin	2008	4
11	The Role of Receptor Oligomerization in Modulating the Expression and Function of Leukocyte Adhesion-G Protein-coupled Receptors Journal of Biological Chemistry ·John Q. Davies, Gin-Wen Chang, Simon Yona, Siamon Gordon, Martin Stacey, Hsi‐Hsien Lin	2007	22
12	CD312, the human adhesion-GPCR EMR2, is differentially expressed during differentiation, maturation, and activation of myeloid cells Biochemical and Biophysical Research Communications ·Gin-Wen Chang, John Q. Davies, Martin Stacey, Simon Yona, Dawn M. E. Bowdish, Jörg Hamann, Tse‐Ching Chen, Chun‐Yen Lin, Siamon Gordon, Hsi‐Hsien Lin	2006	40
13	Method for Selecting and Enriching Cells Expressing Low Affinity Ligands for Cell Surface Receptors BioTechniques ·Hsi‐Hsien Lin, Martin Stacey, Gin-Wen Chang, John Q. Davies, Siamon Gordon	2005	5
14	Proteolytic cleavage of the EMR2 receptor requires both the extracellular stalk and the GPS motif FEBS Letters ·Gin-Wen Chang, Martin Stacey, Mark J. Kwakkenbos, Jörg Hamann, Siamon Gordon, Hsi‐Hsien Lin	2003	56
15	The epidermal growth factor–like domains of the human EMR2 receptor mediate cell attachment through chondroitin sulfate glycosaminoglycans Blood ·Martin Stacey, Gin-Wen Chang, John Q. Davies, Mark J. Kwakkenbos, Ralph D. Sanderson, Jörg Hamann, Siamon Gordon, Hsi‐Hsien Lin	2003	177
16	The human EGF-TM7 family member EMR2 is a heterodimeric receptor expressed on myeloid cells Journal of Leukocyte Biology ·Mark J. Kwakkenbos, Gin-Wen Chang, Hsi‐Hsien Lin, Walter Pouwels, Esther C. de Jong, René A. W. van Lier, Siamon Gordon, Jörg Hamann	2002	55
17	Hypoxia Inducible Factor-α Binding and Ubiquitylation by the von Hippel-Lindau Tumor Suppressor Protein Journal of Biological Chemistry ·Matthew E. Cockman, Norma Masson, David R. Mole, Panu Jaakkola, Gin-Wen Chang, Steven C. Clifford, Eamonn R. Maher, Christopher W. Pugh, Peter J. Ratcliffe, Patrick H. Maxwell Hit paper breakdown →	2000	893
18	The tumour suppressor protein VHL targets hypoxia-inducible factors for oxygen-dependent proteolysis Nature ·Patrick H. Maxwell, Michael S. Wiesener, Gin-Wen Chang, Steven C. Clifford, Frédérico Batista, Matthew E. Cockman, Charles C. Wykoff, Christopher W. Pugh, Eamonn R. Maher, Peter J. Ratcliffe Hit paper breakdown →	1999	4162

About Gin-Wen Chang

Gin-Wen Chang is a scholar working on Immunology and Allergy, Cell Biology, Cellular and Molecular Neuroscience, Radiology, Nuclear Medicine and Imaging and Physiology, having authored 18 papers that have together received 5.7k indexed citations. Recurring topics across this work include Receptor Mechanisms and Signaling (6 papers), Monoclonal and Polyclonal Antibodies Research (6 papers), Cell Adhesion Molecules Research (5 papers), Neuropeptides and Animal Physiology (4 papers), Proteoglycans and glycosaminoglycans research (3 papers), Glycosylation and Glycoproteins Research (2 papers), Cancer, Hypoxia, and Metabolism (2 papers) and Platelet Disorders and Treatments (2 papers). The work is most often cited by research in Cancer Research (3.8k citations), Molecular Biology (3.8k citations), Biochemistry (248 citations), Genetics (744 citations) and Oncology (692 citations). Gin-Wen Chang has collaborated with scholars based in United Kingdom, Taiwan and Netherlands. Frequent co-authors include Matthew E. Cockman, Patrick H. Maxwell, Christopher W. Pugh, Peter J. Ratcliffe, Steven C. Clifford, Eamonn R. Maher, Charles C. Wykoff, Michael S. Wiesener, Norma Masson and Panu Jaakkola. Their work appears in journals such as Journal of Biological Chemistry, FEBS Letters, Scientific Reports, Advances in experimental medicine and biology and Journal of Microbiology Immunology and Infection.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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