John C. Reed

134.3k citations
798 papers · 107.7k · 36 hit papers · h-index 161

Impact in

    • Cell death mechanisms and regulation
    • Mitochondrial Function and Pathology
    • RNA Interference and Gene Delivery
    • Ubiquitin and proteasome pathways
  • Immunology top 0.01%

Papers in

    • Cell death mechanisms and regulation 350
    • RNA Interference and Gene Delivery 79
    • Ubiquitin and proteasome pathways 54
    • interferon and immune responses 51
    • Immune Response and Inflammation 48

John C. Reed

786 papers receiving 105.7k citations

John C. Reed's Hit Papers

ER stress-induced cell death mechanisms 2013 · 1.6k citations
1.6k0+9+18Years since publication50010001.5k

Peers

John C. Reed
Comparison fields: 5 of 202
  • Molecular Biology 69.2k
  • Immunology 19.1k
  • Cancer Research 11.0k
  • Oncology 18.9k
  • Cell Biology 9.5k
Replace Stanley J. Korsmeyer with:
Stanley J. Korsmeyer United States
Douglas R. Green United States
Roger J. Davis United States
Tak W. Mak Canada
Pierre Chambon France
Lewis C. Cantley United States
Douglas Hanahan United States
Tony Hunter United States
Ronald M. Evans United States
Craig B. Thompson United States
John C. Reed relative to Stanley J. Korsmeyer United States Stanley J. Korsmeyer's profile →
Citations per field
00.5×1.7×
Stanley J. Korsmeyer · 1×
Citations per year

Countries citing papers authored by John C. Reed

Since Specialization
Citations

This map shows the geographic impact of John C. Reed's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by John C. Reed with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites John C. Reed more than expected).

Fields of papers citing papers by John C. Reed

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by John C. Reed. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by John C. Reed. The network helps show where John C. Reed may publish in the future.

Co-authors

The 25 scholars most cited alongside John C. Reed, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with John C. Reed Line = papers co-authored together John C. Reed links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 798 papers — load more, or switch the sort, to bring in the rest.

#Work
1
Mitochondria and Apoptosis
Hit paper breakdown →
19987887
2
Regulation of Cell Death Protease Caspase-9 by Phosphorylation
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19982620
3
IAP family proteins---suppressors of apoptosis
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19992150
4
Tumor suppressor p53 is a regulator of bcl-2 and bax gene expression in vitro and in vivo.
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19941882
5
Ordering the Cytochrome c–initiated Caspase Cascade: Hierarchical Activation of Caspases-2, -3, -6, -7, -8, and -10 in a Caspase-9–dependent Manner
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19991651
6
ER stress-induced cell death mechanisms
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20131627
7
Cell death and endoplasmic reticulum stress: disease relevance and therapeutic opportunities
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20081541
8
Apoptosis in the Failing Human Heart
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19971354
9
Bax directly induces release of cytochrome c from isolated mitochondria
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19981350
10
Somatic Frameshift Mutations in the BAX Gene in Colon Cancers of the Microsatellite Mutator Phenotype
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19971072
11
Bax and Adenine Nucleotide Translocator Cooperate in the Mitochondrial Control of Apoptosis
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19981011
12
Ca 2+ -Induced Apoptosis Through Calcineurin Dephosphorylation of BAD
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1999972
13
Investigation of the subcellular distribution of the bcl-2 oncoprotein: residence in the nuclear envelope, endoplasmic reticulum, and outer mitochondrial membranes.
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1993911
14
Anchorage dependence, integrins, and apoptosis
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1994866
15
Dysregulation of Apoptosis in Cancer
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1999806
16
Cloning and functional analysis of BAG-1: A novel Bcl-2-binding protein with anti-cell death activity
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1995747
17
Bcl-2 family proteins and cancer
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2008723
18
Cytochrome c: Can't Live with It—Can't Live without It
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1997660
19
Pro-caspase-3 Is a Major Physiologic Target of Caspase-8
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1998650
20
Changes in intramitochondrial and cytosolic pH: early events that modulate caspase activation during apoptosis
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2000646

About John C. Reed

John C. Reed is a scholar working on Molecular Biology, Immunology, Oncology, Cancer Research and Cell Biology, having authored 798 papers that have together received 107.7k indexed citations. Recurring topics across this work include Cell death mechanisms and regulation (350 papers), Cancer-related Molecular Pathways (100 papers), RNA Interference and Gene Delivery (79 papers), NF-κB Signaling Pathways (66 papers), Ubiquitin and proteasome pathways (54 papers), Chronic Lymphocytic Leukemia Research (54 papers), interferon and immune responses (51 papers) and Immune Response and Inflammation (48 papers). The work is most often cited by research in Molecular Biology (69.2k citations), Immunology (19.1k citations), Cancer Research (11.0k citations), Oncology (18.9k citations) and Cell Biology (9.5k citations). John C. Reed has collaborated with scholars based in United States, Germany and Canada. Frequent co-authors include Douglas R. Green, Stanisław Krajewski, Quinn L. Deveraux, Shinichi Kitada, Maryla Krajewska, Shinichi Takayama, Hong‐Gang Wang, Zhihua Xie, Renata Sano and Guy S. Salvesen. Their work appears in journals such as Journal of Biological Chemistry, Blood, Proceedings of the National Academy of Sciences, The Journal of Immunology and Oncogene.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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