Guido Kroemer
- Molecular Biology top 0.01%
- Immunology top 0.01%
- Epidemiology top 0.01%
- Oncology top 0.01%
- Cancer Research top 0.01%
- Co-authors
- Lorenzo GalluzziLaurence ZitvogelBeth LevineOliver KeppNaoufal ZamzamiSantos A. SusínDaolin TangCarlos López-Otı́n
- Topics
- Autophagy in Disease and Therapy (285 papers)Cell death mechanisms and regulation (270 papers)Immunotherapy and Immune Responses (179 papers)
- Cited by
- AgingImmunologyCancer Research
- Partner nations
- FranceSwedenUnited States
In The Last Decade
Guido Kroemer
1.3k papers receiving 232.6k citations
Hit Papers
Peers
Comparison fields: 5 of 213
- Molecular Biology 128.2k
- Immunology 51.4k
- Epidemiology 47.5k
- Oncology 45.9k
- Cancer Research 28.9k
Countries citing papers authored by Guido Kroemer
This map shows the geographic impact of Guido Kroemer's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Guido Kroemer with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Guido Kroemer more than expected).
Fields of papers citing papers by Guido Kroemer
This network shows the impact of papers produced by Guido Kroemer. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Guido Kroemer. The network helps show where Guido Kroemer may publish in the future.
Co-authorship network of co-authors of Guido Kroemer
This figure shows the co-authorship network connecting the top 25 collaborators of Guido Kroemer. A scholar is included among the top collaborators of Guido Kroemer based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Guido Kroemer. Guido Kroemer is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 2 | |
| 2 | Immunogenic cell death in cancer: concept and therapeutic implicationsbreakdown → | 94 |
| 3 | 23 | |
| 4 | 26 | |
| 5 | 26 | |
| 6 | 100 | |
| 7 | 101 | |
| 8 | 108 | |
| 9 | 77 | |
| 10 | 302 | |
| 11 | 124 | |
| 12 | 286 | |
| 13 | 23 | |
| 14 | 239 | |
| 15 | 87 | |
| 16 | 23 | |
| 17 | 109 | |
| 18 | 80 | |
| 19 | Tumor cells convert immature myeloid dendritic cells into TGF-β–secreting cells inducing CD4 + CD25 + regulatory T cell proliferationbreakdown → | 588 |
| 20 | 80 |
About Guido Kroemer
Guido Kroemer is a scholar working on Immunology, Physiology and Aging, having authored 1.4k papers that have together received 235.5k indexed citations. Recurring topics across this work include Autophagy in Disease and Therapy (285 papers), Cell death mechanisms and regulation (270 papers) and Immunotherapy and Immune Responses (179 papers). The work is most often cited by research in Aging (5.9k citations), Immunology (51.4k citations) and Cancer Research (28.9k citations). Guido Kroemer has collaborated with scholars based in France, Sweden and United States. Frequent co-authors include Lorenzo Galluzzi, Laurence Zitvogel, Beth Levine, Oliver Kepp, Naoufal Zamzami, Santos A. Susín, Daolin Tang, Carlos López-Otı́n, Douglas R. Green and Rui Kang. Their work appears in journals such as Nature, Science and Cell.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.