Lewis C. Cantley
- Cancer Research top 0.01%
- Cancer, Hypoxia, and Metabolism 79
- Molecular Biology top 0.01%
- PI3K/AKT/mTOR signaling in cancer 148
- Protein Kinase Regulation and GTPase Signaling 132
- Metabolism, Diabetes, and Cancer 77
- Ubiquitin and proteasome pathways 32
- Cell Biology top 0.01%
- Cellular transport and secretion 65
- Oncology top 0.01%
- Genetics top 0.02%
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- Pancreatic function and diabetes 57
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- Monoclonal and Polyclonal Antibodies Research 33
- Co-authors
- Matthew G. Vander HeidenCraig B. ThompsonBrendan D. ManningJi LuoJeffrey A. EngelmanReuben J. ShawAlex TokerZhou Songyang
- Partner nations
- United StatesIsraelGermany
In The Last Decade
Lewis C. Cantley
696 papers receiving 147.7k citations
Hit Papers
Peers
Comparison fields: 5 of 200
- Cancer Research 29.7k
- Molecular Biology 104.9k
- Cell Biology 19.6k
- Oncology 27.4k
- Genetics 8.6k
Countries citing papers authored by Lewis C. Cantley
This map shows the geographic impact of Lewis C. Cantley's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Lewis C. Cantley with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Lewis C. Cantley more than expected).
Fields of papers citing papers by Lewis C. Cantley
This network shows the impact of papers produced by Lewis C. Cantley. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Lewis C. Cantley. The network helps show where Lewis C. Cantley may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Lewis C. Cantley, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2015 | 307 | |
| 2 | 2015 | 139 | |
| 3 | Vitamin C selectively kills KRAS and BRAF mutant colorectal cancer cells by targeting GAPDHbreakdown → | 2015 | 696 |
| 4 | 2014 | 136 | |
| 5 | 2013 | 45 | |
| 6 | 2013 | 45 | |
| 7 | 2013 | 7 | |
| 8 | Influence of Threonine Metabolism on S -Adenosylmethionine and Histone Methylationbreakdown → | 2012 | 506 |
| 9 | 2012 | 336 | |
| 10 | 2011 | 9 | |
| 11 | 2010 | 125 | |
| 12 | 2009 | 75 | |
| 13 | 2009 | 1 | |
| 14 | MET Amplification Leads to Gefitinib Resistance in Lung Cancer by Activating ERBB3 Signalingbreakdown → | 2007 | 3570 |
| 15 | 2007 | 216 | |
| 16 | 2004 | 77 | |
| 17 | The tumor suppressor LKB1 kinase directly activates AMP-activated kinase and regulates apoptosis in response to energy stressbreakdown → | 2004 | 1484 |
| 18 | Large-scale characterization of HeLa cell nuclear phosphoproteinsbreakdown → | 2004 | 1190 |
| 19 | 2003 | 129 | |
| 20 | Tuberous sclerosis complex-1 and -2 gene products function together to inhibit mammalian target of rapamycin (mTOR)-mediated downstream signalingbreakdown → | 2002 | 647 |
About Lewis C. Cantley
Lewis C. Cantley is a scholar working on Molecular Biology, Cancer Research and Cell Biology, having authored 710 papers that have together received 150.4k indexed citations. Recurring topics across this work include PI3K/AKT/mTOR signaling in cancer (148 papers), Protein Kinase Regulation and GTPase Signaling (132 papers), Cancer, Hypoxia, and Metabolism (79 papers), Metabolism, Diabetes, and Cancer (77 papers), Cellular transport and secretion (65 papers), Pancreatic function and diabetes (57 papers), Monoclonal and Polyclonal Antibodies Research (33 papers) and Ubiquitin and proteasome pathways (32 papers). The work is most often cited by research in Cancer Research (29.7k citations), Molecular Biology (104.9k citations) and Cell Biology (19.6k citations). Lewis C. Cantley has collaborated with scholars based in United States, Israel and Germany. Frequent co-authors include Matthew G. Vander Heiden, Craig B. Thompson, Brendan D. Manning, Ji Luo, Jeffrey A. Engelman, Reuben J. Shaw, Alex Toker, Zhou Songyang, John M. Asara and Stephen P. Soltoff. Their work appears in journals such as Nature, Science and New England Journal of Medicine.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.