James K. V. Willson

36.9k citations
175 papers · 22.6k · 10 hit papers · h-index 64

Impact in

Papers in

    • TGF-β signaling in diseases 23
    • DNA Repair Mechanisms 15
    • Cancer therapeutics and mechanisms 14
    • Colorectal Cancer Treatments and Studies 26
    • Cancer-related Molecular Pathways 12

James K. V. Willson

173 papers receiving 22.1k citations

James K. V. Willson's Hit Papers

Glucose Deprivation Contributes to the Development of KRAS Pathway Mutations in Tumor Cells 2009 · 724 citations
7240+12+24Years since publication50010001.5k2.0k2.5k

Peers

James K. V. Willson
Comparison fields: 5 of 163
  • Pathology and Forensic Medicine 7.0k
  • Cancer Research 5.8k
  • Oncology 9.8k
  • Molecular Biology 14.0k
  • Cell Biology 1.8k
Replace Sanford D. Markowitz with:
Sanford D. Markowitz United States
Christoph Lengauer United States
Scott E. Kern United States
Ayşegül A. Şahin United States
Julie Teruya‐Feldstein United States
Kathleen R. Cho United States
Neal Rosen United States
Michael J. Birrer United States
Joseph R. Testa United States
Levi A. Garraway United States
James K. V. Willson relative to Sanford D. Markowitz United States Sanford D. Markowitz's profile →
Citations per field
00.5×1.5×
Sanford D. Markowitz · 1×
Citations per year

Countries citing papers authored by James K. V. Willson

Since Specialization
Citations

This map shows the geographic impact of James K. V. Willson's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by James K. V. Willson with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites James K. V. Willson more than expected).

Fields of papers citing papers by James K. V. Willson

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by James K. V. Willson. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by James K. V. Willson. The network helps show where James K. V. Willson may publish in the future.

Co-authors

The 25 scholars most cited alongside James K. V. Willson, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with James K. V. Willson Line = papers co-authored together James K. V. Willson links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 175 papers — load more, or switch the sort, to bring in the rest.

#Work
1
High Frequency of Mutations of the PIK3CA Gene in Human Cancers
Hit paper breakdown →
20042708
2
Suppression of Human Colorectal Carcinoma Cell Growth by Wild-Type p53
Hit paper breakdown →
19901577
3
Incidence and functional consequences of hMLH1 promoter hypermethylation in colorectal carcinoma
Hit paper breakdown →
19981538
4
Mutations of mitotic checkpoint genes in human cancers
Hit paper breakdown →
19981198
5
p53 gene mutations occur in combination with 17p allelic deletions as late events in colorectal tumorigenesis.
Hit paper breakdown →
1990906
6
Glucose Deprivation Contributes to the Development of KRAS Pathway Mutations in Tumor Cells
Hit paper breakdown →
2009724
7
Somatic mutations of the mitochondrial genome in human colorectal tumours
Hit paper breakdown →
1998689
8
Mismatch repair gene defects in sporadic colorectal cancers with microsatellite instability
Hit paper breakdown →
1995678
9
Microsatellite instability and mutations of the transforming growth factor beta type II receptor gene in colorectal cancer.
Hit paper breakdown →
1995628
10
Evaluation of candidate tumour suppressor genes on chromosome 18 in colorectal cancers
Hit paper breakdown →
1996501
11 2005443
12 2004412
13 1995406
14 2003370
15 1993369
16
Deletion of p16 and p15 genes in brain tumors.
1994342
17
Mutational inactivation of transforming growth factor beta receptor type II in microsatellite stable colon cancers.
1999307
18 1995284
19 2005283
20 1994279

About James K. V. Willson

James K. V. Willson is a scholar working on Molecular Biology, Oncology, Pathology and Forensic Medicine, Cancer Research and Computer Networks and Communications, having authored 175 papers that have together received 22.6k indexed citations. Recurring topics across this work include Genetic factors in colorectal cancer (43 papers), Colorectal Cancer Treatments and Studies (26 papers), TGF-β signaling in diseases (23 papers), DNA Repair Mechanisms (15 papers), Cancer Genomics and Diagnostics (15 papers), Cancer therapeutics and mechanisms (14 papers), Mobile Ad Hoc Networks (12 papers) and Cancer-related Molecular Pathways (12 papers). The work is most often cited by research in Pathology and Forensic Medicine (7.0k citations), Cancer Research (5.8k citations), Oncology (9.8k citations), Molecular Biology (14.0k citations) and Cell Biology (1.8k citations). James K. V. Willson has collaborated with scholars based in United States, China and United Kingdom. Frequent co-authors include Sanford D. Markowitz, Bert Vogelstein, Kenneth W. Kinzler, Suzanne J. Baker, Christoph Lengauer, Gregory J. Riggins, Stanley R. Hamilton, Victor E. Velculescu, Eric R. Fearon and Natalie Silliman. Their work appears in journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry, Oncogene, Science and Nature Genetics.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

Explore authors with similar magnitude of impact