Michael Kane

18.3k total citations · 6 hit papers
35 papers, 12.8k citations indexed

About

Michael Kane is a scholar working on Molecular Biology, Pathology and Forensic Medicine and Cancer Research. According to data from OpenAlex, Michael Kane has authored 35 papers receiving a total of 12.8k indexed citations (citations by other indexed papers that have themselves been cited), including 27 papers in Molecular Biology, 21 papers in Pathology and Forensic Medicine and 13 papers in Cancer Research. Recurrent topics in Michael Kane's work include Genetic factors in colorectal cancer (21 papers), DNA Repair Mechanisms (14 papers) and RNA Research and Splicing (9 papers). Michael Kane is often cited by papers focused on Genetic factors in colorectal cancer (21 papers), DNA Repair Mechanisms (14 papers) and RNA Research and Splicing (9 papers). Michael Kane collaborates with scholars based in United States, United Kingdom and Sweden. Michael Kane's co-authors include Richard D. Kolodner, Richard Fishel, Mary Kay Lescoe, Manchanahalli R. Satyanarayana Rao, Judy E. Garber, N G Copeland, Nancy A. Jenkins, Thomas A. Kunkel, J. Russell Lipford and Asad Umar and has published in prestigious journals such as Nature, Science and Cell.

In The Last Decade

Michael Kane

35 papers receiving 12.5k citations

Hit Papers

The human mutator gene homolog MSH2 and its association w... 1993 2026 2004 2015 1993 1994 1998 2016 2019 500 1000 1.5k 2.0k
What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

  1. The human mutator gene homolog MSH2 and its association with hereditary nonpolyposis colon cancer
    1993 2.3k citations Richard Fishel, Mary Kay Lescoe et al. Cell profile →
  2. Mutation in the DNA mismatch repair gene homologue hMLH 1 is associated with hereditary non-polyposis colon cancer
    1994 1.6k citations Christian Bronner, Sean M. Baker et al. Nature profile →
  3. Incidence and functional consequences of hMLH1 promoter hypermethylation in colorectal carcinoma
    1998 1.5k citations Asad Umar, Michael Kane et al. Proceedings of the National Academy of Sciences profile →
  4. Local regulation of gene expression by lncRNA promoters, transcription and splicing
    2016 905 citations J Engreitz, Jenna E. Haines et al. Nature profile →
  5. Activity-by-contact model of enhancer–promoter regulation from thousands of CRISPR perturbations
    2019 522 citations Charles P. Fulco, Joseph Nasser et al. Nature Genetics profile →
  6. Systematic mapping of functional enhancer–promoter connections with CRISPR interference
    2016 390 citations Charles P. Fulco, Mathias Munschauer et al. Science profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Michael Kane United States 32 7.8k 6.9k 4.6k 4.0k 1.8k 35 12.8k
Winfried Edelmann United States 58 7.9k 1.0× 2.8k 0.4× 1.8k 0.4× 2.0k 0.5× 1.9k 1.0× 140 11.3k
Javier Benı́tez Spain 51 3.9k 0.5× 1.5k 0.2× 2.1k 0.5× 2.2k 0.5× 2.1k 1.2× 243 7.6k
Denise Sheer United Kingdom 51 6.0k 0.8× 1.8k 0.3× 1.3k 0.3× 2.1k 0.5× 1.9k 1.0× 178 10.9k
N G Copeland United States 34 4.6k 0.6× 2.0k 0.3× 1.3k 0.3× 1.6k 0.4× 1.4k 0.8× 49 7.6k
C M Croce United States 46 5.2k 0.7× 1.5k 0.2× 1.3k 0.3× 2.4k 0.6× 1.4k 0.7× 102 8.8k
A. Malcolm R. Taylor United Kingdom 50 7.7k 1.0× 1.2k 0.2× 2.7k 0.6× 3.5k 0.9× 1.3k 0.7× 132 9.9k
Bruce A.J. Ponder United Kingdom 45 5.0k 0.6× 1.2k 0.2× 1.6k 0.4× 1.9k 0.5× 3.8k 2.1× 88 9.4k
Vladimír Kořínek Czechia 29 10.4k 1.3× 1.6k 0.2× 972 0.2× 2.6k 0.7× 1.7k 0.9× 72 12.8k
Luís Teixeira da Costa Portugal 17 4.7k 0.6× 1.0k 0.2× 1.3k 0.3× 3.1k 0.8× 1.5k 0.8× 45 7.3k
Yasuhito Yuasa Japan 46 4.3k 0.6× 1.4k 0.2× 1.8k 0.4× 1.8k 0.5× 1.2k 0.7× 147 6.7k

Countries citing papers authored by Michael Kane

Since Specialization
Citations

This map shows the geographic impact of Michael Kane's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Michael Kane with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Michael Kane more than expected).

Fields of papers citing papers by Michael Kane

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Michael Kane. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Michael Kane. The network helps show where Michael Kane may publish in the future.

Co-authorship network of co-authors of Michael Kane

This figure shows the co-authorship network connecting the top 25 collaborators of Michael Kane. A scholar is included among the top collaborators of Michael Kane based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Michael Kane. Michael Kane is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Bergman, Drew T., Thouis R. Jones, Vincent Liu, et al.. (2022). Compatibility rules of human enhancer and promoter sequences. Nature. 607(7917). 176–184. 81 indexed citations
2.
Fulco, Charles P., Joseph Nasser, Thouis R. Jones, et al.. (2019). Activity-by-contact model of enhancer–promoter regulation from thousands of CRISPR perturbations. Nature Genetics. 51(12). 1664–1669. 522 indexed citations breakdown →
3.
Fulco, Charles P., Mathias Munschauer, Rockwell Anyoha, et al.. (2016). Systematic mapping of functional enhancer–promoter connections with CRISPR interference. Science. 354(6313). 769–773. 390 indexed citations breakdown →
4.
Engreitz, J, Jenna E. Haines, Elizabeth M. Perez, et al.. (2016). Local regulation of gene expression by lncRNA promoters, transcription and splicing. Nature. 539(7629). 452–455. 905 indexed citations breakdown →
5.
Chou, Richard C., et al.. (2016). Treatment for Rheumatoid Arthritis and Risk of Alzheimer’s Disease: A Nested Case-Control Analysis. CNS Drugs. 30(11). 1111–1120. 142 indexed citations
6.
Meijer, Dimphna H., Yu Sun, Tao Liu, et al.. (2014). An Amino Terminal Phosphorylation Motif Regulates Intranuclear Compartmentalization of Olig2 in Neural Progenitor Cells. Journal of Neuroscience. 34(25). 8507–8518. 18 indexed citations
7.
Meijer, Dimphna H., Michael Kane, Shwetal Mehta, et al.. (2012). Separated at birth? The functional and molecular divergence of OLIG1 and OLIG2. Nature reviews. Neuroscience. 13(12). 819–831. 140 indexed citations
8.
Sun, Yu, Dimphna H. Meijer, John A. Alberta, et al.. (2011). Phosphorylation State of Olig2 Regulates Proliferation of Neural Progenitors. Neuron. 69(5). 906–917. 93 indexed citations
9.
Mehta, Shwetal, Emmanuelle Huillard, Santosh Kesari, et al.. (2011). The Central Nervous System-Restricted Transcription Factor Olig2 Opposes p53 Responses to Genotoxic Damage in Neural Progenitors and Malignant Glioma. Cancer Cell. 19(3). 359–371. 121 indexed citations
10.
Ligon, Keith L., Emmanuelle Huillard, Shwetal Mehta, et al.. (2007). Olig2-Regulated Lineage-Restricted Pathway Controls Replication Competence in Neural Stem Cells and Malignant Glioma. Neuron. 53(4). 503–517. 378 indexed citations
11.
Wang, Yuxun, Christopher D. Putnam, Michael Kane, et al.. (2005). Mutation in Rpa1 results in defective DNA double-strand break repair, chromosomal instability and cancer in mice. Nature Genetics. 37(7). 750–755. 122 indexed citations
12.
Wei, Kaichun, Allan Clark, Michael Kane, et al.. (2003). Inactivation of Exonuclease 1 in mice results in DNA mismatch repair defects, increased cancer susceptibility, and male and female sterility. Genes & Development. 17(5). 603–614. 261 indexed citations
13.
Kucherlapati, Melanie H., Kan Yang, Mari Kuraguchi, et al.. (2002). Haploinsufficiency of Flap endonuclease ( Fen1 ) leads to rapid tumor progression. Proceedings of the National Academy of Sciences. 99(15). 9924–9929. 197 indexed citations
14.
Verma, Manjusha, Michael Kane, Cecilia Brassett, et al.. (1999). Mononucleotide microsatellite instability and germline MSH6 mutation analysis in early onset colorectal cancer.. PubMed Central. 36(9). 678–82. 75 indexed citations
15.
Alani, Eric, Suman Lee, Michael Kane, Jack D. Griffith, & Richard D. Kolodner. (1997). Saccharomyces cerevisiae MSH2, a mispaired base recognition protein, also recognizes Holliday junctions in DNA. Journal of Molecular Biology. 265(3). 289–301. 83 indexed citations
16.
Edelmann, Winfried, Kan Yang, Asad Umar, et al.. (1997). Mutation in the Mismatch Repair Gene Msh6 Causes Cancer Susceptibility. Cell. 91(4). 467–477. 304 indexed citations
17.
Marsischky, Gerald, et al.. (1996). Redundancy of Saccharomyces cerevisiae MSH3 and MSH6 in MSH2-dependent mismatch repair.. Genes & Development. 10(4). 407–420. 482 indexed citations
18.
Kolodner, Richard D., Nigel R. Hall, J. Russell Lipford, et al.. (1994). Structure of the Human MSH2 Locus and Analysis of Two Muir-Torre Kindreds for msh2 Mutations. Genomics. 24(3). 516–526. 228 indexed citations
19.
Bronner, Christian, Sean M. Baker, Paul T. Morrison, et al.. (1994). Mutation in the DNA mismatch repair gene homologue hMLH 1 is associated with hereditary non-polyposis colon cancer. Nature. 368(6468). 258–261. 1605 indexed citations breakdown →
20.
Fishel, Richard, Mary Kay Lescoe, Manchanahalli R. Satyanarayana Rao, et al.. (1993). The human mutator gene homolog MSH2 and its association with hereditary nonpolyposis colon cancer. Cell. 75(5). 1027–1038. 2259 indexed citations breakdown →

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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