Mieke Schutte

14.3k total citations · 5 hit papers
63 papers, 8.7k citations indexed

About

Mieke Schutte is a scholar working on Oncology, Molecular Biology and Cancer Research. According to data from OpenAlex, Mieke Schutte has authored 63 papers receiving a total of 8.7k indexed citations (citations by other indexed papers that have themselves been cited), including 29 papers in Oncology, 28 papers in Molecular Biology and 20 papers in Cancer Research. Recurrent topics in Mieke Schutte's work include Cancer Genomics and Diagnostics (18 papers), Genetic factors in colorectal cancer (18 papers) and BRCA gene mutations in cancer (14 papers). Mieke Schutte is often cited by papers focused on Cancer Genomics and Diagnostics (18 papers), Genetic factors in colorectal cancer (18 papers) and BRCA gene mutations in cancer (14 papers). Mieke Schutte collaborates with scholars based in Netherlands, United States and United Kingdom. Mieke Schutte's co-authors include Stephan A. Hahn, Ralph H. Hruban, Scott E. Kern, Craig L. Weinstein, Luís Teixeira da Costa, Christopher A. Moskaluk, Charles J. Yeo, Ester Rozenblum, Aryeh Fischer and A.T.M. Shamsul Hoque and has published in prestigious journals such as Science, Proceedings of the National Academy of Sciences and Nucleic Acids Research.

In The Last Decade

Mieke Schutte

62 papers receiving 8.5k citations

Hit Papers

DPC4 , A Candidate Tumor Suppressor Gene at Human Chromos... 1994 2026 2004 2015 1996 1994 1996 1997 1996 500 1000 1.5k
What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

  1. DPC4 , A Candidate Tumor Suppressor Gene at Human Chromosome 18q21.1
    1996 1.9k citations Stephan A. Hahn, Mieke Schutte et al. profile →
  2. Frequent somatic mutations and homozygous deletions of the p16 (MTS1) gene in pancreatic adenocarcinoma
    1994 912 citations Carlos Caldas, Stephan A. Hahn et al. Nature Genetics profile →
  3. DPC4 gene in various tumor types.
    1996 570 citations Mieke Schutte, Ralph H. Hruban et al. profile →
  4. Tumor-suppressive pathways in pancreatic carcinoma.
    1997 554 citations Ester Rozenblum, Mieke Schutte et al. profile →
  5. Evaluation of candidate tumour suppressor genes on chromosome 18 in colorectal cancers
    1996 501 citations Sam Thiagalingam, Christoph Lengauer et al. Nature Genetics profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Mieke Schutte Netherlands 38 5.3k 5.2k 2.4k 2.1k 1.3k 63 8.7k
Charles Theillet France 55 4.0k 0.8× 5.3k 1.0× 2.6k 1.1× 1.0k 0.5× 1.9k 1.5× 140 8.7k
Frederic M. Waldman United States 53 2.5k 0.5× 3.9k 0.7× 2.9k 1.2× 1.6k 0.8× 2.0k 1.6× 136 8.0k
Gennaro Chiappetta Italy 50 2.1k 0.4× 4.3k 0.8× 1.9k 0.8× 916 0.4× 834 0.7× 110 7.4k
Rachael Natrajan United Kingdom 48 3.7k 0.7× 4.7k 0.9× 2.9k 1.2× 1.2k 0.6× 1.1k 0.9× 112 7.9k
Italia Bongarzone Italy 45 2.9k 0.5× 3.8k 0.7× 755 0.3× 1.1k 0.5× 1.2k 1.0× 113 7.4k
Montse Sánchez‐Céspedes Spain 51 2.6k 0.5× 7.8k 1.5× 3.5k 1.5× 1.7k 0.8× 637 0.5× 107 10.1k
Kay Savage United Kingdom 42 2.6k 0.5× 2.8k 0.6× 2.1k 0.9× 1.0k 0.5× 630 0.5× 66 5.7k
Wolfgang Dietmaier Germany 41 2.3k 0.4× 2.5k 0.5× 1.4k 0.6× 1.9k 0.9× 509 0.4× 135 6.2k
Ruth Holm Norway 45 2.8k 0.5× 2.8k 0.6× 1.3k 0.6× 991 0.5× 403 0.3× 187 6.1k
John I. Risinger United States 41 1.6k 0.3× 3.2k 0.6× 1.7k 0.7× 2.1k 1.0× 850 0.7× 94 5.8k

Countries citing papers authored by Mieke Schutte

Since Specialization
Citations

This map shows the geographic impact of Mieke Schutte's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mieke Schutte with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mieke Schutte more than expected).

Fields of papers citing papers by Mieke Schutte

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Mieke Schutte. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mieke Schutte. The network helps show where Mieke Schutte may publish in the future.

Co-authorship network of co-authors of Mieke Schutte

This figure shows the co-authorship network connecting the top 25 collaborators of Mieke Schutte. A scholar is included among the top collaborators of Mieke Schutte based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Mieke Schutte. Mieke Schutte is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Leyvraz, Serge, Mieke Schutte, Torsten Keßler, et al.. (2020). 1142P Treatment of metastatic uveal melanoma (mUM) through genomic profiling. Annals of Oncology. 31. S764–S764.
2.
Riaz, Muhammad, Buddy Setyono-Han, Mieke Timmermans, et al.. (2014). Growth and metastatic behavior of molecularly well-characterized human breast cancer cell lines in mice. Breast Cancer Research and Treatment. 148(1). 19–31. 6 indexed citations
3.
Hollestelle, Antoinette, Justine Peeters, Marcel Smid, et al.. (2013). Loss of E-cadherin is not a necessity for epithelial to mesenchymal transition in human breast cancer. Breast Cancer Research and Treatment. 138(1). 47–57. 103 indexed citations
4.
Hollestelle, Antoinette, Maartje J. Hooning, Ellen Crepin, et al.. (2010). Prevalence of the variant allele rs61764370 T>G in the 3′UTR of KRAS among Dutch BRCA1, BRCA2 and non-BRCA1/BRCA2 breast cancer families. Breast Cancer Research and Treatment. 128(1). 79–84. 28 indexed citations
5.
Wasielewski, Marijke, Maartje Nielsen, Ans van den Ouweland, et al.. (2010). Increased MUTYH mutation frequency among Dutch families with breast cancer and colorectal cancer. Breast Cancer Research and Treatment. 124(3). 635–641. 39 indexed citations
6.
Hollestelle, Antoinette, Fons Elstrodt, Mieke Timmermans, et al.. (2009). Four human breast cancer cell lines with biallelic inactivating α-catenin gene mutations. Breast Cancer Research and Treatment. 122(1). 125–133. 35 indexed citations
7.
Hollestelle, Antoinette, Fons Elstrodt, Jord H.A. Nagel, Wouter W. Kallemeijn, & Mieke Schutte. (2007). Phosphatidylinositol-3-OH Kinase or RAS Pathway Mutations in Human Breast Cancer Cell Lines. Molecular Cancer Research. 5(2). 195–201. 251 indexed citations
8.
Elstrodt, Fons, Antoinette Hollestelle, Jord H.A. Nagel, et al.. (2006). BRCA1 Mutation Analysis of 41 Human Breast Cancer Cell Lines Reveals Three New Deleterious Mutants. Cancer Research. 66(1). 41–45. 215 indexed citations
9.
Wasielewski, Marijke, Fons Elstrodt, Jan G.M. Klijn, Els M.J.J. Berns, & Mieke Schutte. (2006). Thirteen new p53 gene mutants identified among 41 human breast cancer cell lines. Breast Cancer Research and Treatment. 99(1). 97–101. 89 indexed citations
10.
Bock, Geertruida H. de, M J E Mourits, Mieke Schutte, et al.. (2006). Association between the CHEK2*1100delC germ line mutation and estrogen receptor status. International Journal of Gynecological Cancer. 16(s2). 552–555. 17 indexed citations
11.
Szabo, Csilla I., Mieke Schutte, Annegien Broeks, et al.. (2004). Are ATM Mutations 7271T→G and IVS10-6T→G Really High-Risk Breast Cancer-Susceptibility Alleles?. Cancer Research. 64(3). 840–843. 43 indexed citations
12.
Elstrodt, Fons, Winand N.M. Dinjens, Michel Molier, et al.. (2003). Allelotype of 28 human breast cancer cell lines and xenografts. British Journal of Cancer. 89(12). 2289–2292. 15 indexed citations
13.
Su, Gloria H., Jason Jungsik Song, Elizabeth A. Repasky, Mieke Schutte, & Scott E. Kern. (2001). Mutation rate of MAP2K4/MKK4 in breast carcinoma. Human Mutation. 19(1). 81–81. 49 indexed citations
14.
Dai, Jia Le, et al.. (1999). Transforming growth factor-? responsiveness inDPC4/SMAD4-null cancer cells. Molecular Carcinogenesis. 26(1). 37–43. 74 indexed citations
15.
Thiagalingam, Sam, Christoph Lengauer, Mieke Schutte, et al.. (1996). Evaluation of candidate tumour suppressor genes on chromosome 18 in colorectal cancers. Nature Genetics. 13(3). 343–346. 501 indexed citations breakdown →
16.
Schutte, Mieke, Luís Teixeira da Costa, Ester Rozenblum, et al.. (1995). Isolation of YAC insert sequences by representational difference analysis. Nucleic Acids Research. 23(20). 4127–4133. 7 indexed citations
17.
Caldas, Carlos, Stephan A. Hahn, Luís Teixeira da Costa, et al.. (1994). Frequent somatic mutations and homozygous deletions of the p16 (MTS1) gene in pancreatic adenocarcinoma. Nature Genetics. 8(1). 27–32. 912 indexed citations breakdown →
18.
Ebeling, Saskia B., Mieke Schutte, & Ton Logtenberg. (1993). The majority of human tonsillar CD5+ B cells express somatically mutated Vχ4 genes. European Journal of Immunology. 23(6). 1405–1408. 20 indexed citations
19.
20.
Schutte, Mieke, et al.. (1991). Antibody specificity and immunoglobulin VH utilization of human monoclonal CD5+ B cell lines. European Journal of Immunology. 21(5). 1115–1121. 51 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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