Joseph Willis

26.4k total citations · 1 hit paper
162 papers, 8.8k citations indexed

About

Joseph Willis is a scholar working on Surgery, Molecular Biology and Oncology. According to data from OpenAlex, Joseph Willis has authored 162 papers receiving a total of 8.8k indexed citations (citations by other indexed papers that have themselves been cited), including 77 papers in Surgery, 51 papers in Molecular Biology and 46 papers in Oncology. Recurrent topics in Joseph Willis's work include Esophageal Cancer Research and Treatment (49 papers), Genetic factors in colorectal cancer (38 papers) and Gastric Cancer Management and Outcomes (28 papers). Joseph Willis is often cited by papers focused on Esophageal Cancer Research and Treatment (49 papers), Genetic factors in colorectal cancer (38 papers) and Gastric Cancer Management and Outcomes (28 papers). Joseph Willis collaborates with scholars based in United States, Canada and China. Joseph Willis's co-authors include Sanford D. Markowitz, Michael Sivak, Amitabh Chak, William M. Grady, James Lutterbaugh, Dawn Dawson, Lois L. Myeroff, James K. V. Willson, Andrew M. Rollins and Kishore Guda and has published in prestigious journals such as Science, Proceedings of the National Academy of Sciences and Nature Communications.

In The Last Decade

Joseph Willis

157 papers receiving 8.7k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Comparative lesion sequencing provides insights into tumor evolution

2008 596 citations Siân Jones, Wei-Dong Chen et al. Proceedings of the National Academy of Sciences profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Joseph Willis United States	48	3.8k	2.6k	2.2k	1.8k	1.8k	162	8.8k
Hoguen Kim South Korea	53	4.5k 1.2×	4.4k 1.7×	2.1k 0.9×	1.5k 0.8×	2.3k 1.3×	205	10.3k
Wael El‐Rifai United States	56	5.3k 1.4×	2.6k 1.0×	2.6k 1.2×	2.7k 1.5×	1.1k 0.6×	209	10.4k
Robert S. Warren United States	43	3.5k 0.9×	3.9k 1.5×	1.3k 0.6×	1.1k 0.6×	1.3k 0.7×	123	8.7k
Mengchao Wu China	59	4.8k 1.3×	3.7k 1.4×	3.5k 1.6×	2.0k 1.1×	931 0.5×	312	13.7k
Raheela Ashfaq United States	58	4.0k 1.1×	3.8k 1.5×	3.0k 1.3×	1.8k 1.0×	974 0.5×	194	10.1k
Hiroaki Nagano Japan	60	5.0k 1.3×	4.2k 1.6×	3.5k 1.6×	1.7k 0.9×	915 0.5×	451	13.1k
Shigeki Arii Japan	60	5.4k 1.4×	3.4k 1.3×	3.3k 1.5×	1.5k 0.8×	1.3k 0.7×	307	14.4k
Luigi Maria Larocca Italy	54	3.5k 0.9×	3.3k 1.3×	1.3k 0.6×	1.1k 0.6×	2.0k 1.1×	338	10.4k
Robert Grützmann Germany	49	3.2k 0.8×	5.0k 1.9×	2.3k 1.0×	1.5k 0.8×	938 0.5×	298	8.8k
Teresa A. Brentnall United States	51	3.0k 0.8×	4.1k 1.6×	2.0k 0.9×	626 0.3×	1.9k 1.1×	119	8.5k

Countries citing papers authored by Joseph Willis

Since Specialization

Citations

This map shows the geographic impact of Joseph Willis's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Joseph Willis with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Joseph Willis more than expected).

Fields of papers citing papers by Joseph Willis

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Joseph Willis. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Joseph Willis. The network helps show where Joseph Willis may publish in the future.

Co-authorship network of co-authors of Joseph Willis

This figure shows the co-authorship network connecting the top 25 collaborators of Joseph Willis. A scholar is included among the top collaborators of Joseph Willis based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Joseph Willis. Joseph Willis is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

Sort: Min cites: Since: Top N: Style:

20 of 20 papers shown

Devall, Matthew A.M., Xiangqing Sun, Steven M. Powell, et al.. (2025). Association between dietary fructose and human colon DNA methylation: implication for racial disparities in colorectal cancer risk using a cross-sectional study. American Journal of Clinical Nutrition. 121(3). 522–534. 3 indexed citations

Fu, Pingfu, Sirvan Khalighi, Mojgan Mokhtari, et al.. (2025). Collagen disorder architecture features are associated with clinical, molecular, genetic factors and survival outcomes in colon cancer. npj Precision Oncology. 9(1). 304–304.

Fu, Pingfu, et al.. (2024). Machine Vision–Detected Peritumoral Lymphocytic Aggregates Are Associated With Disease-Free Survival in Patients With Papillary Thyroid Carcinoma. Laboratory Investigation. 104(12). 102168–102168. 2 indexed citations

Moinova, Helen, Suman Verma, John A. Dumot, et al.. (2024). Multicenter, Prospective Trial of Nonendoscopic Biomarker-Driven Detection of Barrett's Esophagus and Esophageal Adenocarcinoma. The American Journal of Gastroenterology. 119(11). 2206–2214. 7 indexed citations

Chandar, Apoorva K., William M. Grady, Marcia Irene Canto, et al.. (2023). Patients With Esophageal Adenocarcinoma With Prior Gastroesophageal Reflux Disease Symptoms Are Similar to Those Without Gastroesophageal Reflux Disease: A Cross-Sectional Study. The American Journal of Gastroenterology. 119(5). 823–829. 2 indexed citations

Yu, Ming, Helen Moinova, Amber Willbanks, et al.. (2022). Novel DNA Methylation Biomarker Panel for Detection of Esophageal Adenocarcinoma and High-Grade Dysplasia. Clinical Cancer Research. 28(17). 3761–3769. 11 indexed citations

Miller, Tyler E., Michael Yang, David J. Bajor, et al.. (2018). Clinical utility of reflex testing using focused next-generation sequencing for management of patients with advanced lung adenocarcinoma. Journal of Clinical Pathology. 71(12). 1108–1115. 34 indexed citations

Moinova, Helen, Thomas LaFramboise, James Lutterbaugh, et al.. (2018). Identifying DNA methylation biomarkers for non-endoscopic detection of Barrett’s esophagus. Science Translational Medicine. 10(424). 104 indexed citations

Yu, Ming, Sean K. Maden, Matthew D. Stachler, et al.. (2018). Subtypes of Barrett’s oesophagus and oesophageal adenocarcinoma based on genome-wide methylation analysis. Gut. 68(3). 389–399. 33 indexed citations

10.

Blum, Andrew E., Yan Guo, Lakshmeswari Ravi, et al.. (2016). RNA Sequencing Identifies Transcriptionally Viable Gene Fusions in Esophageal Adenocarcinomas. Cancer Research. 76(19). 5628–5633. 20 indexed citations

11.

Sun, Xiangqing, Robert C. Elston, Jill S. Barnholtz‐Sloan, et al.. (2016). Predicting Barrett's Esophagus in Families: An Esophagus Translational Research Network (BETRNet) Model Fitting Clinical Data to a Familial Paradigm. Cancer Epidemiology Biomarkers & Prevention. 25(5). 727–735. 10 indexed citations

12.

Yu, Ming, Andrew M. Kaz, Shelli M. Morris, et al.. (2015). Methylated B3GAT2 and ZNF793 Are Potential Detection Biomarkers for Barrett's Esophagus. Cancer Epidemiology Biomarkers & Prevention. 24(12). 1890–1897. 11 indexed citations

13.

Guda, Kishore, Stephen P. Fink, Ginger L. Milne, et al.. (2014). Inactivating Mutation in the Prostaglandin Transporter Gene, SLCO2A1 , Associated with Familial Digital Clubbing, Colon Neoplasia, and NSAID Resistance. Cancer Prevention Research. 7(8). 805–812. 27 indexed citations

14.

Moinova, Helen, Rom S. Leidner, Lakshmeswari Ravi, et al.. (2012). Aberrant Vimentin Methylation Is Characteristic of Upper Gastrointestinal Pathologies. Cancer Epidemiology Biomarkers & Prevention. 21(4). 594–600. 34 indexed citations

15.

Zhou, Dawang, Yongyou Zhang, Hongtan Wu, et al.. (2011). Mst1 and Mst2 protein kinases restrain intestinal stem cell proliferation and colonic tumorigenesis by inhibition of Yes-associated protein (Yap) overabundance. Proceedings of the National Academy of Sciences. 108(49). E1312–20. 377 indexed citations

16.

Zhao, Yiqing, Xiaodong Zhang, Kishore Guda, et al.. (2010). Identification and functional characterization of paxillin as a target of protein tyrosine phosphatase receptor T. Proceedings of the National Academy of Sciences. 107(6). 2592–2597. 63 indexed citations

17.

Sun, Xiangqing, Robert C. Elston, Jill S. Barnholtz‐Sloan, et al.. (2010). A Segregation Analysis of Barrett's Esophagus and Associated Adenocarcinomas. Cancer Epidemiology Biomarkers & Prevention. 19(3). 666–674. 33 indexed citations

18.

Willis, Joseph, et al.. (2010). New Museums and Historical Sites. 38(1). 144. 1 indexed citations

19.

Jones, Siân, Wei-Dong Chen, Giovanni Parmigiani, et al.. (2008). Comparative lesion sequencing provides insights into tumor evolution. Proceedings of the National Academy of Sciences. 105(11). 4283–4288. 596 indexed citations breakdown →

20.

Hao, Xingpei, Joseph Willis, T G Pretlow, et al.. (2000). Loss of fragile histidine triad expression in colorectal carcinomas and premalignant lesions.. PubMed. 60(1). 18–21. 87 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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