James Lutterbaugh

6.9k total citations · 1 hit paper
32 papers, 3.5k citations indexed

About

James Lutterbaugh is a scholar working on Pathology and Forensic Medicine, Molecular Biology and Oncology. According to data from OpenAlex, James Lutterbaugh has authored 32 papers receiving a total of 3.5k indexed citations (citations by other indexed papers that have themselves been cited), including 20 papers in Pathology and Forensic Medicine, 17 papers in Molecular Biology and 14 papers in Oncology. Recurrent topics in James Lutterbaugh's work include Genetic factors in colorectal cancer (20 papers), Cancer Genomics and Diagnostics (7 papers) and Epigenetics and DNA Methylation (6 papers). James Lutterbaugh is often cited by papers focused on Genetic factors in colorectal cancer (20 papers), Cancer Genomics and Diagnostics (7 papers) and Epigenetics and DNA Methylation (6 papers). James Lutterbaugh collaborates with scholars based in United States, China and Germany. James Lutterbaugh's co-authors include Sanford D. Markowitz, Joseph Willis, Lois L. Myeroff, James K. V. Willson, Lakshmi Kasturi, Martina Veigl, Seong‐Jin Kim, William M. Grady, W. David Sedwick and Petra Platzer and has published in prestigious journals such as Science, Proceedings of the National Academy of Sciences and Journal of Clinical Investigation.

In The Last Decade

James Lutterbaugh

32 papers receiving 3.4k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Biallelic inactivation of hMLH 1 by epigenetic gene silencing, a novel mechanism causing human MSI cancers

1998 507 citations Martina Veigl, Lakshmi Kasturi et al. Proceedings of the National Academy of Sciences profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
James Lutterbaugh United States	25	2.1k	1.5k	1.4k	940	415	32	3.5k
Lois L. Myeroff United States	20	2.5k 1.2×	1.5k 1.1×	1.5k 1.0×	791 0.8×	361 0.9×	22	3.6k
Stanley R. Hamilton United States	11	1.1k 0.5×	1.4k 1.0×	1.4k 1.0×	567 0.6×	252 0.6×	12	2.5k
Prashant Bavi Saudi Arabia	34	1.4k 0.7×	679 0.5×	929 0.7×	699 0.7×	347 0.8×	76	2.8k
Abdul K. Siraj Saudi Arabia	29	1.5k 0.7×	623 0.4×	812 0.6×	577 0.6×	293 0.7×	121	2.7k
Hiroya Asou Japan	29	2.3k 1.1×	535 0.4×	1.2k 0.9×	839 0.9×	342 0.8×	70	3.9k
Ja‐Lok Ku South Korea	33	1.5k 0.7×	707 0.5×	1.0k 0.7×	690 0.7×	191 0.5×	112	2.8k
Zhaoshi Zeng United States	27	1.0k 0.5×	686 0.5×	1.3k 0.9×	897 1.0×	182 0.4×	37	2.6k
Harith Rajagopalan United States	13	2.3k 1.1×	827 0.6×	1.5k 1.1×	1.0k 1.1×	382 0.9×	26	3.8k
Marian Grade Germany	31	1.5k 0.7×	570 0.4×	1.4k 1.0×	879 0.9×	219 0.5×	85	3.0k
E Tahara Japan	31	1.2k 0.6×	456 0.3×	935 0.7×	405 0.4×	252 0.6×	106	2.6k

Countries citing papers authored by James Lutterbaugh

Since Specialization

Citations

This map shows the geographic impact of James Lutterbaugh's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by James Lutterbaugh with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites James Lutterbaugh more than expected).

Fields of papers citing papers by James Lutterbaugh

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by James Lutterbaugh. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by James Lutterbaugh. The network helps show where James Lutterbaugh may publish in the future.

Co-authorship network of co-authors of James Lutterbaugh

This figure shows the co-authorship network connecting the top 25 collaborators of James Lutterbaugh. A scholar is included among the top collaborators of James Lutterbaugh based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with James Lutterbaugh. James Lutterbaugh is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Moinova, Helen, Thomas LaFramboise, James Lutterbaugh, et al.. (2018). Identifying DNA methylation biomarkers for non-endoscopic detection of Barrett’s esophagus. Science Translational Medicine. 10(424). 104 indexed citations

Varadan, Vinay, Lakshmeswari Ravi, James Lutterbaugh, et al.. (2016). Biochemical and functional characterization of glycosylation-associated mutational landscapes in colon cancer. Scientific Reports. 6(1). 23642–23642. 51 indexed citations

Varadan, Vinay, Salendra Singh, Arman Nosrati, et al.. (2015). ENVE: a novel computational framework characterizes copy-number mutational landscapes in colorectal cancers from African American patients. Genome Medicine. 7(1). 69–69. 2 indexed citations

Guda, Kishore, Martina Veigl, Vinay Varadan, et al.. (2015). Novel recurrently mutated genes in African American colon cancers. Proceedings of the National Academy of Sciences. 112(4). 1149–1154. 92 indexed citations

Lutterbaugh, James, et al.. (2014). GNAS Mutations Identify a Set of Right-Sided, RAS Mutant, Villous Colon Cancers. PLoS ONE. 9(1). e87966–e87966. 37 indexed citations

Luo, Yanxin, Chao-Jen Wong, Andrew M. Kaz, et al.. (2014). Differences in DNA Methylation Signatures Reveal Multiple Pathways of Progression From Adenoma to Colorectal Cancer. Gastroenterology. 147(2). 418–429.e8. 150 indexed citations

Thompson, Cheryl L., Stephen P. Fink, James Lutterbaugh, et al.. (2013). Genetic Variation in 15-Hydroxyprostaglandin Dehydrogenase and Colon Cancer Susceptibility. PLoS ONE. 8(5). e64122–e64122. 10 indexed citations

Luo, Yanxin, Andrew M. Kaz, Samornmas Kanngurn, et al.. (2013). NTRK3 Is a Potential Tumor Suppressor Gene Commonly Inactivated by Epigenetic Mechanisms in Colorectal Cancer. PLoS Genetics. 9(7). e1003552–e1003552. 74 indexed citations

Moinova, Helen, Rom S. Leidner, Lakshmeswari Ravi, et al.. (2012). Aberrant Vimentin Methylation Is Characteristic of Upper Gastrointestinal Pathologies. Cancer Epidemiology Biomarkers & Prevention. 21(4). 594–600. 34 indexed citations

10.

Akhtar‐Zaidi, Batool, Lingyun Song, Cynthia F. Bartels, et al.. (2012). H3K4me3 inversely correlates with DNA methylation at a large class of non-CpG-island-containing start sites. Genome Medicine. 4(5). 47–47. 29 indexed citations

11.

Guda, Kishore, James Lutterbaugh, Georgia L. Wiesner, et al.. (2009). Infrequent Detection of Germline Allele-Specific Expression of TGFBR1 in Lymphoblasts and Tissues of Colon Cancer Patients. Cancer Research. 69(12). 4959–4961. 15 indexed citations

12.

Chen, Wei-Dong, Zhifen Han, Joel Skoletsky, et al.. (2005). Detection in Fecal DNA of Colon Cancer–Specific Methylation of the Nonexpressed Vimentin Gene. JNCI Journal of the National Cancer Institute. 97(15). 1124–1132. 283 indexed citations

13.

Wilson, Keith E., David H. Mack, Dawn Dawson, et al.. (2003). PMEPA1, a transforming growth factor-beta-induced marker of terminal colonocyte differentiation whose expression is maintained in primary and metastatic colon cancer.. PubMed. 63(7). 1568–75. 76 indexed citations

14.

Platzer, Petra, Madhvi B. Upender, Keith E. Wilson, et al.. (2002). Silence of chromosomal amplifications in colon cancer.. PubMed. 62(4). 1134–8. 125 indexed citations

15.

Grady, William M., et al.. (2001). Detection of aberrantly methylated hMLH1 promoter DNA in the serum of patients with microsatellite unstable colon cancer. Gastroenterology. 120(5). A291–A291. 76 indexed citations

16.

Ma, Ai‐Hong, Liang Xia, Susan J. Littman, et al.. (2000). Somatic mutation of hPMS2 as a possible cause of sporadic human colon cancer with microsatellite instability. Oncogene. 19(18). 2249–2256. 23 indexed citations

17.

Grady, William M., Lois L. Myeroff, Sandra E. Swinler, et al.. (1999). Mutational inactivation of transforming growth factor beta receptor type II in microsatellite stable colon cancers.. PubMed. 59(2). 320–4. 307 indexed citations

18.

Eshleman, James R., Preston S. Donover, Susan J. Littman, et al.. (1998). Increased transversions in a novel mutator colon cancer cell line. Oncogene. 16(9). 1125–1130. 13 indexed citations

19.

Eshleman, James R., et al.. (1996). Spontaneous apoptosis in human colon tumor cell lines and the relation of wt p53 to apoptosis.. PubMed. 109(7). 537–41. 8 indexed citations

20.

Markowitz, Sanford D., et al.. (1994). A benign cultured colon adenoma bears three genetically altered colon cancer oncogenes, but progresses to tumorigenicity and transforming growth factor-beta independence without inactivating the p53 tumor suppressor gene.. Journal of Clinical Investigation. 93(3). 1005–1013. 54 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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