Kathleen Ottina
- Molecular Biology top 5%
- Cellular and Molecular Neuroscience top 2%
- Genetics top 5%
- Neurology top 2%
- Cell Biology top 5%
- Co-authors
- David M. SabatiniRudolph E. TanziJames F. GusellaJoseph B. MartinMary Anne AndersonAnne B. YoungNancy S. WexlerAlan Y. Sakaguchi
- Topics
- PI3K/AKT/mTOR signaling in cancer (6 papers)Protein Kinase Regulation and GTPase Signaling (4 papers)Lipid Membrane Structure and Behavior (3 papers)
- Partner nations
- United StatesJapanCzechia
In The Last Decade
Kathleen Ottina
11 papers receiving 2.9k citations
Hit Papers
Peers
Comparison fields: 5 of 122
- Molecular Biology 2.1k
- Cellular and Molecular Neuroscience 884
- Genetics 612
- Neurology 442
- Cell Biology 233
Countries citing papers authored by Kathleen Ottina
This map shows the geographic impact of Kathleen Ottina's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Kathleen Ottina with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Kathleen Ottina more than expected).
Fields of papers citing papers by Kathleen Ottina
This network shows the impact of papers produced by Kathleen Ottina. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Kathleen Ottina. The network helps show where Kathleen Ottina may publish in the future.
Co-authorship network of co-authors of Kathleen Ottina
This figure shows the co-authorship network connecting the top 25 collaborators of Kathleen Ottina. A scholar is included among the top collaborators of Kathleen Ottina based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Kathleen Ottina. Kathleen Ottina is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 135 | |
| 2 | 3 | |
| 3 | mTORC1 Phosphorylation Sites Encode Their Sensitivity to Starvation and Rapamycin | 349 |
| 4 | 55 | |
| 5 | 26 | |
| 6 | The mTOR-Regulated Phosphoproteome Reveals a Mechanism of mTORC1-Mediated Inhibition of Growth Factor Signalingbreakdown → | 840 |
| 7 | 17 | |
| 8 | A polymorphic DNA marker genetically linked to Huntington's diseasebreakdown → | 1592 |
| 9 | 38 | |
| 10 | 9 | |
| 11 | 4 |
About Kathleen Ottina
Kathleen Ottina is a scholar working on Geriatrics and Gerontology, Molecular Biology and Biochemistry, having authored 11 papers that have together received 3.1k indexed citations. Recurring topics across this work include PI3K/AKT/mTOR signaling in cancer (6 papers), Protein Kinase Regulation and GTPase Signaling (4 papers) and Lipid Membrane Structure and Behavior (3 papers). The work is most often cited by research in Cellular and Molecular Neuroscience (884 citations), Aging (55 citations) and Molecular Biology (2.1k citations). Kathleen Ottina has collaborated with scholars based in United States, Japan and Czechia. Frequent co-authors include David M. Sabatini, Rudolph E. Tanzi, James F. Gusella, Joseph B. Martin, Mary Anne Anderson, Anne B. Young, Nancy S. Wexler, Alan Y. Sakaguchi, Ernesto Bonilla and Ira Shoulson. Their work appears in journals such as Nature, Science and Journal of Biological Chemistry.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.