Dianna E. Willis

4.7k total citations
53 papers, 3.7k citations indexed

About

Dianna E. Willis is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Cell Biology. According to data from OpenAlex, Dianna E. Willis has authored 53 papers receiving a total of 3.7k indexed citations (citations by other indexed papers that have themselves been cited), including 38 papers in Molecular Biology, 25 papers in Cellular and Molecular Neuroscience and 8 papers in Cell Biology. Recurrent topics in Dianna E. Willis's work include Nerve injury and regeneration (17 papers), RNA Research and Splicing (15 papers) and Signaling Pathways in Disease (9 papers). Dianna E. Willis is often cited by papers focused on Nerve injury and regeneration (17 papers), RNA Research and Splicing (15 papers) and Signaling Pathways in Disease (9 papers). Dianna E. Willis collaborates with scholars based in United States, United Kingdom and Germany. Dianna E. Willis's co-authors include Jeffery L. Twiss, Tanuja T. Merianda, Deepika Vuppalanchi, Mike Fainzilber, C. Thong, Rajiv R. Ratan, Jay Chang, Christine E. Holt, Eran Perlson and Brett Langley and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Neuron.

In The Last Decade

Dianna E. Willis

53 papers receiving 3.7k citations

Author Peers

Peers are selected by citation overlap in the author's most active subfields. citations · hero ref

Author Last Decade Papers Cites
Dianna E. Willis 2.4k 1.7k 647 579 318 53 3.7k
Rebecca Matsas 2.3k 0.9× 2.1k 1.3× 305 0.5× 682 1.2× 394 1.2× 97 4.0k
Hiroko Yano 1.5k 0.6× 1.3k 0.8× 409 0.6× 447 0.8× 204 0.6× 56 2.8k
Markus Plomann 2.2k 0.9× 1.3k 0.8× 1.1k 1.6× 427 0.7× 335 1.1× 49 3.7k
Keiichi Uyemura 1.6k 0.7× 1.4k 0.8× 653 1.0× 550 0.9× 167 0.5× 104 3.1k
Steven H. Nye 2.2k 0.9× 1.2k 0.7× 349 0.5× 516 0.9× 227 0.7× 22 3.8k
Hiroaki Asou 1.6k 0.7× 988 0.6× 530 0.8× 760 1.3× 275 0.9× 126 3.4k
Hosung Jung 1.8k 0.7× 1.1k 0.7× 407 0.6× 304 0.5× 840 2.6× 56 3.5k
John J. Hemperly 2.4k 1.0× 1.3k 0.8× 782 1.2× 713 1.2× 182 0.6× 43 4.1k
Elisa Barbarese 3.1k 1.3× 816 0.5× 662 1.0× 1.1k 1.9× 232 0.7× 67 4.3k
Victor Nurcombe 2.4k 1.0× 1.3k 0.8× 1.3k 2.0× 511 0.9× 656 2.1× 72 4.3k

Countries citing papers authored by Dianna E. Willis

Since Specialization
Citations

This map shows the geographic impact of Dianna E. Willis's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Dianna E. Willis with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Dianna E. Willis more than expected).

Fields of papers citing papers by Dianna E. Willis

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Dianna E. Willis. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Dianna E. Willis. The network helps show where Dianna E. Willis may publish in the future.

Co-authorship network of co-authors of Dianna E. Willis

This figure shows the co-authorship network connecting the top 25 collaborators of Dianna E. Willis. A scholar is included among the top collaborators of Dianna E. Willis based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Dianna E. Willis. Dianna E. Willis is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Siddiq, Mustafa M., Carlos A. Toro, Jens Hansen, et al.. (2023). Spinal cord injury regulates circular RNA expression in axons. Frontiers in Molecular Neuroscience. 16. 1183315–1183315. 3 indexed citations
2.
Willis, Dianna E. & Peter A. Goldstein. (2022). Targeting Affective Mood Disorders With Ketamine to Prevent Chronic Postsurgical Pain. SHILAP Revista de lepidopterología. 3. 872696–872696. 2 indexed citations
3.
Mellado, Wilfredo, et al.. (2021). Failure to Upregulate the RNA Binding Protein ZBP After Injury Leads to Impaired Regeneration in a Rodent Model of Diabetic Peripheral Neuropathy. Frontiers in Molecular Neuroscience. 14. 728163–728163. 7 indexed citations
4.
Morquette, Barbara, Mohammad Rasool Khazaei, Nicolás Unsain, et al.. (2020). Collapsin Response Mediator Protein 4 (CRMP4) Facilitates Wallerian Degeneration and Axon Regeneration following Sciatic Nerve Injury. eNeuro. 7(2). ENEURO.0479–19.2020. 11 indexed citations
5.
Picci, Cristina, V. Wong, Nazia M. Alam, et al.. (2020). HDAC6 inhibition promotes α-tubulin acetylation and ameliorates CMT2A peripheral neuropathy in mice. Experimental Neurology. 328. 113281–113281. 48 indexed citations
6.
Kalinski, Ashley L., Amar N. Kar, Andrew P. Tosolini, et al.. (2019). Deacetylation of Miro1 by HDAC6 blocks mitochondrial transport and mediates axon growth inhibition. The Journal of Cell Biology. 218(6). 1871–1890. 82 indexed citations
8.
Ekins, Sean, Nadia K. Litterman, Renée J.G. Arnold, et al.. (2015). A brief review of recent Charcot-Marie-Tooth research and priorities. F1000Research. 4. 53–53. 30 indexed citations
9.
Dulin, Jennifer N., Ana Antunes‐Martins, Vijayendran Chandran, et al.. (2015). Transcriptomic Approaches to Neural Repair. Journal of Neuroscience. 35(41). 13860–13867. 24 indexed citations
11.
Vuppalanchi, Deepika, Tanuja T. Merianda, Christopher J. Donnelly, et al.. (2012). Lysophosphatidic acid differentially regulates axonal mRNA translation through 5′UTR elements. Molecular and Cellular Neuroscience. 50(2). 136–146. 28 indexed citations
12.
Willis, Dianna E., Mei Xu, Christopher J. Donnelly, et al.. (2011). Axonal Localization of Transgene mRNA in Mature PNS and CNS Neurons. Journal of Neuroscience. 31(41). 14481–14487. 62 indexed citations
13.
Gumy, Laura F., Giles S.H. Yeo, Yi‐Chun Loraine Tung, et al.. (2010). Transcriptome analysis of embryonic and adult sensory axons reveals changes in mRNA repertoire localization. RNA. 17(1). 85–98. 293 indexed citations
15.
Yoo, Soonmoon, Elena Iavnilovitch, Dianna E. Willis, et al.. (2008). Localized Regulation of Axonal RanGTPase Controls Retrograde Injury Signaling in Peripheral Nerve. Neuron. 59(2). 241–252. 176 indexed citations
16.
Merianda, Tanuja T., Andrew C. Lin, Deepika Vuppalanchi, et al.. (2008). A functional equivalent of endoplasmic reticulum and Golgi in axons for secretion of locally synthesized proteins. Molecular and Cellular Neuroscience. 40(2). 128–142. 147 indexed citations
17.
Willis, Dianna E. & Jeffery L. Twiss. (2006). The evolving roles of axonally synthesized proteins in regeneration. Current Opinion in Neurobiology. 16(1). 111–118. 102 indexed citations
18.
Rajasekaran, Sigrid A., et al.. (2004). Na,K-ATPase β1-Subunit Increases the Translation Efficiency of the α1-Subunit in MSV-MDCK Cells. Molecular Biology of the Cell. 15(7). 3224–3232. 49 indexed citations
19.
Perlson, Eran, Dianna E. Willis, Rada Massarwa, et al.. (2003). Axoplasmic Importins Enable Retrograde Injury Signaling in Lesioned Nerve. Neuron. 40(6). 1095–1104. 392 indexed citations
20.
Jenkins, Gísli, Stephen E. Bottoms, Geoffrey J. Laurent, et al.. (2003). Formation of LID vector complexes in water alters physicochemical properties and enhances pulmonary gene expression in vivo. Gene Therapy. 10(12). 1026–1034. 23 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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