James E. Vince
- Immunology top 0.5%
- Immune Response and Inflammation 15
- interferon and immune responses 14
- Phagocytosis and Immune Regulation 10
- Neutrophil, Myeloperoxidase and Oxidative Mechanisms 7
- Molecular Biology top 0.5%
- Inflammasome and immune disorders 40
- Cell death mechanisms and regulation 29
- Heme Oxygenase-1 and Carbon Monoxide 8
- Cancer Research top 1%
- NF-κB Signaling Pathways 11
- Nephrology top 1%
- Biological Psychiatry top 5%
James E. Vince
71 papers receiving 8.0k citations
Hit Papers
Peers
Comparison fields: 5 of 127
- Immunology 3.5k
- Molecular Biology 6.2k
- Cancer Research 1.3k
- Nephrology 435
- Biological Psychiatry 95
Countries citing papers authored by James E. Vince
This map shows the geographic impact of James E. Vince's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by James E. Vince with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites James E. Vince more than expected).
Fields of papers citing papers by James E. Vince
This network shows the impact of papers produced by James E. Vince. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by James E. Vince. The network helps show where James E. Vince may publish in the future.
Co-authorship network
The 25 scholars most cited alongside James E. Vince, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2025 | 0 | |
| 2 | 2024 | 11 | |
| 3 | 2023 | 44 | |
| 4 | 2023 | 1 | |
| 5 | 2021 | 3 | |
| 6 | 2020 | 1 | |
| 7 | 2019 | 97 | |
| 8 | 2019 | 82 | |
| 9 | Pyroptosis versus necroptosis: similarities, differences, and crosstalkbreakdown → | 2018 | 812 |
| 10 | Active MLKL triggers the NLRP3 inflammasome in a cell-intrinsic mannerbreakdown → | 2017 | 377 |
| 11 | 2017 | 62 | |
| 12 | 2017 | 76 | |
| 13 | 2017 | 38 | |
| 14 | 2016 | 12 | |
| 15 | 2014 | 206 | |
| 16 | AIM2 and NLRP3 inflammasomes activate both apoptotic and pyroptotic death pathways via ASCbreakdown → | 2013 | 416 |
| 17 | 2013 | 99 | |
| 18 | 2010 | 36 | |
| 19 | IAP Antagonists Target cIAP1 to Induce TNFα-Dependent Apoptosisbreakdown → | 2007 | 879 |
| 20 | 2004 | 90 |
About James E. Vince
James E. Vince is a scholar working on Immunology, Molecular Biology, Cancer Research, Endocrinology and Nephrology, having authored 76 papers that have together received 8.1k indexed citations. Recurring topics across this work include Inflammasome and immune disorders (40 papers), Cell death mechanisms and regulation (29 papers), Immune Response and Inflammation (15 papers), interferon and immune responses (14 papers), NF-κB Signaling Pathways (11 papers), Phagocytosis and Immune Regulation (10 papers), Heme Oxygenase-1 and Carbon Monoxide (8 papers) and Neutrophil, Myeloperoxidase and Oxidative Mechanisms (7 papers). The work is most often cited by research in Immunology (3.5k citations), Molecular Biology (6.2k citations), Cancer Research (1.3k citations), Nephrology (435 citations) and Biological Psychiatry (95 citations). James E. Vince has collaborated with scholars based in Australia, United States and Switzerland. Frequent co-authors include Daniel Frank, John Silke, Kate E. Lawlor, David L. Vaux, Rebecca Feltham, Philippe Menu, W. Wei‐Lynn Wong, James M. Murphy, Nufail Khan and Stephen M. Condon. Their work appears in journals such as Cell Death and Differentiation, Nature Communications, Cell Reports, Nature Immunology and PLoS ONE.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.