Kate E. Lawlor

8.8k citations

67 papers · 5.7k indexed · 4 hit papers · h-index 37

Immunology top 0.5%
- Immune Response and Inflammation 20
- interferon and immune responses 13
- Phagocytosis and Immune Regulation 8
- Immune Cell Function and Interaction 7
Rheumatology top 1%
Immunology and Allergy top 2%
- Cell Adhesion Molecules Research 9
Molecular Biology top 2%
- Inflammasome and immune disorders 29
- Cell death mechanisms and regulation 17
Cancer Research top 2%
Oncology
- Cytokine Signaling Pathways and Interactions 7

Co-authors: James E. Vince Ian P. Wicks David L. Vaux Ian K. Campbell Amanda Fosang Christopher B. Little James M. Murphy Seth L. Masters
Cited by: Immunology Rheumatology Immunology and Allergy
Journals: Cell Reports (6 papers)Journal of Clinical Investigation (5 papers)Cell Death and Disease (5 papers)
Partner nations: Australia United States Switzerland

In The Last Decade

Kate E. Lawlor

61 papers receiving 5.6k citations

Hit Papers

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What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

2020 Cell Reports
2017 Proceedings of the National Academy of Sciences
2015 Nature Communications
2005 Nature

Peers

Countries citing papers authored by Kate E. Lawlor

Since Specialization

Citations

This map shows the geographic impact of Kate E. Lawlor's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Kate E. Lawlor with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Kate E. Lawlor more than expected).

Fields of papers citing papers by Kate E. Lawlor

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Kate E. Lawlor. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Kate E. Lawlor. The network helps show where Kate E. Lawlor may publish in the future.

Co-authorship network

The 25 scholars most cited alongside Kate E. Lawlor, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Kate E. Lawlor Line = papers co-authored together Kate E. Lawlor links everyone, so they are left out of the graph.

All Works

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20 of 20 papers shown

#	Work
1	Gene therapy enhances deoxyribonuclease I treatment in antimyeloperoxidase glomerulonephritis JCI Insight ·堀合真一,A. Ladhar,G. Hütsi,Maliha A. Alikhan,Sven Scheil,Lu Lu,Kate E. Lawlor,庄大军,Grymalowski Mr,Ian E. Alexander,A. Richard Kitching,Poh‐Yi Gan,Grant J. Logan,Kim M. O’Sullivan	2025	0
2	NLRP3 inflammasome–driven hemophagocytic lymphohistiocytosis occurs independent of IL-1β and IL-18 and is targetable by BET inhibitors Science Advances ·Kevin Meisner,Muhamad Kusuma Minarta,Anupriya Jain,Xiuquan Ma,James Rickard,Barbara Leonard Gibson,Catherine N. Hall,Andrew J. Kueh,Seth L. Masters,Inmaculada Rioja,Rab K. Prinjha,Marcel Doerflinger,Kate E. Lawlor,Maryam Rashidi,James E. Vince	2025	0
3	Get with the program: regulation of T cell death Trends in Immunology ·Timothy Patton,F. Armero,R. DeSaro,Kate E. Lawlor,Alexandra J. Corbett	2025	0
4	MLKL deficiency elevates testosterone production in male mice independently of necroptotic functions Cell Death and Disease ·P. Wester,Wayne Cawthorne,Suedja Maria Pascoal,中森邦晴,L. O. Androsova,Sarah E. Garnish,Manas Kumar Mallick,Cathrine Hall,Joanne M. Hildebrand,Malcolm J. McConville,Kate E. Lawlor,Edwin D. Hawkins,André L. Samson,James M. Murphy	2024	0
5	Discordance in STING-Induced Activation and Cell Death Between Mouse and Human Dendritic Cell Populations Frontiers in Immunology ·Ee Shan Pang,A.N. Rusakov,Katherine R. Balka,Dominic De Nardo,Christophe Macri,Hubertus Hochrein,Kelly‐Anne Masterman,Peck Szee Tan,Neil Griffiths,Jiahong Zhao,Kinshi NAGAE,Vishal Rathi,Yifan Zhan,Benjamin T. Kile,Kate E. Lawlor,Kristen J. Radford,Mark D. Wright,Meredith O’Keeffe	2022	17
6	TBK1 and IKK epsilon Act Redundantly to Mediate STING-Induced NF-kappa B Responses in Myeloid Cells Cell Reports ·Katherine R. Balka,Cynthia Louis,Tahnee L. Saunders,Amber M. Smith,Dale J. Calleja,Damian B. D’Silva,Fiona Moghaddas,Maximilien Tailler,Kate E. Lawlor,Yifan Zhan,Christopher J. Burns,Ian P. Wicks,Jonathan J. Miner,Benjamin T. Kile,Seth L. Masters,Dominic De Nardo	2020	10
7	The pro-survival Bcl-2 family member A1 delays spontaneous and FAS ligand-induced apoptosis of activated neutrophils Cell Death and Disease ·Robyn L. Schenk,Lahiru Gangoda,Kate E. Lawlor,Lorraine A. O’Reilly,Andreas Strasser,Marco J. Herold	2020	10
8	Macrophages, rather than DCs, are responsible for inflammasome activity in the GM-CSF BMDC model Nature Immunology ·Ziv Erlich,Inbar Shlomovitz,Liat Edry‐Botzer,Hadar Cohen,Daniel Frank,Hanqing Wang,Andrew M. Lew,Kate E. Lawlor,Yifan Zhan,James E. Vince,Motti Gerlic	2019	82
9	Active MLKL triggers the NLRP3 inflammasome in a cell-intrinsic mannerbreakdown → Proceedings of the National Academy of Sciences ·Stephanie A. Conos,Kaiwen Chen,Dominic De Nardo,Hideki Hara,Lachlan Whitehead,Gabriel Núñez,Seth L. Masters,James M. Murphy,Kate Schroder,David L. Vaux,Kate E. Lawlor,Lisa Lindqvist,James E. Vince	2017	377
10	XIAP Loss Triggers RIPK3- and Caspase-8-Driven IL-1β Activation and Cell Death as a Consequence of TLR-MyD88-Induced cIAP1-TRAF2 Degradation Cell Reports ·Kate E. Lawlor,Rebecca Feltham,Monica Yabal,Stephanie A. Conos,Kaiwen Chen,Zhang Lianzen,Carina Graß,Yifan Zhan,Tan Nguyen,Cathrine Hall,Hye-Jin Kim,Simon Chatfield,Damian B. D’Silva,Ken C. Pang,Kate Schroder,John Silke,David L. Vaux,Philipp J. Jost,James E. Vince	2017	105
11	New insights into the regulation of innate immunity by caspase-8 Arthritis Research & Therapy ·Vitaliya Sagulenko,Kate E. Lawlor,James E. Vince	2016	12
12	Eliminating Legionella by inhibiting BCL-XL to induce macrophage apoptosis Nature Microbiology ·Mary Speir,Kate E. Lawlor,Stefan Glaser,Yuan Ling,Seong Hoong Chow,Obando Ulloa Javier Mauricio,Keith E. Schulze,Ralf Schuelein,Lorraine A. O’Reilly,Kylie D. Mason,Elizabeth L. Hartland,Trevor Lithgow,Andreas Strasser,Guillaume Lessène,David C.S. Huang,James E. Vince,Thomas Naderer	2016	57
13	Distinctive pro-inflammatory gene signatures induced in articular chondrocytes by oncostatin M and IL-6 are regulated by Suppressor of Cytokine Signaling-3 Osteoarthritis and Cartilage ·Xiao Liu,Ruijie Liu,Ben A. Croker,Kate E. Lawlor,Gordon K. Smyth,Ian P. Wicks	2015	15
14	Loss of Bak enhances lymphocytosis but does not ameliorate thrombocytopaenia in BCL-2 transgenic mice Cell Death and Differentiation ·Cassandra J. Vandenberg,Emma C. Josefsson,Kirsteen J. Campbell,Chloé James,Kate E. Lawlor,Benjamin T. Kile,Suzanne Cory	2014	14
15	Ambiguities in NLRP3 inflammasome regulation: Is there a role for mitochondria? Biochimica et Biophysica Acta (BBA) - General Subjects ·Kate E. Lawlor,James E. Vince	2013	99
16	Distinct cell-specific control of autoimmunity and infection by FcγRIIb The Journal of Experimental Medicine ·Rebecca J. Brownlie,Kate E. Lawlor,Heather Niederer,Antony J. Cutler,Zou Xiang,Menna R. Clatworthy,R. Andrés Floto,David R. Greaves,Paul Lyons,Kenneth G. C. Smith	2008	129
17	FcγRIIb controls bone marrow plasma cell persistence and apoptosis Nature Immunology ·Zou Xiang,Antony J. Cutler,Rebecca J. Brownlie,Kirsten A. Fairfax,Kate E. Lawlor,Eva Severinson,Jiaran Sun,Rudolf A. Manz,David M. Tarlinton,Kenneth G. C. Smith	2007	239
18	Critical role for granulocyte colony-stimulating factor in inflammatory arthritis Proceedings of the National Academy of Sciences ·Kate E. Lawlor,Ian K. Campbell,Donald Metcalf,Kristy O’Donnell,Annemarie van Nieuwenhuijze,Andrew W. Roberts,Ian P. Wicks	2004	106
19	Molecular and cellular mediators of interleukin-1-dependent acute inflammatory arthritis Arthritis & Rheumatism ·Kate E. Lawlor,Ian K. Campbell,Kristy O’Donnell,Li Wu,Ian P. Wicks	2001	49
20	Initiation of remote hepatic injury in the rat: Interactions between Kupffer cells, tumor necrosis factor-α, and microvascular perfusion Hepatology ·Byung-Sun 방병선Bang,Kate E. Lawlor,Kenneth A. Harris,Richard F. Potter	1999	45

About Kate E. Lawlor

Kate E. Lawlor is a scholar working on Immunology, Immunology and Allergy, Molecular Biology, Rheumatology and Endocrinology, having authored 67 papers that have together received 5.7k indexed citations. Recurring topics across this work include Inflammasome and immune disorders (29 papers), Immune Response and Inflammation (20 papers), Cell death mechanisms and regulation (17 papers), interferon and immune responses (13 papers), Cell Adhesion Molecules Research (9 papers), Phagocytosis and Immune Regulation (8 papers), Immune Cell Function and Interaction (7 papers) and Cytokine Signaling Pathways and Interactions (7 papers). The work is most often cited by research in Immunology (2.6k citations), Rheumatology (882 citations), Immunology and Allergy (311 citations), Molecular Biology (3.3k citations) and Cancer Research (671 citations). Kate E. Lawlor has collaborated with scholars based in Australia, United States and Switzerland. Frequent co-authors include James E. Vince, Ian P. Wicks, David L. Vaux, Ian K. Campbell, Amanda Fosang, Christopher B. Little, James M. Murphy, Seth L. Masters, Pamela J. Farmer and Suzanne B. Golub. Their work appears in journals such as Cell Reports, Journal of Clinical Investigation, Cell Death and Disease, Cell Death and Differentiation and Journal of Leukocyte Biology.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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