Nufail Khan

2.1k total citations · 2 hit papers
11 papers, 1.6k citations indexed

About

Nufail Khan is a scholar working on Molecular Biology, Immunology and Cancer Research. According to data from OpenAlex, Nufail Khan has authored 11 papers receiving a total of 1.6k indexed citations (citations by other indexed papers that have themselves been cited), including 9 papers in Molecular Biology, 7 papers in Immunology and 5 papers in Cancer Research. Recurrent topics in Nufail Khan's work include Cell death mechanisms and regulation (9 papers), NF-κB Signaling Pathways (5 papers) and Phagocytosis and Immune Regulation (4 papers). Nufail Khan is often cited by papers focused on Cell death mechanisms and regulation (9 papers), NF-κB Signaling Pathways (5 papers) and Phagocytosis and Immune Regulation (4 papers). Nufail Khan collaborates with scholars based in Australia, Switzerland and Germany. Nufail Khan's co-authors include James E. Vince, John Silke, David L. Vaux, W. Wei‐Lynn Wong, Stephen M. Condon, Christopher A. Benetatos, Rebecca Feltham, Bernard A. Callus, Diep Chau and Srinivas K. Chunduru and has published in prestigious journals such as Cell, Journal of Biological Chemistry and Nature Communications.

In The Last Decade

Nufail Khan

10 papers receiving 1.6k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

IAP Antagonists Target cIAP1 to Induce TNFα-Dependent Apoptosis

2007 879 citations James E. Vince, W. Wei‐Lynn Wong et al. Cell profile →
RIPK3 promotes cell death and NLRP3 inflammasome activation in the absence of MLKL

2015 510 citations Kate E. Lawlor, Nufail Khan et al. Nature Communications profile →

Peers

Countries citing papers authored by Nufail Khan

Since Specialization

Citations

This map shows the geographic impact of Nufail Khan's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Nufail Khan with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Nufail Khan more than expected).

Fields of papers citing papers by Nufail Khan

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Nufail Khan. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Nufail Khan. The network helps show where Nufail Khan may publish in the future.

Co-authorship network of co-authors of Nufail Khan

This figure shows the co-authorship network connecting the top 25 collaborators of Nufail Khan. A scholar is included among the top collaborators of Nufail Khan based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Nufail Khan. Nufail Khan is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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11 of 11 papers shown

#	Work	Indexed citations
1	Combination of IAP antagonist and IFNγ activates novel caspase-10- and RIPK1-dependent cell death pathways 2017 ·Cell Death and Differentiation ·Maria C. Tanzer, Nufail Khan, James Rickard, Nima Etemadi, Najoua Lalaoui, Sukhdeep K. Spall, Joanne M. Hildebrand, David Segal, Maria Miasari, Diep Chau, W. Wei‐Lynn Wong, Mark A. McKinlay, Srinivas K. Chunduru, Christopher A. Benetatos, Stephen M. Condon, James E. Vince, Marco J. Herold, John Silke	38
2	In TNF-stimulated cells, RIPK1 promotes cell survival by stabilizing TRAF2 and cIAP1, which limits induction of non-canonical NF-κB and activation of caspase-8. 2016 ·Journal of Biological Chemistry ·Ian E. Gentle, W. Wei‐Lynn Wong, (unknown), (unknown), Wendy D. Cook, Nufail Khan, Ueli Nachbur, (unknown), Holly Anderton, Maryline Moulin, Josep M. Lluis, Donia M. Moujalled, John Silke, David L. Vaux	4
3	RIPK3 promotes cell death and NLRP3 inflammasome activation in the absence of MLKL breakdown → 2015 ·Nature Communications ·Kate E. Lawlor, Nufail Khan, (unknown), Motti Gerlic, Ben A. Croker, Akshay A. D’Cruz, Cathrine Hall, Sukhdeep K. Spall, Holly Anderton, Seth L. Masters, Maryam Rashidi, Ian P. Wicks, Warren S. Alexander, Yasuhiro Mitsuuchi, Christopher A. Benetatos, Stephen M. Condon, W. Wei‐Lynn Wong, John Silke, David L. Vaux, James E. Vince	510
4	179 2014 ·Cytokine ·Maria C. Tanzer, Nufail Khan, James Rickard, Nima Etemadi, John Silke	0
5	More to life than death: molecular determinants of necroptotic and non-necroptotic RIP3 kinase signaling 2013 ·Current Opinion in Immunology ·Nufail Khan, Kate E. Lawlor, James M. Murphy, James E. Vince	63
6	IAPS and ubiquitylation 2012 ·IUBMB Life ·Rebecca Feltham, Nufail Khan, John Silke	17
7	In mouse embryonic fibroblasts, neither caspase-8 nor cellular FLICE-inhibitory protein (FLIP) is necessary for TNF to activate NF-κB, but caspase-8 is required for TNF to cause cell death, and induction of FLIP by NF-κB is required to prevent it 2011 ·Cell Death and Differentiation ·Donia M. Moujalled, W D Cook, Josep M. Lluis, Nufail Khan, Afsar U. Ahmed, Bernard A. Callus, David L. Vaux	15
8	In TNF-stimulated Cells, RIPK1 Promotes Cell Survival by Stabilizing TRAF2 and cIAP1, which Limits Induction of Non-canonical NF-κB and Activation of Caspase-8 2011 ·Journal of Biological Chemistry ·Ian E. Gentle, W. Wei‐Lynn Wong, (unknown), (unknown), Wendy D. Cook, Nufail Khan, Ueli Nachbur, James Rickard, Holly Anderton, Maryline Moulin, Josep M. Lluis, Donia M. Moujalled, John Silke, David L. Vaux	81
9	TAK1 Is Required for Survival of Mouse Fibroblasts Treated with TRAIL, and Does So by NF-κB Dependent Induction of cFLIPL 2010 ·PLoS ONE ·Josep M. Lluis, Ueli Nachbur, Wendy D. Cook, Ian E. Gentle, Donia M. Moujalled, Maryline Moulin, W. Wei‐Lynn Wong, Nufail Khan, Diep Chau, Bernard A. Callus, James E. Vince, John Silke, David L. Vaux	19
10	Correction: TAK1 Is Required for Survival of Mouse Fibroblasts Treated with TRAIL, and Does So by NF-κB Dependent Induction of cFLIPL 2010 ·PLoS ONE ·Josep M. Lluis, (unknown), Wendy D. Cook, Ian E. Gentle, Donia M. Moujalled, Maryline Moulin, W. Wei‐Lynn Wong, Nufail Khan, Diep Chau, Bernard A. Callus, James E. Vince, John Silke, David L. Vaux	2
11	IAP Antagonists Target cIAP1 to Induce TNFα-Dependent Apoptosis breakdown → 2007 ·Cell ·James E. Vince, W. Wei‐Lynn Wong, Nufail Khan, Rebecca Feltham, Diep Chau, Afsar U. Ahmed, Christopher A. Benetatos, Srinivas K. Chunduru, Stephen M. Condon, Mark A. McKinlay, Robert Brink, Martin Leverkus, Vinay Tergaonkar, Pascal Schneider, Bernard A. Callus, Frank Köentgen, David L. Vaux, John Silke	879

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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