W. Wei‐Lynn Wong

11.0k total citations · 4 hit papers
71 papers, 8.0k citations indexed

About

W. Wei‐Lynn Wong is a scholar working on Immunology, Molecular Biology and Cancer Research. According to data from OpenAlex, W. Wei‐Lynn Wong has authored 71 papers receiving a total of 8.0k indexed citations (citations by other indexed papers that have themselves been cited), including 43 papers in Immunology, 39 papers in Molecular Biology and 22 papers in Cancer Research. Recurrent topics in W. Wei‐Lynn Wong's work include Cell death mechanisms and regulation (20 papers), NF-κB Signaling Pathways (17 papers) and Immune Response and Inflammation (13 papers). W. Wei‐Lynn Wong is often cited by papers focused on Cell death mechanisms and regulation (20 papers), NF-κB Signaling Pathways (17 papers) and Immune Response and Inflammation (13 papers). W. Wei‐Lynn Wong collaborates with scholars based in United States, Switzerland and Australia. W. Wei‐Lynn Wong's co-authors include John Silke, Linda Z. Penn, Jim Dimitroulakos, David L. Vaux, Holly Anderton, Douglas T. Fearon, James E. Vince, Ueli Nachbur, Lloyd B. Klickstein and Stephen M. Condon and has published in prestigious journals such as Nature, Cell and Proceedings of the National Academy of Sciences.

In The Last Decade

W. Wei‐Lynn Wong

69 papers receiving 7.8k citations

Hit Papers

IAP Antagonists Target cIAP1 to Induce TNFα-Dependent... 1995 2026 2005 2015 2007 2011 1995 2015 250 500 750
What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

  1. IAP Antagonists Target cIAP1 to Induce TNFα-Dependent Apoptosis
    2007 879 citations James E. Vince, W. Wei‐Lynn Wong et al. Cell profile →
  2. Linear ubiquitination prevents inflammation and regulates immune signalling
    2011 725 citations James Rickard, Holly Anderton et al. profile →
  3. Proteolytic activation of protein kinase C delta by an ICE-like protease in apoptotic cells.
    1995 612 citations W. Wei‐Lynn Wong et al. profile →
  4. RIPK3 promotes cell death and NLRP3 inflammasome activation in the absence of MLKL
    2015 510 citations Nufail Khan, Holly Anderton et al. profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
W. Wei‐Lynn Wong United States 39 5.2k 3.7k 2.3k 1.3k 745 71 8.0k
Peter J. Gough United States 45 6.0k 1.2× 4.4k 1.2× 1.4k 0.6× 1.5k 1.2× 1.2k 1.6× 80 9.6k
Nils Holler Switzerland 20 5.0k 1.0× 3.9k 1.1× 1.5k 0.7× 1.1k 0.9× 917 1.2× 20 8.3k
Lorraine A. O’Reilly Australia 50 5.5k 1.1× 3.9k 1.1× 990 0.4× 1.6k 1.2× 1.3k 1.7× 118 9.7k
Nathalie Droin France 39 4.2k 0.8× 2.7k 0.7× 1.2k 0.5× 1.9k 1.5× 802 1.1× 109 7.8k
Manuel O. Landázuri Spain 47 2.7k 0.5× 2.8k 0.8× 1.8k 0.8× 900 0.7× 395 0.5× 109 6.9k
Yuji Hinoda Japan 56 6.5k 1.3× 1.8k 0.5× 2.7k 1.2× 3.0k 2.4× 528 0.7× 272 10.3k
Hailing Hsu United States 17 4.8k 0.9× 4.2k 1.2× 3.1k 1.4× 1.7k 1.3× 667 0.9× 23 8.3k
Bryant G. Darnay United States 40 4.5k 0.9× 2.4k 0.7× 2.9k 1.3× 1.7k 1.3× 474 0.6× 65 6.9k
Veronika Sexl Austria 51 3.3k 0.6× 4.4k 1.2× 1.1k 0.5× 3.6k 2.8× 400 0.5× 183 9.5k
Julianna Blagih Canada 23 3.2k 0.6× 3.2k 0.9× 2.0k 0.9× 1.4k 1.1× 731 1.0× 29 6.8k

Countries citing papers authored by W. Wei‐Lynn Wong

Since Specialization
Citations

This map shows the geographic impact of W. Wei‐Lynn Wong's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by W. Wei‐Lynn Wong with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites W. Wei‐Lynn Wong more than expected).

Fields of papers citing papers by W. Wei‐Lynn Wong

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by W. Wei‐Lynn Wong. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by W. Wei‐Lynn Wong. The network helps show where W. Wei‐Lynn Wong may publish in the future.

Co-authorship network of co-authors of W. Wei‐Lynn Wong

This figure shows the co-authorship network connecting the top 25 collaborators of W. Wei‐Lynn Wong. A scholar is included among the top collaborators of W. Wei‐Lynn Wong based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with W. Wei‐Lynn Wong. W. Wei‐Lynn Wong is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Keller, Jennifer A., Takashi Satoh, Barbara Meier, et al.. (2025). LL37 complexed to double-stranded RNA induces RIG-I-like receptor signalling and Gasdermin E activation facilitating IL-36γ release from keratinocytes. Cell Death and Disease. 16(1). 198–198.
2.
3.
Tong, Jing, et al.. (2023). Measuring Caspase Activity Using a Fluorometric Assay or Flow Cytometry. Journal of Visualized Experiments. 1 indexed citations
4.
Kaplinsky, Joseph, Fredrik Ivars, William W. Agace, et al.. (2019). SMAC mimetics promote NIK-dependent inhibition of CD4 + T H 17 cell differentiation. Science Signaling. 12(596). 12 indexed citations
5.
Spilgies, Lisanne M., Monica Yabal, Erika Owsley, et al.. (2019). TNFR2 induced priming of the inflammasome leads to a RIPK1-dependent cell death in the absence of XIAP. Cell Death and Disease. 10(10). 56–56. 28 indexed citations
6.
Corazza, Nadia, et al.. (2018). Loss of BID Delays FASL-Induced Cell Death of Mouse Neutrophils and Aggravates DSS-Induced Weight Loss. International Journal of Molecular Sciences. 19(3). 684–684. 9 indexed citations
7.
Hänggi, Kay, Aı̈da Valls, Rosario Yerbes, et al.. (2017). RIPK1/RIPK3 promotes vascular permeability to allow tumor cell extravasation independent of its necroptotic function. Cell Death and Disease. 8(2). e2588–e2588. 62 indexed citations
8.
Wong, W. Wei‐Lynn, et al.. (2016). Loss of XIAP facilitates switch to TNFα-induced necroptosis in mouse neutrophils. Cell Death and Disease. 7(10). e2422–e2422. 70 indexed citations
9.
Gentle, Ian E., W. Wei‐Lynn Wong, Wendy D. Cook, et al.. (2011). In TNF-stimulated Cells, RIPK1 Promotes Cell Survival by Stabilizing TRAF2 and cIAP1, which Limits Induction of Non-canonical NF-κB and Activation of Caspase-8. Journal of Biological Chemistry. 286(15). 13282–13291. 81 indexed citations
10.
Feltham, Rebecca, Maryline Moulin, James E. Vince, et al.. (2010). Tumor Necrosis Factor (TNF) Signaling, but Not TWEAK (TNF-like Weak Inducer of Apoptosis)-triggered cIAP1 (Cellular Inhibitor of Apoptosis Protein 1) Degradation, Requires cIAP1 RING Dimerization and E2 Binding. Journal of Biological Chemistry. 285(23). 17525–17536. 36 indexed citations
11.
Geserick, Peter, Mike Hupe, Maryline Moulin, et al.. (2009). Cellular IAPs inhibit a cryptic CD95-induced cell death by limiting RIP1 kinase recruitment. The Journal of Cell Biology. 187(7). 1037–1054. 198 indexed citations
12.
Wong, W. Wei‐Lynn & Hamsa Puthalakath. (2008). Bcl‐2 family proteins: The sentinels of the mitochondrial apoptosis pathway. IUBMB Life. 60(6). 390–397. 152 indexed citations
13.
Wong, W. Wei‐Lynn, James W. Clendening, Աննա Մարտիրոսյան, et al.. (2007). Determinants of sensitivity to lovastatin-induced apoptosis in multiple myeloma. Molecular Cancer Therapeutics. 6(6). 1886–1897. 60 indexed citations
14.
Vince, James E., W. Wei‐Lynn Wong, Nufail Khan, et al.. (2007). IAP Antagonists Target cIAP1 to Induce TNFα-Dependent Apoptosis. Cell. 131(4). 682–693. 879 indexed citations breakdown →
15.
Molina, Hector, W. Wei‐Lynn Wong, Taroh Kinoshita, et al.. (1992). Distinct receptor and regulatory properties of recombinant mouse complement receptor 1 (CR1) and Crry, the two genetic homologues of human CR1.. The Journal of Experimental Medicine. 175(1). 121–129. 122 indexed citations
16.
Wong, W. Wei‐Lynn, Christine Kennedy, Margaret J. Morris, et al.. (1989). Structure of the human CR1 gene. Molecular basis of the structural and quantitative polymorphisms and identification of a new CR1-like allele.. The Journal of Experimental Medicine. 169(3). 847–863. 80 indexed citations
17.
Wilson, James G., Ellen Murphy, W. Wei‐Lynn Wong, et al.. (1986). Identification of a restriction fragment length polymorphism by a CR1 cDNA that correlates with the number of CR1 on erythrocytes.. The Journal of Experimental Medicine. 164(1). 50–59. 143 indexed citations
18.
Wong, W. Wei‐Lynn, Christine Kennedy, James G. Wilson, et al.. (1986). Analysis of multiple restriction fragment length polymorphisms of the gene for the human complement receptor type I. Duplication of genomic sequences occurs in association with a high molecular mass receptor allotype.. The Journal of Experimental Medicine. 164(5). 1531–1546. 31 indexed citations
19.
Weis, Janis J., Douglas T. Fearon, Lloyd B. Klickstein, et al.. (1986). Identification of a partial cDNA clone for the C3d/Epstein-Barr virus receptor of human B lymphocytes: homology with the receptor for fragments C3b and C4b of the third and fourth components of complement.. Proceedings of the National Academy of Sciences. 83(15). 5639–5643. 58 indexed citations
20.
Nazar, Ross N. & W. Wei‐Lynn Wong. (1985). Is the 5S RNA a primitive ribosomal sequence?. Proceedings of the National Academy of Sciences. 82(17). 5608–5611. 5 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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