Peter D. Mace

3.4k total citations
57 papers, 2.4k citations indexed

About

Peter D. Mace is a scholar working on Molecular Biology, Oncology and Cell Biology. According to data from OpenAlex, Peter D. Mace has authored 57 papers receiving a total of 2.4k indexed citations (citations by other indexed papers that have themselves been cited), including 51 papers in Molecular Biology, 15 papers in Oncology and 13 papers in Cell Biology. Recurrent topics in Peter D. Mace's work include Ubiquitin and proteasome pathways (17 papers), Cell death mechanisms and regulation (15 papers) and Protein Kinase Regulation and GTPase Signaling (8 papers). Peter D. Mace is often cited by papers focused on Ubiquitin and proteasome pathways (17 papers), Cell death mechanisms and regulation (15 papers) and Protein Kinase Regulation and GTPase Signaling (8 papers). Peter D. Mace collaborates with scholars based in New Zealand, United States and Australia. Peter D. Mace's co-authors include Catherine L. Day, Stefan J. Riedl, John Silke, David L. Vaux, James M. Murphy, Bernhard C. Lechtenberg, Clyde A. Smith, Katrin Linke, Rebecca Feltham and Sarah Shirley and has published in prestigious journals such as Nature, Science and Proceedings of the National Academy of Sciences.

In The Last Decade

Peter D. Mace

55 papers receiving 2.4k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Peter D. Mace New Zealand 27 2.0k 579 532 351 318 57 2.4k
Laurence Dubrez France 28 2.2k 1.1× 645 1.1× 561 1.1× 335 1.0× 267 0.8× 51 2.9k
Min-Jung Lee United States 28 1.8k 0.9× 663 1.1× 392 0.7× 234 0.7× 175 0.6× 96 2.8k
Howard O. Fearnhead Ireland 26 2.0k 1.0× 489 0.8× 496 0.9× 327 0.9× 235 0.7× 53 2.6k
Jean‐Bernard Denault Canada 25 1.7k 0.9× 341 0.6× 386 0.7× 268 0.8× 364 1.1× 49 2.3k
Shu‐ichi Matsuzawa United States 29 1.9k 0.9× 528 0.9× 664 1.2× 404 1.2× 335 1.1× 65 2.6k
Diego Miranda‐Saavedra United Kingdom 28 1.8k 0.9× 401 0.7× 582 1.1× 175 0.5× 317 1.0× 43 2.8k
Hwain Shin United States 10 2.0k 1.0× 407 0.7× 521 1.0× 326 0.9× 264 0.8× 12 2.4k
Ingo H. Engels Germany 19 1.9k 0.9× 411 0.7× 477 0.9× 248 0.7× 257 0.8× 23 2.7k
Stéphanie Plenchette France 22 1.7k 0.9× 465 0.8× 622 1.2× 472 1.3× 217 0.7× 34 2.3k

Countries citing papers authored by Peter D. Mace

Since Specialization
Citations

This map shows the geographic impact of Peter D. Mace's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Peter D. Mace with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Peter D. Mace more than expected).

Fields of papers citing papers by Peter D. Mace

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Peter D. Mace. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Peter D. Mace. The network helps show where Peter D. Mace may publish in the future.

Co-authorship network of co-authors of Peter D. Mace

This figure shows the co-authorship network connecting the top 25 collaborators of Peter D. Mace. A scholar is included among the top collaborators of Peter D. Mace based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Peter D. Mace. Peter D. Mace is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Burke, John E., et al.. (2025). DET1 dynamics underlie cooperative ubiquitination by CRL4 DET1-COP1 complexes. Science Advances. 11(9). eadq4187–eadq4187. 2 indexed citations
2.
Currie, Michael, Mariafrancesca Scalise, J. D. Wright, et al.. (2024). Structural and biophysical analysis of a Haemophilus influenzae tripartite ATP-independent periplasmic (TRAP) transporter. eLife. 12. 3 indexed citations
3.
Currie, Michael, Mariafrancesca Scalise, J. D. Wright, et al.. (2023). Structural and biophysical analysis of a Haemophilus influenzae tripartite ATP-independent periplasmic (TRAP) transporter. eLife. 12. 4 indexed citations
4.
Currie, Michael, Rachel A. North, Mariafrancesca Scalise, et al.. (2023). Structure and mechanism of a tripartite ATP-independent periplasmic TRAP transporter. Nature Communications. 14(1). 1120–1120. 18 indexed citations
5.
Middleton, A.J., et al.. (2022). Ubiquitin and a charged loop regulate the ubiquitin E3 ligase activity of Ark2C. Nature Communications. 13(1). 1181–1181. 15 indexed citations
6.
Petley, Emma V., Kathryn J. Farrand, Wanting Jiao, et al.. (2020). The Synthesis and Anti‐tumour Properties of Poly Ethoxy Ethyl Glycinamide (PEE−G) Scaffolds with Multiple PD‐1 Peptides Attached. ChemMedChem. 15(13). 1128–1138. 4 indexed citations
7.
Parker, Benjamin L., et al.. (2020). Structure-based mechanism of preferential complex formation by apoptosis signal–regulating kinases. Science Signaling. 13(622). 20 indexed citations
8.
Chen, Shiyu, S. Lovell, Sumin Lee, et al.. (2020). Identification of highly selective covalent inhibitors by phage display. Nature Biotechnology. 39(4). 490–498. 91 indexed citations
9.
Morgan, Benjamin J., Thomas W.R. Harrop, George Taiaroa, et al.. (2019). Nonsynonymous SNPs in LPA homologous to plasminogen deficiency mutants represent novel null apo(a) alleles. Journal of Lipid Research. 61(3). 432–444. 14 indexed citations
10.
Middleton, A.J., et al.. (2018). A bidentate Polycomb Repressive-Deubiquitinase complex is required for efficient activity on nucleosomes. Nature Communications. 9(1). 3932–3932. 25 indexed citations
11.
Ruan, Zheng, et al.. (2018). Substrate binding allosterically relieves autoinhibition of the pseudokinase TRIB1. Science Signaling. 11(549). 46 indexed citations
12.
Wright, J. D., Peter D. Mace, & Catherine L. Day. (2016). Noncovalent Ubiquitin Interactions Regulate the Catalytic Activity of Ubiquitin Writers. Trends in Biochemical Sciences. 41(11). 924–937. 25 indexed citations
13.
Lechtenberg, Bernhard C., Akhil Rajput, Ruslan Sanishvili, et al.. (2016). Structure of a HOIP/E2~ubiquitin complex reveals RBR E3 ligase mechanism and regulation. Nature. 529(7587). 546–550. 139 indexed citations
14.
Wright, J. D., Peter D. Mace, & Catherine L. Day. (2015). Secondary ubiquitin-RING docking enhances Arkadia and Ark2C E3 ligase activity. Nature Structural & Molecular Biology. 23(1). 45–52. 49 indexed citations
15.
Lechtenberg, Bernhard C., Peter D. Mace, & Stefan J. Riedl. (2014). Structural mechanisms in NLR inflammasome signaling. Current Opinion in Structural Biology. 29. 17–25. 91 indexed citations
16.
Mace, Peter D., Stefan J. Riedl, & Guy S. Salvesen. (2014). Caspase Enzymology and Activation Mechanisms. Methods in enzymology on CD-ROM/Methods in enzymology. 544. 161–178. 26 indexed citations
17.
Feltham, Rebecca, Maryline Moulin, James E. Vince, et al.. (2010). Tumor Necrosis Factor (TNF) Signaling, but Not TWEAK (TNF-like Weak Inducer of Apoptosis)-triggered cIAP1 (Cellular Inhibitor of Apoptosis Protein 1) Degradation, Requires cIAP1 RING Dimerization and E2 Binding. Journal of Biological Chemistry. 285(23). 17525–17536. 36 indexed citations
18.
Mace, Peter D. & Stefan J. Riedl. (2010). Molecular cell death platforms and assemblies. Current Opinion in Cell Biology. 22(6). 828–836. 61 indexed citations
19.
Mace, Peter D., Katrin Linke, Rebecca Feltham, et al.. (2008). Structures of the cIAP2 RING Domain Reveal Conformational Changes Associated with Ubiquitin-conjugating Enzyme (E2) Recruitment. Journal of Biological Chemistry. 283(46). 31633–31640. 137 indexed citations
20.
Linke, Katrin, Peter D. Mace, Clyde A. Smith, et al.. (2008). Structure of the MDM2/MDMX RING domain heterodimer reveals dimerization is required for their ubiquitylation in trans. Cell Death and Differentiation. 15(5). 841–848. 231 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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