Wolfgang Liedtke

20.5k total citations · 7 hit papers
160 papers, 15.8k citations indexed

About

Wolfgang Liedtke is a scholar working on Sensory Systems, Physiology and Molecular Biology. According to data from OpenAlex, Wolfgang Liedtke has authored 160 papers receiving a total of 15.8k indexed citations (citations by other indexed papers that have themselves been cited), including 87 papers in Sensory Systems, 49 papers in Physiology and 36 papers in Molecular Biology. Recurrent topics in Wolfgang Liedtke's work include Ion Channels and Receptors (86 papers), Pain Mechanisms and Treatments (19 papers) and Ion channel regulation and function (17 papers). Wolfgang Liedtke is often cited by papers focused on Ion Channels and Receptors (86 papers), Pain Mechanisms and Treatments (19 papers) and Ion channel regulation and function (17 papers). Wolfgang Liedtke collaborates with scholars based in United States, Germany and Canada. Wolfgang Liedtke's co-authors include Farshid Guilak, Jeffrey M. Friedman, Jeffrey M. Gimble, Daniel M. Cohen, Bradley T. Estes, Christopher S. Chen, Holly A. Leddy, Yong Chen, Marc A. Martı́-Renom and Yong Choe and has published in prestigious journals such as Science, Cell and Proceedings of the National Academy of Sciences.

In The Last Decade

Wolfgang Liedtke

159 papers receiving 15.6k citations

Hit Papers

Control of Stem Cell Fate by Physical Interactions with t... 2000 2026 2008 2017 2009 2000 2002 2003 2012 400 800 1.2k

Peers

Wolfgang Liedtke
Martin Koltzenburg United Kingdom
Nigel W. Bunnett United States
Brian M. Davis United States
Y. S. Prakash United States
Susan D. Brain United Kingdom
Gary C. Sieck United States
Martin Koltzenburg United Kingdom
Wolfgang Liedtke
Citations per year, relative to Wolfgang Liedtke Wolfgang Liedtke (= 1×) peers Martin Koltzenburg

Countries citing papers authored by Wolfgang Liedtke

Since Specialization
Citations

This map shows the geographic impact of Wolfgang Liedtke's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Wolfgang Liedtke with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Wolfgang Liedtke more than expected).

Fields of papers citing papers by Wolfgang Liedtke

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Wolfgang Liedtke. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Wolfgang Liedtke. The network helps show where Wolfgang Liedtke may publish in the future.

Co-authorship network of co-authors of Wolfgang Liedtke

This figure shows the co-authorship network connecting the top 25 collaborators of Wolfgang Liedtke. A scholar is included among the top collaborators of Wolfgang Liedtke based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Wolfgang Liedtke. Wolfgang Liedtke is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Zhang, Qiaojuan, Minji Jang, Qian Zeng, et al.. (2025). Neuronal Mechanisms of Psoriatic Itch: Role of IL-17R/ERK/TRPV4 Signaling Pathway. Journal of Investigative Dermatology. 145(11). 2753–2762.e3. 2 indexed citations
2.
Ye, Yun, et al.. (2024). Epidemiology of myasthenia gravis in the United States. Frontiers in Neurology. 15. 1339167–1339167. 12 indexed citations
3.
Li, Mei, Sabrina Schulz, Juliana Falivene, et al.. (2024). Calcium-activated Potassium Channels as Amplifiers of TRPV4-mediated Pulmonary Edema Formation in Male Mice. Anesthesiology. 141(5). 913–928. 2 indexed citations
4.
Kamermans, Alwin, Bert van het Hof, Ruud D. Fontijn, et al.. (2024). Inflammation-induced TRPV4 channels exacerbate blood–brain barrier dysfunction in multiple sclerosis. Journal of Neuroinflammation. 21(1). 72–72. 18 indexed citations
5.
Llorente, Itzel, Ariela Vergara‐Jaque, Marcelino Arciniega, et al.. (2023). Modes of action of lysophospholipids as endogenous activators of the TRPV4 ion channel. The Journal of Physiology. 601(9). 1655–1673. 13 indexed citations
6.
Dicks, Amanda, Grigory Maksaev, Alireza Savadipour, et al.. (2023). Skeletal dysplasia-causing TRPV4 mutations suppress the hypertrophic differentiation of human iPSC-derived chondrocytes. eLife. 12. 12 indexed citations
7.
Ouyang, Chen, Huaxin Sheng, Yikai Luo, et al.. (2023). A mast cell–thermoregulatory neuron circuit axis regulates hypothermia in anaphylaxis. Science Immunology. 8(81). eadc9417–eadc9417. 42 indexed citations
8.
Liang, Pengfei, Liheng Yang, Liping Feng, et al.. (2022). Functional coupling between TRPV4 channel and TMEM16F modulates human trophoblast fusion. eLife. 11. 25 indexed citations
9.
Nims, Robert J., Alireza Savadipour, Christopher J. O’Conor, et al.. (2021). A synthetic mechanogenetic gene circuit for autonomous drug delivery in engineered tissues. Science Advances. 7(5). 63 indexed citations
10.
Liedtke, Wolfgang, et al.. (2019). TRPV4 inhibition prevents increased water diffusion and blood-retina barrier breakdown in the retina of streptozotocin-induced diabetic mice. PLoS ONE. 14(5). e0212158–e0212158. 23 indexed citations
11.
Rodriguez, Erica, Katsuyasu Sakurai, Yong Chen, et al.. (2017). A craniofacial-specific monosynaptic circuit enables heightened affective pain. Nature Neuroscience. 20(12). 1734–1743. 133 indexed citations
12.
Lin, Mike T., Ming‐Yuan Jian, Mark S. Taylor, et al.. (2015). Functional Coupling of TRPV4, IK, and SK Channels Contributes to Ca2+‐Dependent Endothelial Injury in Rodent Lung. Pulmonary Circulation. 5(2). 279–290. 34 indexed citations
13.
O’Conor, Christopher J., Holly A. Leddy, Halei C. Benefield, Wolfgang Liedtke, & Farshid Guilak. (2014). TRPV4-mediated mechanotransduction regulates the metabolic response of chondrocytes to dynamic loading. Proceedings of the National Academy of Sciences. 111(4). 1316–1321. 366 indexed citations breakdown →
14.
Lee, Whasil, Holly A. Leddy, Yong Chen, et al.. (2014). Synergy between Piezo1 and Piezo2 channels confers high-strain mechanosensitivity to articular cartilage. Proceedings of the National Academy of Sciences. 111(47). E5114–22. 352 indexed citations
15.
Moore, Carlene, Ferda Cevikbas, H. Amalia Pasolli, et al.. (2013). UVB radiation generates sunburn pain and affects skin by activating epidermal TRPV4 ion channels and triggering endothelin-1 signaling. Proceedings of the National Academy of Sciences. 110(34). E3225–34. 192 indexed citations
16.
Sonkusare, Swapnil K., Adrian D. Bonev, Jonathan Ledoux, et al.. (2012). Elementary Ca 2+ Signals Through Endothelial TRPV4 Channels Regulate Vascular Function. Science. 336(6081). 597–601. 441 indexed citations breakdown →
17.
Shukla, Arun K., Ji‐Hee Kim, Seungkirl Ahn, et al.. (2010). Arresting a Transient Receptor Potential (TRP) Channel. Journal of Biological Chemistry. 285(39). 30115–30125. 92 indexed citations
18.
Liedtke, Wolfgang, et al.. (2007). Expression of Transient Receptor Potential (TRP) Channels in the Outflow Pathway and Ciliary Body of Porcine Eyes. Investigative Ophthalmology & Visual Science. 48(13). 2066–2066. 1 indexed citations
19.
Liedtke, Wolfgang. (2006). Transient receptor potential vanilloid channels functioning in transduction of osmotic stimuli. Journal of Endocrinology. 191(3). 515–523. 64 indexed citations
20.
Liedtke, Wolfgang, et al.. (1998). Experimental autoimmune encephalomyelitis in mice lacking glial fibrillary acidic protein is characterized by a more severe clinical course and an infiltrative central nervous system lesion.. Europe PMC (PubMed Central). 152(1). 251–9. 114 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

Explore authors with similar magnitude of impact

Rankless by CCL
2026