Mark S. Taylor

1.6k total citations
44 papers, 1.3k citations indexed

About

Mark S. Taylor is a scholar working on Molecular Biology, Physiology and Cardiology and Cardiovascular Medicine. According to data from OpenAlex, Mark S. Taylor has authored 44 papers receiving a total of 1.3k indexed citations (citations by other indexed papers that have themselves been cited), including 22 papers in Molecular Biology, 22 papers in Physiology and 13 papers in Cardiology and Cardiovascular Medicine. Recurrent topics in Mark S. Taylor's work include Nitric Oxide and Endothelin Effects (21 papers), Ion Channels and Receptors (12 papers) and Ion channel regulation and function (12 papers). Mark S. Taylor is often cited by papers focused on Nitric Oxide and Endothelin Effects (21 papers), Ion Channels and Receptors (12 papers) and Ion channel regulation and function (12 papers). Mark S. Taylor collaborates with scholars based in United States, Canada and Japan. Mark S. Taylor's co-authors include Michael Francis, Mark T. Nelson, Adrian D. Bonev, Viktoriya Solodushko, Xun Qian, Joseph E. Brayden, Jonathan Ledoux, John P. Adelman, Chris T. Bond and Delrae M. Eckman and has published in prestigious journals such as Proceedings of the National Academy of Sciences, PLoS ONE and Circulation Research.

In The Last Decade

Mark S. Taylor

43 papers receiving 1.3k citations

Peers

Countries citing papers authored by Mark S. Taylor

Since Specialization

Citations

This map shows the geographic impact of Mark S. Taylor's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mark S. Taylor with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mark S. Taylor more than expected).

Fields of papers citing papers by Mark S. Taylor

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Mark S. Taylor. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mark S. Taylor. The network helps show where Mark S. Taylor may publish in the future.

Co-authorship network of co-authors of Mark S. Taylor

This figure shows the co-authorship network connecting the top 25 collaborators of Mark S. Taylor. A scholar is included among the top collaborators of Mark S. Taylor based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Mark S. Taylor. Mark S. Taylor is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

#	Work	Indexed citations
1	Algorithm for biological second messenger analysis with dynamic regions of interest 2023 ·PLoS ONE ·(unknown), (unknown), (unknown), (unknown), Thomas C. Rich, Mark S. Taylor, Joel Andrews, (unknown), C. Michael Francis	2
2	Ion Channel–Based Reporters for cAMP Detection 2022 ·Methods in molecular biology ·Thomas C. Rich, Wenkuan Xin, Silas J. Leavesley, C. Michael Francis, Mark S. Taylor	0
3	Restricted Intimal Ca2+ Signaling Associated With Cardiovascular Disease 2022 ·Frontiers in Physiology ·Mark S. Taylor, (unknown), Chung‐Sik Choi, Michael Francis	2
4	Augmented KCa2.3 Channel Feedback Regulation of Oxytocin Stimulated Uterine Strips from Nonpregnant Mice 2021 ·International Journal of Molecular Sciences ·(unknown), (unknown), Trudy L. Cornwell, Mark S. Taylor	4
5	Development of an endothelial cell-restricted transgenic reporter rat: a resource for physiological studies of vascular biology 2020 ·American Journal of Physiology-Heart and Circulatory Physiology ·Mikhail Alexeyev, Aron M. Geurts, (unknown), C. Michael Francis, Silas J. Leavesley, Thomas C. Rich, Mark S. Taylor, Mike T. Lin, Ron Balczon, (unknown), Diego F. Alvarez, Troy Stevens	4
6	Functional Coupling of TRPV4, IK, and SK Channels Contributes to Ca²⁺‐Dependent Endothelial Injury in Rodent Lung 2015 ·Pulmonary Circulation ·Mike T. Lin, Ming‐Yuan Jian, Mark S. Taylor, Donna L. Cioffi, (unknown), Wolfgang Liedtke, Mary I. Townsley	34
7	Channel-Based Reporters for cAMP Detection 2015 ·Methods in molecular biology ·Thomas C. Rich, Wenkuan Xin, Silas J. Leavesley, Mark S. Taylor	3
8	Developmental acceleration of bradykinin-dependent relaxation by prenatal chronic hypoxia impedes normal development after birth 2015 ·American Journal of Physiology-Lung Cellular and Molecular Physiology ·(unknown), (unknown), (unknown), (unknown), (unknown), (unknown), (unknown), Arlin B. Blood, Michael Francis, Mark S. Taylor, Lawrence D. Longo, William J. Pearce, Sean M. Wilson	13
9	Automated Analysis of Dynamic Ca<sup>2+</sup> Signals in Image Sequences 2014 ·Journal of Visualized Experiments ·Michael Francis, (unknown), Xun Qian, Mark S. Taylor	21
10	Decoding dynamic Ca2+ signaling in the vascular endothelium 2014 ·Frontiers in Physiology ·Mark S. Taylor, Michael Francis	21
11	TRPC4 Inactivation Confers a Survival Benefit in Severe Pulmonary Arterial Hypertension 2013 ·American Journal Of Pathology ·Abdallah Alzoubi, (unknown), Michie Toba, (unknown), Xun Qian, Michael Francis, Mark S. Taylor, Mikhail Alexeyev, Ivan F. McMurtry, Masahiko Oka, Troy Stevens	39
12	Dynamic Ca²⁺ Signal Modalities in the Vascular Endothelium 2012 ·Microcirculation ·Mark S. Taylor, Michael Francis, Xun Qian, Viktoriya Solodushko	11
13	Cell–Cell Communication in the Resistance Vasculature: The Past, Present, and Future 2012 ·Microcirculation ·Donald G. Welsh, Mark S. Taylor	4
14	Optical Recording Reveals Novel Properties of GSK1016790A-Induced Vanilloid Transient Receptor Potential Channel TRPV4 Activity in Primary Human Endothelial Cells 2012 ·Molecular Pharmacology ·Michelle N. Sullivan, Michael Francis, (unknown), Mark S. Taylor, Scott Earley	62
15	Endothelial Ca²⁺ wavelets and the induction of myoendothelial feedback 2012 ·American Journal of Physiology-Cell Physiology ·Cam Ha T. Tran, Mark S. Taylor, Frances Plane, Sridevi Nagaraja, Nikolaos M. Tsoukias, (unknown), Edward J. Vigmond, Tobias Fürstenhaupt, (unknown), Donald G. Welsh	99
16	Recruitment of Dynamic Endothelial Ca²⁺ Signals by the TRPA1 Channel Activator AITC in Rat Cerebral Arteries 2012 ·Microcirculation ·Xun Qian, Michael Francis, Viktoriya Solodushko, Scott Earley, Mark S. Taylor	64
17	Glucose-6-Phosphate Dehydrogenase Is a Regulator of Vascular Smooth Muscle Contraction 2010 ·Antioxidants and Redox Signaling ·Rakhee S. Gupte, (unknown), (unknown), (unknown), Mark S. Taylor, Rikuo Ochi, Sachin A. Gupte	32
18	Functional architecture of inositol 1,4,5-trisphosphate signaling in restricted spaces of myoendothelial projections 2008 ·Proceedings of the National Academy of Sciences ·Jonathan Ledoux, Mark S. Taylor, Adrian D. Bonev, Rachael M. Hannah, Viktoriya Solodushko, Bo Shui, Yvonne N. Tallini, Michael I. Kotlikoff, Mark T. Nelson	237
19	Inhibition of cGMP-Dependent Protein Kinase by the Cell-Permeable Peptide DT-2 Reveals a Novel Mechanism of Vasoregulation 2004 ·Molecular Pharmacology ·Mark S. Taylor, (unknown), Christian K. Nickl, Werner Tegge, Joseph E. Brayden, Wolfgang R. Dostmann	49
20	Analysis of shorthorn sculpin antifreeze protein stereospecific binding to (2–1 0) faces of ice 1996 ·Biophysical Journal ·Anthony S. Wierzbicki, Mark S. Taylor, Charles A. Knight, Jeffry D. Madura, John P. Harrington, C. Steven Sikes	62

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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