Matthew J. Rowling

1.8k total citations · 1 hit paper
29 papers, 1.4k citations indexed

About

Matthew J. Rowling is a scholar working on Physiology, Pathology and Forensic Medicine and Molecular Biology. According to data from OpenAlex, Matthew J. Rowling has authored 29 papers receiving a total of 1.4k indexed citations (citations by other indexed papers that have themselves been cited), including 17 papers in Physiology, 14 papers in Pathology and Forensic Medicine and 7 papers in Molecular Biology. Recurrent topics in Matthew J. Rowling's work include Vitamin D Research Studies (14 papers), Diet and metabolism studies (14 papers) and Folate and B Vitamins Research (5 papers). Matthew J. Rowling is often cited by papers focused on Vitamin D Research Studies (14 papers), Diet and metabolism studies (14 papers) and Folate and B Vitamins Research (5 papers). Matthew J. Rowling collaborates with scholars based in United States, France and Sweden. Matthew J. Rowling's co-authors include Kevin L. Schalinske, JoEllen Welsh, Elizabeth M. Whitley, Terri D. Boylston, Diane F. Birt, Samuel A. Moore, M. Paul Scott, John F. McClelland, Gregory J. Phillips and Suzanne Hendrich and has published in prestigious journals such as Journal of Biological Chemistry, PLoS ONE and Journal of Agricultural and Food Chemistry.

In The Last Decade

Matthew J. Rowling

28 papers receiving 1.4k citations

Hit Papers

Resistant Starch: Promise for Improving Human Health 2013 2026 2017 2021 2013 200 400 600
What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

  1. Resistant Starch: Promise for Improving Human Health
    2013 652 citations Diane F. Birt, Terri D. Boylston et al. Advances in Nutrition profile →

Peers

Matthew J. Rowling
Marie-Jeanne Davicco France
Sachie Ikegami Japan
Mette Axelsen Denmark
LU Thompson Canada
Atsutane Ohta Japan
Tianjing Liu China
Teresa Nestares Spain
Clara Ruiz‐Fernández Spain
Jameela Banu United States
Tatsuya Ohkawara Japan
Marie-Jeanne Davicco France
Matthew J. Rowling
Citations per year, relative to Matthew J. Rowling Matthew J. Rowling (= 1×) peers Marie-Jeanne Davicco

Countries citing papers authored by Matthew J. Rowling

Since Specialization
Citations

This map shows the geographic impact of Matthew J. Rowling's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Matthew J. Rowling with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Matthew J. Rowling more than expected).

Fields of papers citing papers by Matthew J. Rowling

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Matthew J. Rowling. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Matthew J. Rowling. The network helps show where Matthew J. Rowling may publish in the future.

Co-authorship network of co-authors of Matthew J. Rowling

This figure shows the co-authorship network connecting the top 25 collaborators of Matthew J. Rowling. A scholar is included among the top collaborators of Matthew J. Rowling based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Matthew J. Rowling. Matthew J. Rowling is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Day, Tim A., Michael J. Kimber, Rudy J. Valentine, et al.. (2020). Whole egg consumption increases gene expression within the glutathione pathway in the liver of Zucker Diabetic Fatty rats. PLoS ONE. 15(11). e0240885–e0240885. 3 indexed citations
2.
Rowling, Matthew J., et al.. (2020). Whole Egg Consumption Decreases Cumulative Weight Gain in Diet-Induced Obese Rats. Journal of Nutrition. 150(7). 1818–1823. 5 indexed citations
3.
Webb, J. L., et al.. (2019). Whole Egg Consumption Impairs Insulin Sensitivity in a Rat Model of Obesity and Type 2 Diabetes. Current Developments in Nutrition. 3(4). nzz015–nzz015. 4 indexed citations
4.
Webb, J. L., et al.. (2019). Dietary Whole Egg Reduces Body Weight Gain in a Dose-Dependent Manner in Zucker Diabetic Fatty Rats. Journal of Nutrition. 149(10). 1766–1775. 4 indexed citations
5.
6.
Koh, Gar Yee & Matthew J. Rowling. (2017). Resistant starch as a novel dietary strategy to maintain kidney health in diabetes mellitus. Nutrition Reviews. 75(5). 350–360. 30 indexed citations
7.
Koh, Gar Yee, et al.. (2015). Whole Egg Consumption Prevents Diminished Serum 25-Hydroxycholecalciferol Concentrations in Type 2 Diabetic Rats. Journal of Agricultural and Food Chemistry. 64(1). 120–124. 11 indexed citations
8.
9.
Birt, Diane F., Terri D. Boylston, Suzanne Hendrich, et al.. (2013). Resistant Starch: Promise for Improving Human Health. Advances in Nutrition. 4(6). 587–601. 652 indexed citations breakdown →
10.
Borcherding, Nicholas, et al.. (2013). Dietary Resistant Starch Prevents Urinary Excretion of 25-Hydroxycholecalciferol and Vitamin D-Binding Protein in Type 1 Diabetic Rats1,2. Journal of Nutrition. 143(7). 1123–1128. 17 indexed citations
11.
Anderson, Rachel & Matthew J. Rowling. (2009). Renal 25‐hydroxycholecalciferol reabsorption is modulated in a type II diabetic rat model.. The FASEB Journal. 23(S1). 2 indexed citations
12.
Chlon, Timothy M., David A. Taffany, JoEllen Welsh, & Matthew J. Rowling. (2008). Retinoids Modulate Expression of the Endocytic Partners Megalin, Cubilin, and Disabled-2 and Uptake of Vitamin D-Binding Protein in Human Mammary Cells. Journal of Nutrition. 138(7). 1323–1328. 41 indexed citations
13.
Rowling, Matthew J., Christy Gliniak, JoEllen Welsh, & James C. Fleet. (2007). High Dietary Vitamin D Prevents Hypocalcemia and Osteomalacia in CYP27B1 Knockout Mice ,. Journal of Nutrition. 137(12). 2608–2615. 77 indexed citations
14.
Rowling, Matthew J., et al.. (2006). Megalin-Mediated Endocytosis of Vitamin D Binding Protein Correlates with 25-Hydroxycholecalciferol Actions in Human Mammary Cells. Journal of Nutrition. 136(11). 2754–2759. 138 indexed citations
15.
Nieman, Kristin M., Matthew J. Rowling, Timothy A. Garrow, & Kevin L. Schalinske. (2004). Modulation of Methyl Group Metabolism by Streptozotocin-induced Diabetes and All-trans-retinoic Acid. Journal of Biological Chemistry. 279(44). 45708–45712. 51 indexed citations
16.
Rowling, Matthew J. & Kevin L. Schalinske. (2003). Retinoic Acid and Glucocorticoid Treatment Induce Hepatic Glycine N-Methyltransferase and Lower Plasma Homocysteine Concentrations in Rats and Rat Hepatoma Cells. Journal of Nutrition. 133(11). 3392–3398. 39 indexed citations
17.
Rowling, Matthew J., et al.. (2002). Hepatic Glycine N-Methyltransferase Is Up-Regulated by Excess Dietary Methionine in Rats. Journal of Nutrition. 132(9). 2545–2550. 71 indexed citations
18.
Rowling, Matthew J., et al.. (2002). Activation and induction of glycine N-methyltransferase by retinoids are tissue- and gender-specific. Archives of Biochemistry and Biophysics. 401(1). 73–80. 26 indexed citations
19.
Rowling, Matthew J., et al.. (2002). Vitamin A and Its Derivatives Induce Hepatic Glycine N-Methyltransferase and Hypomethylation of DNA in Rats. Journal of Nutrition. 132(3). 365–369. 70 indexed citations
20.
Rowling, Matthew J. & Kevin L. Schalinske. (2001). Retinoid Compounds Activate and Induce Hepatic Glycine N-Methyltransferase in Rats. Journal of Nutrition. 131(7). 1914–1917. 28 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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