Timothy M. Chlon

1.3k total citations
32 papers, 936 citations indexed

About

Timothy M. Chlon is a scholar working on Molecular Biology, Hematology and Genetics. According to data from OpenAlex, Timothy M. Chlon has authored 32 papers receiving a total of 936 indexed citations (citations by other indexed papers that have themselves been cited), including 21 papers in Molecular Biology, 15 papers in Hematology and 6 papers in Genetics. Recurrent topics in Timothy M. Chlon's work include Acute Myeloid Leukemia Research (14 papers), RNA modifications and cancer (7 papers) and Epigenetics and DNA Methylation (5 papers). Timothy M. Chlon is often cited by papers focused on Acute Myeloid Leukemia Research (14 papers), RNA modifications and cancer (7 papers) and Epigenetics and DNA Methylation (5 papers). Timothy M. Chlon collaborates with scholars based in United States, France and Switzerland. Timothy M. Chlon's co-authors include John D. Crispino, Daniel T. Starczynowski, Laura Barreyro, Louis C. Doré, Sébastien Malinge, Lauren Diebold, Meghan Bliss‐Moreau, Kevin P. White, Christopher D. Brown and Sandeep Gurbuxani and has published in prestigious journals such as Journal of Clinical Investigation, Blood and Molecular Cell.

In The Last Decade

Timothy M. Chlon

27 papers receiving 925 citations

Peers

Timothy M. Chlon
Christopher Slape United States
Allison Mayle United States
Anthony A. Fernald United States
R Delwel Netherlands
Ngaire Elwood Australia
Christopher Slape United States
Timothy M. Chlon
Citations per year, relative to Timothy M. Chlon Timothy M. Chlon (= 1×) peers Christopher Slape

Countries citing papers authored by Timothy M. Chlon

Since Specialization
Citations

This map shows the geographic impact of Timothy M. Chlon's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Timothy M. Chlon with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Timothy M. Chlon more than expected).

Fields of papers citing papers by Timothy M. Chlon

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Timothy M. Chlon. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Timothy M. Chlon. The network helps show where Timothy M. Chlon may publish in the future.

Co-authorship network of co-authors of Timothy M. Chlon

This figure shows the co-authorship network connecting the top 25 collaborators of Timothy M. Chlon. A scholar is included among the top collaborators of Timothy M. Chlon based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Timothy M. Chlon. Timothy M. Chlon is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Choi, Issac, Courtnee Clough, Aishlin Hassan, et al.. (2026). Scaffolding-dependent CASP1 constrains excessive cell-intrinsic inflammatory signaling in leukemia. Cell chemical biology. 33(1). 59–73.e10.
2.
Kusne, Yael, Talha Badar, Terra L. Lasho, et al.. (2025). Prevalence of cytopenia(s) and somatic variants in patients with DDX41 mutant germline predisposition syndrome. British Journal of Haematology. 206(4). 1109–1120. 2 indexed citations
3.
Badar, Talha, Yael Kusne, James M. Foran, et al.. (2025). Decoding DDX41: Clinical impact of germline and somatic mutations in 77 high-risk myeloid neoplasm patients. Blood. 146(Supplement 1). 1442–1442.
5.
Kusne, Yael, Talha Badar, Terra L. Lasho, et al.. (2024). Prevalence and Clinical Features of Non-Myeloid Neoplasms in Patients with DDX41 Mutant Germline Predisposition Syndrome. Blood. 144(Supplement 1). 440–440.
6.
Kusne, Yael, Talha Badar, Terra L. Lasho, et al.. (2024). Clinical Characteristics of Myeloproliferative Neoplasms in Patients with DDX41MT Germline Predisposition Syndrome. Blood. 144(Supplement 1). 3183–3183.
7.
Badar, Talha, James M. Foran, David S. Viswanatha, et al.. (2023). Clinical and molecular correlates of somatic and germline <i>DDX41</i> variants in patients and families with myeloid neoplasms. Haematologica. 108(11). 3033–3043. 10 indexed citations
8.
Badar, Talha & Timothy M. Chlon. (2022). Germline and Somatic Defects in DDX41 and its Impact on Myeloid Neoplasms. Current Hematologic Malignancy Reports. 17(5). 113–120. 13 indexed citations
9.
Hueneman, Kathleen, Lyndsey Bolanos, Kwangmin Choi, et al.. (2021). The deubiquitinase USP15 modulates cellular redox and is a therapeutic target in acute myeloid leukemia. Leukemia. 36(2). 438–451. 25 indexed citations
10.
Chlon, Timothy M., Courtney E. Hershberger, Kathleen Hueneman, et al.. (2021). Germline DDX41 mutations cause ineffective hematopoiesis and myelodysplasia. Cell stem cell. 28(11). 1966–1981.e6. 57 indexed citations
11.
Witte, David P., Kathryn A. Wikenheiser‐Brokamp, Mathieu Sertorio, et al.. (2020). Inherited DNA Repair Defects Disrupt the Structure and Function of Human Skin. Cell stem cell. 28(3). 424–435.e6. 10 indexed citations
12.
Barreyro, Laura, Timothy M. Chlon, & Daniel T. Starczynowski. (2018). Chronic immune response dysregulation in MDS pathogenesis. Blood. 132(15). 1553–1560. 158 indexed citations
13.
Chlon, Timothy M., Christopher N. Mayhew, Kathryn A. Wikenheiser‐Brokamp, et al.. (2016). Overcoming Pluripotent Stem Cell Dependence on the Repair of Endogenous DNA Damage. Stem Cell Reports. 6(1). 44–54. 26 indexed citations
14.
Chlon, Timothy M., Elizabeth E. Hoskins, Christopher N. Mayhew, et al.. (2014). High-Risk Human Papillomavirus E6 Protein Promotes Reprogramming of Fanconi Anemia Patient Cells through Repression of p53 but Does Not Allow for Sustained Growth of Induced Pluripotent Stem Cells. Journal of Virology. 88(19). 11315–11326. 25 indexed citations
15.
Birger, Yehudit, Liat Goldberg, Timothy M. Chlon, et al.. (2013). Perturbation of fetal hematopoiesis in a mouse model of Down syndrome’s transient myeloproliferative disorder. Blood. 122(6). 988–998. 34 indexed citations
16.
Doré, Louis C., Timothy M. Chlon, Christopher D. Brown, Kevin P. White, & John D. Crispino. (2012). Chromatin occupancy analysis reveals genome-wide GATA factor switching during hematopoiesis. Blood. 119(16). 3724–3733. 87 indexed citations
17.
Malinge, Sébastien, Meghan Bliss‐Moreau, Gina Kirsammer, et al.. (2012). Increased dosage of the chromosome 21 ortholog Dyrk1a promotes megakaryoblastic leukemia in a murine model of Down syndrome. Journal of Clinical Investigation. 122(3). 948–962. 119 indexed citations
18.
Mukhopadhyay, Abhishek, et al.. (2009). HeyL regulates the number of TrkC neurons in dorsal root ganglia. Developmental Biology. 334(1). 142–151. 18 indexed citations
19.
Chlon, Timothy M., David A. Taffany, JoEllen Welsh, & Matthew J. Rowling. (2008). Retinoids Modulate Expression of the Endocytic Partners Megalin, Cubilin, and Disabled-2 and Uptake of Vitamin D-Binding Protein in Human Mammary Cells. Journal of Nutrition. 138(7). 1323–1328. 41 indexed citations
20.
Houston, Isaac B., et al.. (2007). Reduction in PU.1 activity results in a block to B-cell development, abnormal myeloid proliferation, and neonatal lethality. Experimental Hematology. 35(7). 1056–1068. 35 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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