Haoqiang Ying

19.0k total citations · 5 hit papers
59 papers, 8.4k citations indexed

About

Haoqiang Ying is a scholar working on Molecular Biology, Oncology and Cancer Research. According to data from OpenAlex, Haoqiang Ying has authored 59 papers receiving a total of 8.4k indexed citations (citations by other indexed papers that have themselves been cited), including 39 papers in Molecular Biology, 31 papers in Oncology and 22 papers in Cancer Research. Recurrent topics in Haoqiang Ying's work include Cancer-related Molecular Pathways (14 papers), Pancreatic and Hepatic Oncology Research (12 papers) and Cancer, Hypoxia, and Metabolism (11 papers). Haoqiang Ying is often cited by papers focused on Cancer-related Molecular Pathways (14 papers), Pancreatic and Hepatic Oncology Research (12 papers) and Cancer, Hypoxia, and Metabolism (11 papers). Haoqiang Ying collaborates with scholars based in United States, China and Germany. Haoqiang Ying's co-authors include Alec C. Kimmelman, Ronald A. DePinho, Xiaoxu Wang, Costas A. Lyssiotis, John M. Asara, Jayne M. Stommel, Lewis C. Cantley, Marcia C. Haigis, Nabeel Bardeesy and Lynda Chin and has published in prestigious journals such as Nature, Science and Cell.

In The Last Decade

Haoqiang Ying

59 papers receiving 8.3k citations

Hit Papers

Glutamine supports pancreatic cancer growth through a KRA... 2007 2026 2013 2019 2013 2011 2016 2007 2008 400 800 1.2k

Peers

Haoqiang Ying
M. James You United States
Jayne M. Stommel United States
Lyuba Varticovski United States
Othon Iliopoulos United States
Stephanos Pavlides United States
Seth J. Parker United States
Antonio Di Cristofano United States
Haoqiang Ying
Citations per year, relative to Haoqiang Ying Haoqiang Ying (= 1×) peers Julio A. Aguirre‐Ghiso

Countries citing papers authored by Haoqiang Ying

Since Specialization
Citations

This map shows the geographic impact of Haoqiang Ying's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Haoqiang Ying with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Haoqiang Ying more than expected).

Fields of papers citing papers by Haoqiang Ying

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Haoqiang Ying. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Haoqiang Ying. The network helps show where Haoqiang Ying may publish in the future.

Co-authorship network of co-authors of Haoqiang Ying

This figure shows the co-authorship network connecting the top 25 collaborators of Haoqiang Ying. A scholar is included among the top collaborators of Haoqiang Ying based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Haoqiang Ying. Haoqiang Ying is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Denu, Ryan A., Ahsan Farooqi, Davis R. Ingram, et al.. (2024). Impact of ATRX loss on survival and immune microenvironment in multiple sarcoma subtypes.. Journal of Clinical Oncology. 42(16_suppl). 11511–11511. 2 indexed citations
2.
Li, Fei, Inah Hwang, Libo Xu, et al.. (2023). Histone demethylase KDM2A is a selective vulnerability of cancers relying on alternative telomere maintenance. Nature Communications. 14(1). 1756–1756. 19 indexed citations
3.
Liu, Yi, Yasunori Deguchi, Daoyan Wei, et al.. (2022). Rapid acceleration of KRAS-mutant pancreatic carcinogenesis via remodeling of tumor immune microenvironment by PPARδ. Nature Communications. 13(1). 2665–2665. 45 indexed citations
4.
Zhao, Xixi, Yongkun Wei, Yu‐Yi Chu, et al.. (2022). Phosphorylation and Stabilization of PD-L1 by CK2 Suppresses Dendritic Cell Function. Cancer Research. 82(11). 2185–2195. 36 indexed citations
5.
Liang, Yan, Bo Tu, Jun Yao, et al.. (2021). Targeting Glucose Metabolism Sensitizes Pancreatic Cancer to MEK Inhibition. Cancer Research. 81(15). 4054–4065. 30 indexed citations
6.
Oh, Hwanhee, Inah Hwang, Lingxiang Wu, et al.. (2021). Therapy-Induced Transdifferentiation Promotes Glioma Growth Independent of EGFR Signaling. Cancer Research. 81(6). 1528–1539. 7 indexed citations
7.
Han, Yan, Yeran Yang, Jun Yan, et al.. (2021). Loss of the wild-type KRAS allele promotes pancreatic cancer progression through functional activation of YAP1. Oncogene. 40(50). 6759–6771. 17 indexed citations
8.
Deng, Yalan, Xianghou Xia, Yang Zhao, et al.. (2021). Glucocorticoid receptor regulates PD-L1 and MHC-I in pancreatic cancer cells to promote immune evasion and immunotherapy resistance. Nature Communications. 12(1). 7041–7041. 84 indexed citations
9.
Xu, Bo, et al.. (2020). Upregulated lncRNA THRIL/TNF-α Signals Promote Cell Growth and Predict Poor Clinical Outcomes of Osteosarcoma. SHILAP Revista de lepidopterología. 1 indexed citations
10.
Yu, Meifang, Nicholas D. Nguyen, Yanqing Huang, et al.. (2019). Mitochondrial fusion exploits a therapeutic vulnerability of pancreatic cancer. JCI Insight. 4(16). 129 indexed citations
11.
Li, Fei, Zhong Deng, Ling Zhang, et al.. (2019). ATRX loss induces telomere dysfunction and necessitates induction of alternative lengthening of telomeres during human cell immortalization. The EMBO Journal. 38(19). e96659–e96659. 87 indexed citations
12.
Ju, Huai‐Qiang, Haoqiang Ying, Tian Tian, et al.. (2017). Mutant Kras- and p16-regulated NOX4 activation overcomes metabolic checkpoints in development of pancreatic ductal adenocarcinoma. Nature Communications. 8(1). 14437–14437. 78 indexed citations
13.
Klingler, Stefan, Baofeng Guo, Jun Yao, et al.. (2015). Development of Resistance to EGFR-Targeted Therapy in Malignant Glioma Can Occur through EGFR-Dependent and -Independent Mechanisms. Cancer Research. 75(10). 2109–2119. 29 indexed citations
14.
Normolle, Daniel P., Douglas J. Hartman, Haoqiang Ying, et al.. (2014). Neuroplastic Changes Occur Early in the Development of Pancreatic Ductal Adenocarcinoma. Cancer Research. 74(6). 1718–1727. 143 indexed citations
15.
Du, Wei, Yong Yi, Haibo Zhang, et al.. (2013). Correction: Rapamycin Inhibits IGF-1-Mediated Up-Regulation of MDM2 and Sensitizes Cancer Cells to Chemotherapy. PLoS ONE. 8(12). 4 indexed citations
16.
17.
Yang, Shenghong, Xiaoxu Wang, Gianmarco Contino, et al.. (2011). Pancreatic cancers require autophagy for tumor growth. Genes & Development. 25(7). 717–729. 1163 indexed citations breakdown →
18.
Liu, Yunhui, Haoqiang Ying, Yixue Xue, et al.. (2010). Increasing of Blood-tumor Barrier Permeability through Paracellular Pathway by Low-frequency Ultrasound Irradiation In Vitro. Journal of Molecular Neuroscience. 43(3). 541–548. 23 indexed citations
19.
Stommel, Jayne M., Alec C. Kimmelman, Haoqiang Ying, et al.. (2007). Coactivation of Receptor Tyrosine Kinases Affects the Response of Tumor Cells to Targeted Therapies. Science. 318(5848). 287–290. 699 indexed citations breakdown →
20.
Sdek, Patima, Haoqiang Ying, Donny L.F. Chang, et al.. (2005). MDM2 Promotes Proteasome-Dependent Ubiquitin-Independent Degradation of Retinoblastoma Protein. Molecular Cell. 20(5). 699–708. 214 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

Explore authors with similar magnitude of impact

Rankless by CCL
2026