Inah Hwang

1.6k total citations
28 papers, 1.2k citations indexed

About

Inah Hwang is a scholar working on Molecular Biology, Immunology and Epidemiology. According to data from OpenAlex, Inah Hwang has authored 28 papers receiving a total of 1.2k indexed citations (citations by other indexed papers that have themselves been cited), including 22 papers in Molecular Biology, 5 papers in Immunology and 4 papers in Epidemiology. Recurrent topics in Inah Hwang's work include Peroxisome Proliferator-Activated Receptors (4 papers), Ubiquitin and proteasome pathways (3 papers) and Advanced Glycation End Products research (3 papers). Inah Hwang is often cited by papers focused on Peroxisome Proliferator-Activated Receptors (4 papers), Ubiquitin and proteasome pathways (3 papers) and Advanced Glycation End Products research (3 papers). Inah Hwang collaborates with scholars based in South Korea, United States and China. Inah Hwang's co-authors include Hunjoo Ha, Jong Hee Park, Jihye Paik, Jiyoun Lee, Joo Young Huh, Florian L. Müller, Md Jamal Uddin, Ye-Shih Ho, Eun Young Seo and Ho Jae Han and has published in prestigious journals such as Journal of Clinical Investigation, Nature Communications and The EMBO Journal.

In The Last Decade

Inah Hwang

26 papers receiving 1.2k citations

Peers

Inah Hwang

PHYSI

EPIDE

NEPHR

Hao Zhao China

Jun Hao China

Shyue-Fang Battaglia-Hsu France

Sungmi Park South Korea

Meenalakshmi M. Mariappan United States

Takahiro Ueno Japan

Rosita Stanzione Italy

Bardia Askari United States

Souad Belmadani United States

Rody San Martín Chile

Hao Zhao China

Inah Hwang

608 ×0.9

222 ×1.2

PHYSI

232 ×1.4

EPIDE

149 ×1.1

183 ×1.4

NEPHR

Citations per year, relative to Inah Hwang Inah Hwang (= 1×) peers Hao Zhao

Countries citing papers authored by Inah Hwang

Since Specialization

Citations

This map shows the geographic impact of Inah Hwang's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Inah Hwang with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Inah Hwang more than expected).

Fields of papers citing papers by Inah Hwang

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Inah Hwang. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Inah Hwang. The network helps show where Inah Hwang may publish in the future.

Co-authorship network of co-authors of Inah Hwang

This figure shows the co-authorship network connecting the top 25 collaborators of Inah Hwang. A scholar is included among the top collaborators of Inah Hwang based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Inah Hwang. Inah Hwang is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Shin, Sang Chul, Yeonji Oh, Jin Hyeok Kim, et al.. (2025). Targeting USP47 enhances the efficacy of KRAS inhibitor in KRASG12C mutated non-small cell lung cancer by controlling deubiquitination of c-Myc. Pharmacological Research. 215. 107722–107722. 1 indexed citations

Im, Joo‐Young, Soo Jin Kim, Jong‐Lyul Park, et al.. (2024). CYB5R3 functions as a tumor suppressor by inducing ER stress-mediated apoptosis in lung cancer cells via the PERK-ATF4 and IRE1α-JNK pathways. Experimental & Molecular Medicine. 56(1). 235–249. 8 indexed citations

Li, Fei, Inah Hwang, Libo Xu, et al.. (2023). Histone demethylase KDM2A is a selective vulnerability of cancers relying on alternative telomere maintenance. Nature Communications. 14(1). 1756–1756. 19 indexed citations

Oh, Hwanhee, Inah Hwang, Lingxiang Wu, et al.. (2021). Therapy-Induced Transdifferentiation Promotes Glioma Growth Independent of EGFR Signaling. Cancer Research. 81(6). 1528–1539. 7 indexed citations

Hwang, Inah, Hiroki Uchida, Ziwei Dai, et al.. (2021). Cellular stress signaling activates type-I IFN response through FOXO3-regulated lamin posttranslational modification. Nature Communications. 12(1). 640–640. 29 indexed citations

Hwang, Inah, et al.. (2021). Oxidative stress sensing and response in neural stem cell fate. Free Radical Biology and Medicine. 169. 74–83. 21 indexed citations

Hwang, Inah, Youssef Youssef, Wing Keung Chan, et al.. (2021). PRMT5 Inhibition Promotes FOXO1 Tumor Suppressor Activity to Drive a Pro-Apoptotic Program That Creates Vulnerability to Combination Treatment with Venetoclax in Mantle Cell Lymphoma. Blood. 138(Supplement 1). 681–681. 2 indexed citations

Li, Fei, Zhong Deng, Ling Zhang, et al.. (2019). ATRX loss induces telomere dysfunction and necessitates induction of alternative lengthening of telomeres during human cell immortalization. The EMBO Journal. 38(19). e96659–e96659. 87 indexed citations

Choi, Soo An, Adnan Khan, Joo Young Huh, et al.. (2019). Integrative Omics Reveals Metabolic and Transcriptomic Alteration of Nonalcoholic Fatty Liver Disease in Catalase Knockout Mice. Biomolecules & Therapeutics. 27(2). 134–144. 15 indexed citations

10.

Berger, Adeline, Nicholas J. Brady, Rohan Bareja, et al.. (2019). N-Myc–mediated epigenetic reprogramming drives lineage plasticity in advanced prostate cancer. Journal of Clinical Investigation. 129(9). 3924–3940. 116 indexed citations

11.

Hwang, Inah, Md Jamal Uddin, Eun‐Jung Jin, et al.. (2019). The impaired redox balance in peroxisomes of catalase knockout mice accelerates nonalcoholic fatty liver disease through endoplasmic reticulum stress. Free Radical Biology and Medicine. 148. 22–32. 43 indexed citations

12.

Hwang, Inah, et al.. (2018). Peroxiredoxin 3 deficiency accelerates chronic kidney injury in mice through interactions between macrophages and tubular epithelial cells. Free Radical Biology and Medicine. 131. 162–172. 33 indexed citations

13.

Hwang, Inah, Dongqing Cao, Tuo Zhang, et al.. (2018). Far Upstream Element-Binding Protein 1 Regulates LSD1 Alternative Splicing to Promote Terminal Differentiation of Neural Progenitors. Stem Cell Reports. 10(4). 1208–1221. 29 indexed citations

14.

Hwang, Inah, Hwanhee Oh, Evan E. Santo, et al.. (2017). FOXO protects against age‐progressive axonal degeneration. Aging Cell. 17(1). 51 indexed citations

15.

Uddin, Md Jamal, et al.. (2016). Novel Role of Endogenous Catalase in Macrophage Polarization in Adipose Tissue. Mediators of Inflammation. 2016. 1–14. 24 indexed citations

16.

Hwang, Inah, et al.. (2015). Functional regulation of FoxO1 in neural stem cell differentiation. Cell Death and Differentiation. 22(12). 2034–2045. 72 indexed citations

17.

Lee, Jiyoun, et al.. (2013). The Selective A3AR Antagonist LJ-1888 Ameliorates UUO-Induced Tubulointerstitial Fibrosis. American Journal Of Pathology. 183(5). 1488–1497. 41 indexed citations

18.

Hwang, Inah, et al.. (2012). Catalase Deficiency Accelerates Diabetic Renal Injury Through Peroxisomal Dysfunction. Diabetes. 61(3). 728–738. 147 indexed citations

19.

Hwang, Inah, Eun Young Seo, & Hunjoo Ha. (2009). Wnt/β-catenin signaling: A novel target for therapeutic intervention of fibrotic kidney disease. Archives of Pharmacal Research. 32(12). 1653–1662. 60 indexed citations

20.

Han, Ki‐Hwan, U‐Young Lee, Inah Hwang, et al.. (2006). Differential regulation of B/K protein expression in proximal and distal tubules of rat kidneys with ischemia-reperfusion injury. American Journal of Physiology-Renal Physiology. 292(1). F100–F106. 9 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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