Christopher A. Bristow

36.9k total citations
30 papers, 995 citations indexed

About

Christopher A. Bristow is a scholar working on Molecular Biology, Cancer Research and Oncology. According to data from OpenAlex, Christopher A. Bristow has authored 30 papers receiving a total of 995 indexed citations (citations by other indexed papers that have themselves been cited), including 17 papers in Molecular Biology, 9 papers in Cancer Research and 8 papers in Oncology. Recurrent topics in Christopher A. Bristow's work include Cancer Genomics and Diagnostics (6 papers), Developmental Biology and Gene Regulation (4 papers) and CRISPR and Genetic Engineering (3 papers). Christopher A. Bristow is often cited by papers focused on Cancer Genomics and Diagnostics (6 papers), Developmental Biology and Gene Regulation (4 papers) and CRISPR and Genetic Engineering (3 papers). Christopher A. Bristow collaborates with scholars based in United States, Italy and United Kingdom. Christopher A. Bristow's co-authors include Timothy P. Heffernan, Manolis Kellis, Jeffrey J. Kovacs, Joseph R. Marszalek, Margaret A. Goodell, Ayala Tovy, Jianhua Zhang, Michael Peoples, P. Andrew Futreal and Alessandro Carugo and has published in prestigious journals such as Nucleic Acids Research, Nature Communications and Bioinformatics.

In The Last Decade

Christopher A. Bristow

28 papers receiving 991 citations

Peers

Christopher A. Bristow
Meenakshi Mehrotra United States
Henry Lee-Six United Kingdom
Anthony A. Fernald United States
Ilka Warshawsky United States
Mareike Roth Austria
Christopher A. Bristow
Citations per year, relative to Christopher A. Bristow Christopher A. Bristow (= 1×) peers Björn Schneider

Countries citing papers authored by Christopher A. Bristow

Since Specialization
Citations

This map shows the geographic impact of Christopher A. Bristow's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Christopher A. Bristow with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Christopher A. Bristow more than expected).

Fields of papers citing papers by Christopher A. Bristow

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Christopher A. Bristow. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Christopher A. Bristow. The network helps show where Christopher A. Bristow may publish in the future.

Co-authorship network of co-authors of Christopher A. Bristow

This figure shows the co-authorship network connecting the top 25 collaborators of Christopher A. Bristow. A scholar is included among the top collaborators of Christopher A. Bristow based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Christopher A. Bristow. Christopher A. Bristow is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
House, Nealia C., Maxine Chen, Sima Khazaei, et al.. (2025). CDK2 inhibition enhances CDK4/6 inhibitor antitumor activity in comprehensive breast cancer PDX model screen. npj Breast Cancer. 11(1). 135–135. 1 indexed citations
2.
Morris, Van K., Stefania Napolitano, Christopher A. Bristow, et al.. (2024). Epigenome Reprogramming Through H3K27 and H3K4 Trimethylation as a Resistance Mechanism to DNA Methylation Inhibition in BRAFV600E-Mutated Colorectal Cancer. Clinical Cancer Research. 30(22). 5166–5179. 7 indexed citations
3.
Wong, Chi Wut, Rosalba Minelli, Michael Peoples, et al.. (2024). Abstract 3280: ASCL2+ tumor cells modulate the response of colorectal cancer to MAPK targeting therapy. Cancer Research. 84(6_Supplement). 3280–3280.
4.
Colic, Medina, et al.. (2022). PICKLES v3: the updated database of pooled in vitro CRISPR knockout library essentiality screens. Nucleic Acids Research. 51(D1). D1117–D1121. 6 indexed citations
5.
Kim, Eiru, et al.. (2022). Dynamic rewiring of biological activity across genotype and lineage revealed by context-dependent functional interactions. Genome biology. 23(1). 140–140. 10 indexed citations
6.
Class, Caleb A., et al.. (2022). Easy NanoString nCounter data analysis with the NanoTube. Bioinformatics. 39(1). 11 indexed citations
7.
Liang, Yan, Bo Tu, Jun Yao, et al.. (2021). Targeting Glucose Metabolism Sensitizes Pancreatic Cancer to MEK Inhibition. Cancer Research. 81(15). 4054–4065. 30 indexed citations
8.
Shariati, Maryam, Kurt W. Evans, Xiaofeng Zheng, et al.. (2021). Combined inhibition of DDR1 and CDK4/6 induces synergistic effects in ER-positive, HER2-negative breast cancer with PIK3CA/AKT1 mutations. Oncogene. 40(26). 4425–4439. 17 indexed citations
9.
Konen, Jessica, B. Leticia Rodriguez, Jared J. Fradette, et al.. (2021). Targeting CDK4 overcomes EMT-mediated tumor heterogeneity and therapeutic resistance in KRAS-mutant lung cancer. JCI Insight. 6(17). 14 indexed citations
10.
Coyaud, Étienne, Samrat T. Kundu, David H. Peng, et al.. (2019). ZEB1/NuRD complex suppresses TBC1D2b to stimulate E-cadherin internalization and promote metastasis in lung cancer. Nature Communications. 10(1). 5125–5125. 73 indexed citations
11.
Powell, Emily, Jiansu Shao, Christopher A. Bristow, et al.. (2018). A functional genomic screen in vivo identifies CEACAM5 as a clinically relevant driver of breast cancer metastasis. npj Breast Cancer. 4(1). 9–9. 39 indexed citations
12.
Loree, Jonathan M., Ann M. Bailey, Amber M. Johnson, et al.. (2018). Molecular Landscape ofERBB2/ERBB3Mutated Colorectal Cancer. JNCI Journal of the National Cancer Institute. 110(12). 1409–1417. 48 indexed citations
13.
Hsu, Joanne I., Tajhal Dayaram, Ayala Tovy, et al.. (2018). PPM1D Mutations Drive Clonal Hematopoiesis in Response to Cytotoxic Chemotherapy. Cell stem cell. 23(5). 700–713.e6. 268 indexed citations
14.
Κατσιαμπούρα, Αναστασία, Kanwal Raghav, Zhi-Qin Jiang, et al.. (2017). Modeling of Patient-Derived Xenografts in Colorectal Cancer. Molecular Cancer Therapeutics. 16(7). 1435–1442. 38 indexed citations
15.
Vangapandu, Hima V., Mary Ayres, Christopher A. Bristow, et al.. (2017). The Stromal Microenvironment Modulates Mitochondrial Oxidative Phosphorylation in Chronic Lymphocytic Leukemia Cells. Neoplasia. 19(10). 762–771. 33 indexed citations
16.
Yang, Lixing, Mi-Sook Lee, Hengyu Lu, et al.. (2016). Analyzing Somatic Genome Rearrangements in Human Cancers by Using Whole-Exome Sequencing. The American Journal of Human Genetics. 98(5). 843–856. 29 indexed citations
17.
Marbach, Daniel, Sushmita Roy, Ferhat Ay, et al.. (2012). Predictive regulatory models in Drosophila melanogaster by integrative inference of transcriptional networks. Genome Research. 22(7). 1334–1349. 89 indexed citations
18.
Yakoby, Nir, Christopher A. Bristow, Xenia Schafer, et al.. (2008). A Combinatorial Code for Pattern Formation in Drosophila Oogenesis. Developmental Cell. 15(5). 725–737. 63 indexed citations
19.
Zartman, Jeremiah J., Nir Yakoby, Christopher A. Bristow, et al.. (2008). Cad74A is regulated by BR and is required for robust dorsal appendage formation in Drosophila oogenesis. Developmental Biology. 322(2). 289–301. 17 indexed citations
20.
Yakoby, Nir, Christopher A. Bristow, I. Gouzman, et al.. (2005). Systems-level questions in Drosophila oogenesis. PubMed. 152(4). 276–276. 14 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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