Gil Blander

12 papers receiving 3.8k citations

Hit Papers

SIRT1 Deacetylates and Positively Regulates the Nuclear Receptor LXR 2007 · 523 citations
52320042026201120184008001.2k

Peers

Gil Blander
Comparison fields: 5 of 109
  • Geriatrics and Gerontology 2.1k
  • Aging 211
  • Physiology 452
  • Physiology 1.1k
  • Epidemiology 1.0k
Replace Eriko Michishita with:
Eriko Michishita Japan
Hwei-Ling Cheng United States
Carlos Sebastián United States
Bjoern Schwer United States
Tiara L.A. Kawahara United States
Jianyuan Luo China
Yana Cen United States
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Marcella Fulco United States
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Citations per field
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Citations per year

Countries citing papers authored by Gil Blander

Since Specialization
Citations

This map shows the geographic impact of Gil Blander's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Gil Blander with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Gil Blander more than expected).

Fields of papers citing papers by Gil Blander

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Gil Blander. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Gil Blander. The network helps show where Gil Blander may publish in the future.

Co-authors

The 25 scholars most cited alongside Gil Blander, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Gil Blander Line = papers co-authored together Gil Blander links everyone, so they are left out of the graph.

All Works

12 of 12 papers shown
#Work
1
The Sir2 Family of Protein Deacetylases
Hit paper breakdown →
20041261
2
SIRT4 Inhibits Glutamate Dehydrogenase and Opposes the Effects of Calorie Restriction in Pancreatic β Cells
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2006930
3
SIRT1 Deacetylates and Positively Regulates the Nuclear Receptor LXR
Hit paper breakdown →
2007523
4 2008477
5 1999147
6 2003145
7 2002111
8 200896
9 200292
10 200558
11 200033
12 201218

About Gil Blander

Gil Blander is a scholar working on Geriatrics and Gerontology, Aging, Physiology, Endocrine and Autonomic Systems and Oncology, having authored 12 papers that have together received 3.9k indexed citations. Recurring topics across this work include Sirtuins and Resveratrol in Medicine (6 papers), Adipose Tissue and Metabolism (5 papers), DNA Repair Mechanisms (4 papers), Cancer-related Molecular Pathways (3 papers), Genomics and Chromatin Dynamics (2 papers), Genetics, Aging, and Longevity in Model Organisms (2 papers), Biochemical effects in animals (2 papers) and Telomeres, Telomerase, and Senescence (1 paper). The work is most often cited by research in Geriatrics and Gerontology (2.1k citations), Aging (211 citations), Physiology (452 citations), Physiology (1.1k citations) and Epidemiology (1.0k citations). Gil Blander has collaborated with scholars based in United States, Israel and Switzerland. Frequent co-authors include Leonard Guarente, Marcia C. Haigis, Xiaoling Li, Songwen Zhang, Monty Krieger, Raúl Mostoslavsky, Kevin M. Haigis, Andrew Murphy, Danos C. Christodoulou and Cynthia Wolberger. Their work appears in journals such as Journal of Biological Chemistry, PLoS ONE, Annual Review of Biochemistry, Journal of Investigative Dermatology and Molecular Cell.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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