William C. Hahn
Impact in
- Cancer Research top 0.02%
- Cancer Genomics and Diagnostics
- Oncology top 0.02%
- Cancer-related Molecular Pathways
- Cancer Cells and Metastasis
Papers in
-
- Ubiquitin and proteasome pathways 34
- Protein Degradation and Inhibitors 32
- Genomics and Chromatin Dynamics 24
- Epigenetics and DNA Methylation 24
- Cancer-related gene regulation 23
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- Cancer Genomics and Diagnostics 40
- Co-authors
- Robert A. Weinberg (15 shared papers)Mary W. Brooks (8 shared papers)Roderick L. Beijersbergen (3 shared papers)Christopher M Counter (3 shared papers)Ante S. Lundberg (4 shared papers)Matthew Meyerson (16 shared papers)Sheila A. Stewart (9 shared papers)Brent R. Stockwell (2 shared papers)
- Journals
- Cell (19 papers)Proceedings of the National Academy of Sciences (18 papers)Cancer Cell (17 papers)Cancer Research (17 papers)Nature (16 papers)
- Partner nations
- United StatesGermanyCanada
In The Last Decade
William C. Hahn
379 papers receiving 74.0k citations
William C. Hahn's Hit Papers
Peers
Comparison fields: 5 of 195
- Cancer Research 14.1k
- Oncology 19.4k
- Molecular Biology 45.6k
- Aging 962
- Hematology 4.0k
Countries citing papers authored by William C. Hahn
This map shows the geographic impact of William C. Hahn's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by William C. Hahn with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites William C. Hahn more than expected).
Fields of papers citing papers by William C. Hahn
This network shows the impact of papers produced by William C. Hahn. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by William C. Hahn. The network helps show where William C. Hahn may publish in the future.
Co-authors
The 25 scholars most cited alongside William C. Hahn, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
Showing the 20 most-cited of 386 papers — load more, or switch the sort, to bring in the rest.
| # | Work | ||
|---|---|---|---|
| 1 | Systematic RNA interference reveals that oncogenic KRAS-driven cancers require TBK1 Hit paper breakdown → | 2009 | 2831 |
| 2 | BET Bromodomain Inhibition as a Therapeutic Strategy to Target c-Myc Hit paper breakdown → | 2011 | 2412 |
| 3 | Next-generation characterization of the Cancer Cell Line Encyclopedia Hit paper breakdown → | 2019 | 2326 |
| 4 | Defining a Cancer Dependency Map Hit paper breakdown → | 2017 | 2149 |
| 5 | Creation of human tumour cells with defined genetic elements Hit paper breakdown → | 1999 | 1936 |
| 6 | Dependency of a therapy-resistant state of cancer cells on a lipid peroxidase pathway Hit paper breakdown → | 2017 | 1593 |
| 7 | A Lentiviral RNAi Library for Human and Mouse Genes Applied to an Arrayed Viral High-Content Screen Hit paper breakdown → | 2006 | 1479 |
| 8 | Computational correction of copy number effect improves specificity of CRISPR–Cas9 essentiality screens in cancer cells Hit paper breakdown → | 2017 | 1355 |
| 9 | Lentivirus-delivered stable gene silencing by RNAi in primary cells Hit paper breakdown → | 2003 | 1248 |
| 10 | Analysis of Fusobacterium persistence and antibiotic response in colorectal cancer Hit paper breakdown → | 2017 | 1248 |
| 11 | Identification of genotype-selective antitumor agents using synthetic lethal chemical screening in engineered human tumor cells Hit paper breakdown → | 2003 | 1245 |
| 12 | COT drives resistance to RAF inhibition through MAP kinase pathway reactivation Hit paper breakdown → | 2010 | 1220 |
| 13 | Transformation from committed progenitor to leukaemia stem cell initiated by MLL–AF9 Hit paper breakdown → | 2006 | 1217 |
| 14 | Organoid Modeling of the Tumor Immune Microenvironment Hit paper breakdown → | 2018 | 1215 |
| 15 | Initial genome sequencing and analysis of multiple myeloma Hit paper breakdown → | 2011 | 1207 |
| 16 | Human Keratinocytes That Express hTERT and Also Bypass a p16 INK4a -Enforced Mechanism That Limits Life Span Become Immortal yet Retain Normal Growth and Differentiation Characteristics Hit paper breakdown → | 2000 | 939 |
| 17 | Recurrent BRAF mutations in Langerhans cell histiocytosis Hit paper breakdown → | 2010 | 924 |
| 18 | Inhibition of telomerase limits the growth of human cancer cells Hit paper breakdown → | 1999 | 896 |
| 19 | Genomic Characterization of Brain Metastases Reveals Branched Evolution and Potential Therapeutic Targets Hit paper breakdown → | 2015 | 849 |
| 20 | Dissecting Therapeutic Resistance to RAF Inhibition in Melanoma by Tumor Genomic Profiling Hit paper breakdown → | 2011 | 825 |
About William C. Hahn
William C. Hahn is a scholar working on Molecular Biology, Cancer Research, Oncology, Physiology and Immunology, having authored 386 papers that have together received 75.6k indexed citations. Recurring topics across this work include Telomeres, Telomerase, and Senescence (47 papers), Cancer Genomics and Diagnostics (40 papers), Ubiquitin and proteasome pathways (34 papers), Protein Degradation and Inhibitors (32 papers), Cancer-related Molecular Pathways (30 papers), Genomics and Chromatin Dynamics (24 papers), Epigenetics and DNA Methylation (24 papers) and Cancer-related gene regulation (23 papers). The work is most often cited by research in Cancer Research (14.1k citations), Oncology (19.4k citations), Molecular Biology (45.6k citations), Aging (962 citations) and Hematology (4.0k citations). William C. Hahn has collaborated with scholars based in United States, Germany and Canada. Frequent co-authors include Robert A. Weinberg, Mary W. Brooks, Roderick L. Beijersbergen, Christopher M Counter, Ante S. Lundberg, Matthew Meyerson, Sheila A. Stewart, Brent R. Stockwell, David E. Root and Sonam Dolma. Their work appears in journals such as Cell, Proceedings of the National Academy of Sciences, Cancer Cell, Cancer Research and Nature.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.