Alpaslan Dedeoglu

6.5k total citations · 1 hit paper
62 papers, 5.3k citations indexed

About

Alpaslan Dedeoglu is a scholar working on Cellular and Molecular Neuroscience, Molecular Biology and Physiology. According to data from OpenAlex, Alpaslan Dedeoglu has authored 62 papers receiving a total of 5.3k indexed citations (citations by other indexed papers that have themselves been cited), including 33 papers in Cellular and Molecular Neuroscience, 31 papers in Molecular Biology and 22 papers in Physiology. Recurrent topics in Alpaslan Dedeoglu's work include Genetic Neurodegenerative Diseases (23 papers), Alzheimer's disease research and treatments (18 papers) and Mitochondrial Function and Pathology (16 papers). Alpaslan Dedeoglu is often cited by papers focused on Genetic Neurodegenerative Diseases (23 papers), Alzheimer's disease research and treatments (18 papers) and Mitochondrial Function and Pathology (16 papers). Alpaslan Dedeoglu collaborates with scholars based in United States, Türkiye and France. Alpaslan Dedeoglu's co-authors include Robert J. Ferrante, Ole A. Andreassen, Bruce G. Jenkins, Steven M. Hersch, M. Flint Beal, Neil W. Kowall, Ji‐Kyung Choi, M. Flint Beal, Péter Klivènyi and Isabel Carreras and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Neuroscience and Annals of Neurology.

In The Last Decade

Alpaslan Dedeoglu

62 papers receiving 5.2k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Mice Deficient in Cellular Glutathione Peroxidase Show Increased Vulnerability to Malonate, 3-Nitropropionic Acid, and 1-Methyl-4-Phenyl-1,2,5,6-Tetrahydropyridine

2000 980 citations Péter Klivènyi, Ole A. Andreassen et al. Journal of Neuroscience profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Alpaslan Dedeoglu United States	36	2.6k	2.3k	1.2k	1.1k	526	62	5.3k
Péter Klivènyi Hungary	40	3.1k 1.2×	2.3k 1.0×	1.9k 1.6×	1.0k 0.9×	780 1.5×	214	7.2k
Giambattista Bonanno Italy	45	3.1k 1.2×	3.4k 1.5×	836 0.7×	685 0.6×	516 1.0×	204	6.8k
William O. Whetsell United States	36	2.5k 1.0×	3.1k 1.4×	1.2k 1.0×	867 0.8×	608 1.2×	105	6.5k
Peter R. Dodd Australia	45	2.8k 1.1×	3.3k 1.4×	619 0.5×	1.6k 1.4×	937 1.8×	170	6.5k
Mario Herrera‐Marschitz Sweden	47	2.6k 1.0×	4.5k 2.0×	1.2k 1.0×	874 0.8×	446 0.8×	209	7.2k
Klaus Fink Germany	39	4.0k 1.5×	2.8k 1.2×	721 0.6×	982 0.9×	1.0k 2.0×	82	7.4k
Rita Sattler United States	27	3.0k 1.1×	2.3k 1.0×	1.4k 1.2×	686 0.6×	723 1.4×	50	5.2k
Catherine Bergeron Canada	31	2.0k 0.8×	1.8k 0.8×	1.4k 1.2×	2.5k 2.2×	1.1k 2.0×	58	5.6k
Henry J. Waldvogel New Zealand	54	3.5k 1.3×	4.1k 1.8×	2.1k 1.7×	1.8k 1.5×	1.2k 2.2×	157	8.0k
Benjamin Drukarch Netherlands	43	1.9k 0.7×	1.6k 0.7×	1.4k 1.2×	809 0.7×	1.1k 2.1×	143	5.4k

Countries citing papers authored by Alpaslan Dedeoglu

Since Specialization

Citations

This map shows the geographic impact of Alpaslan Dedeoglu's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Alpaslan Dedeoglu with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Alpaslan Dedeoglu more than expected).

Fields of papers citing papers by Alpaslan Dedeoglu

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Alpaslan Dedeoglu. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Alpaslan Dedeoglu. The network helps show where Alpaslan Dedeoglu may publish in the future.

Co-authorship network of co-authors of Alpaslan Dedeoglu

This figure shows the co-authorship network connecting the top 25 collaborators of Alpaslan Dedeoglu. A scholar is included among the top collaborators of Alpaslan Dedeoglu based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Alpaslan Dedeoglu. Alpaslan Dedeoglu is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Zhou, Wen, et al.. (2024). Gα11 deficiency increases fibroblast growth factor 23 levels in a mouse model of familial hypocalciuric hypercalcemia. JCI Insight. 9(9). 1 indexed citations

Carreras, Isabel, et al.. (2023). Fingolimod mitigates memory loss in a mouse model of Gulf War Illness amid decreasing the activation of microglia, protein kinase R, and NFκB. NeuroToxicology. 96. 197–206. 4 indexed citations

Qiu, Wei, Xiaofang Zhu, Xiangfen Li, et al.. (2022). The Periodontal Pathogen Fusobacterium nucleatum Exacerbates Alzheimer’s Pathogenesis via Specific Pathways. Frontiers in Aging Neuroscience. 14. 912709–912709. 36 indexed citations

Carreras, Isabel, Nurgül Aytan, Ji‐Kyung Choi, et al.. (2019). Dual dose-dependent effects of fingolimod in a mouse model of Alzheimer’s disease. Scientific Reports. 9(1). 10972–10972. 42 indexed citations

Korkmaz, Orhan Tansel, Hakan Ay, Nurgül Aytan, et al.. (2018). Vasoactive Intestinal Peptide Decreases β-Amyloid Accumulation and Prevents Brain Atrophy in the 5xFAD Mouse Model of Alzheimer’s Disease. Journal of Molecular Neuroscience. 68(3). 389–396. 26 indexed citations

Carreras, Isabel, Nurgül Aytan, Tiffany J. Mellott, et al.. (2017). Anxiety, neuroinflammation, cholinergic and GABAergic abnormalities are early markers of Gulf War illness in a mouse model of the disease. Brain Research. 1681. 34–43. 28 indexed citations

Jenkins, Bruce G., Aijun Zhu, Pekka Poutiainen, et al.. (2015). Functional modulation of G-protein coupled receptors during Parkinson disease-like neurodegeneration. Neuropharmacology. 108. 462–473. 9 indexed citations

Korkmaz, Orhan Tansel, Nurgül Aytan, Isabel Carreras, et al.. (2014). 7,8-Dihydroxyflavone improves motor performance and enhances lower motor neuronal survival in a mouse model of amyotrophic lateral sclerosis. Neuroscience Letters. 566. 286–291. 64 indexed citations

Zhu, Haihao, Hongmei Li, Isabel Carreras, et al.. (2014). Intraperitoneal injection of the pancreatic peptide amylin potently reduces behavioral impairment and brain amyloid pathology in murine models of Alzheimer’s disease. Molecular Psychiatry. 20(2). 252–262. 92 indexed citations

10.

Carreras, Isabel, Ann C. McKee, Ji‐Kyung Choi, et al.. (2013). R-flurbiprofen improves tau, but not Aß pathology in a triple transgenic model of Alzheimer's disease. Brain Research. 1541. 115–127. 29 indexed citations

11.

Choi, Ji‐Kyung, Isabel Carreras, Alpaslan Dedeoglu, & Bruce G. Jenkins. (2010). Detection of increased scyllo-inositol in brain with magnetic resonance spectroscopy after dietary supplementation in Alzheimer’s disease mouse models. Neuropharmacology. 59(4-5). 353–357. 23 indexed citations

12.

Carreras, Isabel, Sinan Yürüker, Nurgül Aytan, et al.. (2009). Moderate exercise delays the motor performance decline in a transgenic model of ALS. Brain Research. 1313. 192–201. 74 indexed citations

13.

Stack, Edward C., Alpaslan Dedeoglu, Karen Smith, et al.. (2007). Neuroprotective Effects of Synaptic Modulation in Huntington's Disease R6/2 Mice. Journal of Neuroscience. 27(47). 12908–12915. 61 indexed citations

14.

Choi, Ji‐Kyung, Alpaslan Dedeoglu, & Bruce G. Jenkins. (2007). Application of MRS to mouse models of neurodegenerative illness. NMR in Biomedicine. 20(3). 216–237. 99 indexed citations

15.

Jenkins, Bruce G., Ole A. Andreassen, Alpaslan Dedeoglu, et al.. (2005). Effects of CAG repeat length, HTT protein length and protein context on cerebral metabolism measured using magnetic resonance spectroscopy in transgenic mouse models of Huntington's disease. Journal of Neurochemistry. 95(2). 553–562. 60 indexed citations

16.

Brownell, Anna‐Liisa, Y. Iris Chen, Meixiang Yu, et al.. (2004). 3‐Nitropropionic acid‐induced neurotoxicity – assessed by ultra high resolution positron emission tomography with comparison to magnetic resonance spectroscopy. Journal of Neurochemistry. 89(5). 1206–1214. 24 indexed citations

17.

Ferrante, Robert J., Alpaslan Dedeoglu, James K. Kubilus, et al.. (2002). Cytochrome C and Caspase-9 Expression in Huntington 's Disease. NeuroMolecular Medicine. 1(3). 183–196. 104 indexed citations

18.

Andreassen, Ole A., Robert J. Ferrante, Péter Klivènyi, et al.. (2001). Transgenic ALS Mice Show Increased Vulnerability to the Mitochondrial Toxins MPTP and 3-Nitropropionic Acid. Experimental Neurology. 168(2). 356–363. 19 indexed citations

19.

Ferrante, Robert J., et al.. (2001). Therapeutic Efficacy of EGb761 (Gingko biloba Extract) in a Transgenic Mouse Model of Amyotrophic Lateral Sclerosis. Journal of Molecular Neuroscience. 17(1). 89–96. 55 indexed citations

20.

Andreassen, Ole A., Robert J. Ferrante, Duncan B. Hughes, et al.. (2000). Malonate and 3‐Nitropropionic Acid Neurotoxicity Are Reduced in Transgenic Mice Expressing a Caspase‐1 Dominant‐Negative Mutant. Journal of Neurochemistry. 75(2). 847–852. 36 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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