Robert J. Ferrante

34.2k total citations · 10 hit papers
160 papers, 26.2k citations indexed

About

Robert J. Ferrante is a scholar working on Cellular and Molecular Neuroscience, Molecular Biology and Neurology. According to data from OpenAlex, Robert J. Ferrante has authored 160 papers receiving a total of 26.2k indexed citations (citations by other indexed papers that have themselves been cited), including 102 papers in Cellular and Molecular Neuroscience, 95 papers in Molecular Biology and 77 papers in Neurology. Recurrent topics in Robert J. Ferrante's work include Genetic Neurodegenerative Diseases (88 papers), Mitochondrial Function and Pathology (69 papers) and Amyotrophic Lateral Sclerosis Research (34 papers). Robert J. Ferrante is often cited by papers focused on Genetic Neurodegenerative Diseases (88 papers), Mitochondrial Function and Pathology (69 papers) and Amyotrophic Lateral Sclerosis Research (34 papers). Robert J. Ferrante collaborates with scholars based in United States, France and Switzerland. Robert J. Ferrante's co-authors include M. Flint Beal, Neil W. Kowall, M. Flint Beal, Steven M. Hersch, Ole A. Andreassen, Edward P. Richardson, Hoon Ryu, Jean‐Paul Vonsattel, Edward D. Bird and Alpaslan Dedeoglu and has published in prestigious journals such as Nature, Science and Proceedings of the National Academy of Sciences.

In The Last Decade

Robert J. Ferrante

160 papers receiving 25.7k citations

Hit Papers

Neuropathological Classification of Huntingtonʼs Disease 1985 2026 1998 2012 1985 2000 1996 2002 2000 500 1000 1.5k 2.0k
What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

  1. Neuropathological Classification of Huntingtonʼs Disease
    1985 2.0k citations Robert J. Ferrante et al. profile →
  2. Mice Deficient in Cellular Glutathione Peroxidase Show Increased Vulnerability to Malonate, 3-Nitropropionic Acid, and 1-Methyl-4-Phenyl-1,2,5,6-Tetrahydropyridine
    2000 980 citations Péter Klivènyi, Ole A. Andreassen et al. Journal of Neuroscience profile →
  3. Motor neurons in Cu/Zn superoxide dismutase-deficient mice develop normally but exhibit enhanced cell death after axonal injury
    1996 966 citations Neil W. Kowall, Robert J. Ferrante et al. profile →
  4. Minocycline inhibits cytochrome c release and delays progression of amyotrophic lateral sclerosis in mice
    2002 877 citations Irina G. Stavrovskaya, Robert J. Ferrante et al. profile →
  5. Minocycline inhibits caspase-1 and caspase-3 expression and delays mortality in a transgenic mouse model of Huntington disease
    2000 869 citations Robert J. Ferrante, Robert M. Friedlander et al. profile →
  6. Wild-Type Nonneuronal Cells Extend Survival of SOD1 Mutant Motor Neurons in ALS Mice
    2003 855 citations Albrecht M. Clement, Minh Dang Nguyen et al. Science profile →
  7. Nuclear and Neuropil Aggregates in Huntington’s Disease: Relationship to Neuropathology
    1999 704 citations Robert J. Ferrante, Steven M. Hersch et al. Journal of Neuroscience profile →
  8. Evidence of Increased Oxidative Damage in Both Sporadic and Familial Amyotrophic Lateral Sclerosis
    1997 635 citations Robert J. Ferrante, Susan Browne et al. Journal of Neurochemistry profile →
  9. Histone Deacetylase Inhibition by Sodium Butyrate Chemotherapy Ameliorates the Neurodegenerative Phenotype in Huntington's Disease Mice
    2003 559 citations Robert J. Ferrante, James K. Kubilus et al. Journal of Neuroscience profile →
  10. Neuroprotective effects of creatine in a transgenic animal model of amyotrophic lateral sclerosis
    1999 554 citations Péter Klivènyi, Robert J. Ferrante et al. profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Robert J. Ferrante United States 85 14.3k 13.0k 9.8k 3.8k 3.1k 160 26.2k
M. Flint Beal United States 105 19.9k 1.4× 15.1k 1.2× 11.7k 1.2× 7.8k 2.1× 4.2k 1.4× 273 36.4k
Lee J. Martin United States 76 9.0k 0.6× 8.7k 0.7× 5.3k 0.5× 4.3k 1.1× 3.2k 1.0× 242 21.6k
Patrik Brundin Sweden 97 14.5k 1.0× 17.7k 1.4× 15.5k 1.6× 5.6k 1.5× 5.2k 1.7× 363 35.5k
Thomas Klockgether Germany 80 8.8k 0.6× 10.2k 0.8× 10.7k 1.1× 3.8k 1.0× 4.1k 1.3× 386 23.9k
Neil W. Kowall United States 66 7.6k 0.5× 6.5k 0.5× 6.0k 0.6× 4.6k 1.2× 2.4k 0.8× 192 18.0k
Serge Przedborski United States 110 14.3k 1.0× 15.3k 1.2× 21.8k 2.2× 7.8k 2.1× 8.4k 2.7× 261 41.6k
Richard L. M. Faull New Zealand 89 11.1k 0.8× 15.0k 1.2× 5.2k 0.5× 4.5k 1.2× 3.9k 1.3× 417 27.4k
Jeffrey D. Rothstein United States 91 15.7k 1.1× 15.0k 1.2× 12.4k 1.3× 5.0k 1.3× 8.2k 2.7× 235 37.2k
Michael K. Lee United States 61 8.0k 0.6× 5.6k 0.4× 8.0k 0.8× 7.6k 2.0× 3.2k 1.0× 155 20.0k
Leonard Petrucelli United States 80 11.2k 0.8× 4.7k 0.4× 12.9k 1.3× 7.1k 1.9× 3.2k 1.0× 232 23.2k

Countries citing papers authored by Robert J. Ferrante

Since Specialization
Citations

This map shows the geographic impact of Robert J. Ferrante's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Robert J. Ferrante with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Robert J. Ferrante more than expected).

Fields of papers citing papers by Robert J. Ferrante

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Robert J. Ferrante. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Robert J. Ferrante. The network helps show where Robert J. Ferrante may publish in the future.

Co-authorship network of co-authors of Robert J. Ferrante

This figure shows the co-authorship network connecting the top 25 collaborators of Robert J. Ferrante. A scholar is included among the top collaborators of Robert J. Ferrante based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Robert J. Ferrante. Robert J. Ferrante is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Taylor, David, Roger Moser, Etienne Régulier, et al.. (2013). MAP Kinase Phosphatase 1 (MKP-1/DUSP1) Is Neuroprotective in Huntington's Disease via Additive Effects of JNK and p38 Inhibition. Journal of Neuroscience. 33(6). 2313–2325. 94 indexed citations
2.
Hu, Yi, Vanita Chopra, Raman Chopra, et al.. (2011). Transcriptional modulator H2A histone family, member Y ( H2AFY ) marks Huntington disease activity in man and mouse. Proceedings of the National Academy of Sciences. 108(41). 17141–17146. 76 indexed citations
3.
Wang, Xin, Kerry Cormier, Karen Smith, et al.. (2011). The Melatonin MT1 Receptor Axis Modulates Mutant Huntingtin-Mediated Toxicity. Journal of Neuroscience. 31(41). 14496–14507. 156 indexed citations
4.
Faideau, Mathilde, Jae-Hwan Kim, Kerry Cormier, et al.. (2010). In vivo expression of polyglutamine-expanded huntingtin by mouse striatal astrocytes impairs glutamate transport: a correlation with Huntington's disease subjects. Human Molecular Genetics. 19(15). 3053–3067. 250 indexed citations
5.
Wang, Xin, Shan Zhu, Irina G. Stavrovskaya, et al.. (2008). Inhibitors of CytochromecRelease with Therapeutic Potential for Huntington's Disease. Journal of Neuroscience. 28(38). 9473–9485. 100 indexed citations
6.
Stack, Edward C., Jin-Ho Kim, Steven J. Del Signore, et al.. (2008). Therapeutic attenuation of mitochondrial dysfunction and oxidative stress in neurotoxin models of Parkinson's disease. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1782(3). 151–162. 47 indexed citations
7.
Stack, Edward C., Alpaslan Dedeoglu, Karen Smith, et al.. (2007). Neuroprotective Effects of Synaptic Modulation in Huntington's Disease R6/2 Mice. Journal of Neuroscience. 27(47). 12908–12915. 61 indexed citations
8.
Stack, Edward C., Karen Smith, Hoon Ryu, et al.. (2005). Combination therapy using minocycline and coenzyme Q10 in R6/2 transgenic Huntington's disease mice. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1762(3). 373–380. 94 indexed citations
9.
Ryu, Hoon, Karen Smith, Sandra Camelo, et al.. (2005). Sodium phenylbutyrate prolongs survival and regulates expression of anti‐apoptotic genes in transgenic amyotrophic lateral sclerosis mice. Journal of Neurochemistry. 93(5). 1087–1098. 290 indexed citations
10.
Ferrante, Robert J., Hoon Ryu, James K. Kubilus, et al.. (2004). Chemotherapy for the Brain: The Antitumor Antibiotic Mithramycin Prolongs Survival in a Mouse Model of Huntington's Disease. Journal of Neuroscience. 24(46). 10335–10342. 172 indexed citations
11.
Hersch, Steven M. & Robert J. Ferrante. (2004). Translating therapies for Huntington’s disease from genetic animal models to clinical trials. Neurotherapeutics. 1(3). 298–306. 2 indexed citations
12.
Hersch, Steven M. & Robert J. Ferrante. (2004). Translating therapies for Huntington’s disease from genetic animal models to clinical trials. PubMed. 1(3). 298–306. 55 indexed citations
13.
Clement, Albrecht M., Minh Dang Nguyen, Elizabeth A. Roberts, et al.. (2003). Wild-Type Nonneuronal Cells Extend Survival of SOD1 Mutant Motor Neurons in ALS Mice. Science. 302(5642). 113–117. 855 indexed citations breakdown →
14.
Ryu, Hoon, Jung-Hee Lee, Beatrix A. Olofsson, et al.. (2003). Histone deacetylase inhibitors prevent oxidative neuronal death independent of expanded polyglutamine repeats via an Sp1-dependent pathway. Proceedings of the National Academy of Sciences. 100(7). 4281–4286. 225 indexed citations
15.
Andreassen, Ole A., Robert J. Ferrante, Péter Klivènyi, et al.. (2001). Transgenic ALS Mice Show Increased Vulnerability to the Mitochondrial Toxins MPTP and 3-Nitropropionic Acid. Experimental Neurology. 168(2). 356–363. 19 indexed citations
16.
Holbert, Sébastien, Adam Rosenblatt, Cheryl L. Wellington, et al.. (2001). The Gln-Ala repeat transcriptional activator CA150 interacts with huntingtin: Neuropathologic and genetic evidence for a role in Huntington's disease pathogenesis. Proceedings of the National Academy of Sciences. 98(4). 1811–1816. 143 indexed citations
17.
Eliasson, Mikael J. L., Zhihong Huang, Robert J. Ferrante, et al.. (1999). Neuronal Nitric Oxide Synthase Activation and Peroxynitrite Formation in Ischemic Stroke Linked to Neural Damage. Journal of Neuroscience. 19(14). 5910–5918. 309 indexed citations
18.
Beal, M. Flint, Robert J. Ferrante, Susan Browne, et al.. (1997). Increased 3‐nitrotyrosine in both sporadic and familial amyotrophic lateral sclerosis. Annals of Neurology. 42(4). 644–654. 479 indexed citations
19.
Ferrante, Robert J., Robert W. Hobson, Masayuki Miyasaka, D. Neil Granger, & Walter N. Durán. (1996). Inhibition of white blood cell adhesion at reperfusion decreases tissue damage in postischemic striated muscle. Journal of Vascular Surgery. 24(2). 187–193. 22 indexed citations
20.
Ferrante, Robert J., et al.. (1993). Excitotoxin Lesions in Primates as a Model for Huntington's Disease: Histopathologic and Neurochemical Characterization. Experimental Neurology. 119(1). 46–71. 185 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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