Sepehr Ehsani

847 total citations
19 papers, 541 citations indexed

About

Sepehr Ehsani is a scholar working on Molecular Biology, Nutrition and Dietetics and Neurology. According to data from OpenAlex, Sepehr Ehsani has authored 19 papers receiving a total of 541 indexed citations (citations by other indexed papers that have themselves been cited), including 16 papers in Molecular Biology, 10 papers in Nutrition and Dietetics and 7 papers in Neurology. Recurrent topics in Sepehr Ehsani's work include Prion Diseases and Protein Misfolding (12 papers), Trace Elements in Health (10 papers) and Neurological diseases and metabolism (7 papers). Sepehr Ehsani is often cited by papers focused on Prion Diseases and Protein Misfolding (12 papers), Trace Elements in Health (10 papers) and Neurological diseases and metabolism (7 papers). Sepehr Ehsani collaborates with scholars based in Canada, United States and United Kingdom. Sepehr Ehsani's co-authors include Gerold Schmitt‐Ulms, David Westaway, Joel C. Watts, Holger Wille, Cosmin L. Pocanschi, Mohadeseh Mehrabian, Priyanka Narayan, Susan Lindquist, George A. Carlson and Yu Bai and has published in prestigious journals such as PLoS ONE, Journal of Molecular Biology and Scientific Reports.

In The Last Decade

Sepehr Ehsani

18 papers receiving 540 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Sepehr Ehsani Canada 13 376 244 162 65 60 19 541
Kateřina Veselá Czechia 15 493 1.3× 54 0.2× 9 0.1× 39 0.6× 32 0.5× 34 722
Guo-Fen Tu Australia 8 444 1.2× 54 0.2× 23 0.1× 48 0.7× 35 0.6× 10 632
Eui-Ju Choi South Korea 7 487 1.3× 73 0.3× 17 0.1× 61 0.9× 5 0.1× 8 694
Nancy Davis United States 11 276 0.7× 102 0.4× 13 0.1× 46 0.7× 8 0.1× 14 625
Lukáš Stibůrek Czechia 13 556 1.5× 63 0.3× 11 0.1× 63 1.0× 18 0.3× 15 660
L E Rutherford United States 6 371 1.0× 26 0.1× 34 0.2× 115 1.8× 15 0.3× 7 589
Raya Dana Israel 8 288 0.8× 21 0.1× 47 0.3× 232 3.6× 12 0.2× 10 619
Christopher Martin United States 7 394 1.0× 76 0.3× 8 0.0× 5 0.1× 26 0.4× 9 486
Rebecca K. Donegan United States 11 272 0.7× 13 0.1× 25 0.2× 62 1.0× 24 0.4× 13 392
Michal Hofer Czechia 16 198 0.5× 12 0.0× 27 0.2× 33 0.5× 21 0.3× 40 539

Countries citing papers authored by Sepehr Ehsani

Since Specialization
Citations

This map shows the geographic impact of Sepehr Ehsani's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Sepehr Ehsani with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Sepehr Ehsani more than expected).

Fields of papers citing papers by Sepehr Ehsani

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Sepehr Ehsani. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Sepehr Ehsani. The network helps show where Sepehr Ehsani may publish in the future.

Co-authorship network of co-authors of Sepehr Ehsani

This figure shows the co-authorship network connecting the top 25 collaborators of Sepehr Ehsani. A scholar is included among the top collaborators of Sepehr Ehsani based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Sepehr Ehsani. Sepehr Ehsani is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

19 of 19 papers shown
1.
Hamidabadi, Hatef Ghasemi, Maryam Nazm Bojnordi, & Sepehr Ehsani. (2021). Recovery potential of transplanted oligoprogenitor cells derived from human dental pulp stem cells in Lysophosphatidyl choline demyelination mode. Bratislavské lekárske listy/Bratislava medical journal. 122(9). 621–625. 1 indexed citations
2.
Schmitt‐Ulms, Gerold, Mohadeseh Mehrabian, Declan Williams, & Sepehr Ehsani. (2021). The IDIP framework for assessing protein function and its application to the prion protein. Biological reviews/Biological reviews of the Cambridge Philosophical Society. 96(5). 1907–1932. 5 indexed citations
3.
5.
Mehrabian, Mohadeseh, Declan Williams, Xinzhu Wang, et al.. (2017). A ZIP6-ZIP10 heteromer controls NCAM1 phosphorylation and integration into focal adhesion complexes during epithelial-to-mesenchymal transition. Scientific Reports. 7(1). 40313–40313. 21 indexed citations
6.
Narayan, Priyanka, Sepehr Ehsani, & Susan Lindquist. (2014). Combating neurodegenerative disease with chemical probes and model systems. Nature Chemical Biology. 10(11). 911–920. 36 indexed citations
7.
Mehrabian, Mohadeseh, Sepehr Ehsani, & Gerold Schmitt‐Ulms. (2014). An emerging role of the cellular prion protein as a modulator of a morphogenetic program underlying epithelial-to-mesenchymal transition. Frontiers in Cell and Developmental Biology. 2. 53–53. 14 indexed citations
8.
Pocanschi, Cosmin L., Sepehr Ehsani, Mohadeseh Mehrabian, et al.. (2013). The ZIP5 Ectodomain Co-Localizes with PrP and May Acquire a PrP-Like Fold That Assembles into a Dimer. PLoS ONE. 8(9). e72446–e72446. 20 indexed citations
9.
Marimuthu, Parthiban, Deena M.A. Gendoo, Sepehr Ehsani, et al.. (2012). PrionHome: A Database of Prions and Other Sequences Relevant to Prion Phenomena. PLoS ONE. 7(2). e31785–e31785. 21 indexed citations
10.
Ehsani, Sepehr, Mohadeseh Mehrabian, Cosmin L. Pocanschi, & Gerold Schmitt‐Ulms. (2012). The ZIP-prion connection. Prion. 6(4). 317–321. 10 indexed citations
11.
Ehsani, Sepehr, William Reginold, Cosmin L. Pocanschi, et al.. (2012). LIV-1 ZIP Ectodomain Shedding in Prion-Infected Mice Resembles Cellular Response to Transition Metal Starvation. Journal of Molecular Biology. 422(4). 556–574. 27 indexed citations
12.
Ehsani, Sepehr, et al.. (2011). Evidence for Retrogene Origins of the Prion Gene Family. PLoS ONE. 6(10). e26800–e26800. 29 indexed citations
13.
Knobbe‐Thomsen, Christiane B., Timothy J. Revett, Yu Bai, et al.. (2011). Choice of Biological Source Material Supersedes Oxidative Stress in Its Influence on DJ-1 in Vivo Interactions with Hsp90. Journal of Proteome Research. 10(10). 4388–4404. 21 indexed citations
14.
Ehsani, Sepehr, Cosmin L. Pocanschi, Joel C. Watts, et al.. (2010). Family reunion – The ZIP/prion gene family. Progress in Neurobiology. 93(3). 405–420. 35 indexed citations
15.
Schmitt‐Ulms, Gerold, Sepehr Ehsani, Joel C. Watts, David Westaway, & Holger Wille. (2009). Evolutionary Descent of Prion Genes from the ZIP Family of Metal Ion Transporters. PLoS ONE. 4(9). e7208–e7208. 105 indexed citations
17.
Watts, Joel C., Yu Bai, Sepehr Ehsani, et al.. (2009). Interactome Analyses Identify Ties of PrPC and Its Mammalian Paralogs to Oligomannosidic N-Glycans and Endoplasmic Reticulum-Derived Chaperones. PLoS Pathogens. 5(10). e1000608–e1000608. 109 indexed citations
18.
Ehsani, Sepehr, Mojgan Hodaie, Norbert J. Liebsch, Fred Gentili, & Tim‐Rasmus Kiehl. (2008). Anaplastic glioma after high-dose proton–photon radiation treatment for low-grade skull base chondrosarcoma. Journal of Neuro-Oncology. 88(2). 231–236. 1 indexed citations
19.
Ehsani, Sepehr, et al.. (2007). Creation of a retrospective searchable neuropathologic database from print archives at Toronto's University Health Network. Laboratory Investigation. 88(1). 89–93. 2 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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