Paul Dent
- Oncology top 0.2%
- Cancer-related Molecular Pathways 52
- Molecular Biology top 0.1%
- Cell death mechanisms and regulation 75
- Histone Deacetylase Inhibitors Research 62
- Melanoma and MAPK Pathways 49
- Protein Kinase Regulation and GTPase Signaling 45
- PI3K/AKT/mTOR signaling in cancer 34
- Cell Biology top 0.2%
- Endoplasmic Reticulum Stress and Disease 37
- Cancer Research top 0.5%
- Hematology top 0.5%
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- Autophagy in Disease and Therapy 55
- Co-authors
- Steven GrantMohamed RahmaniPaul B. FisherThomas W. SturgillAdly YacoubYun DaiKristoffer ValerieLaurence Booth
- Partner nations
- United StatesRussiaCanada
In The Last Decade
Paul Dent
387 papers receiving 22.8k citations
Hit Papers
Peers
Comparison fields: 5 of 153
- Oncology 7.0k
- Molecular Biology 15.6k
- Cell Biology 2.8k
- Cancer Research 2.4k
- Hematology 1.6k
Countries citing papers authored by Paul Dent
This map shows the geographic impact of Paul Dent's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Paul Dent with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Paul Dent more than expected).
Fields of papers citing papers by Paul Dent
This network shows the impact of papers produced by Paul Dent. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Paul Dent. The network helps show where Paul Dent may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Paul Dent, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2024 | 2 | |
| 2 | 2016 | 33 | |
| 3 | 2012 | 46 | |
| 4 | 2012 | 79 | |
| 5 | 2011 | 97 | |
| 6 | 2011 | 101 | |
| 7 | 2010 | 86 | |
| 8 | 2010 | 74 | |
| 9 | 2010 | 102 | |
| 10 | 2010 | 89 | |
| 11 | 2010 | 16 | |
| 12 | 2009 | 154 | |
| 13 | 2009 | 40 | |
| 14 | 2008 | 101 | |
| 15 | 2008 | 41 | |
| 16 | 2008 | 68 | |
| 17 | 2007 | 145 | |
| 18 | 2007 | 132 | |
| 19 | 2005 | 79 | |
| 20 | 1999 | 44 |
About Paul Dent
Paul Dent is a scholar working on Oncology, Molecular Biology, Cell Biology, Physiology and Hematology, having authored 388 papers that have together received 23.1k indexed citations. Recurring topics across this work include Cell death mechanisms and regulation (75 papers), Histone Deacetylase Inhibitors Research (62 papers), Autophagy in Disease and Therapy (55 papers), Cancer-related Molecular Pathways (52 papers), Melanoma and MAPK Pathways (49 papers), Protein Kinase Regulation and GTPase Signaling (45 papers), Endoplasmic Reticulum Stress and Disease (37 papers) and PI3K/AKT/mTOR signaling in cancer (34 papers). The work is most often cited by research in Oncology (7.0k citations), Molecular Biology (15.6k citations), Cell Biology (2.8k citations), Cancer Research (2.4k citations) and Hematology (1.6k citations). Paul Dent has collaborated with scholars based in United States, Russia and Canada. Frequent co-authors include Steven Grant, Mohamed Rahmani, Paul B. Fisher, Thomas W. Sturgill, Adly Yacoub, Yun Dai, Kristoffer Valerie, Laurence Booth, Rupert Schmidt‐Ullrich and Michael P. Hagan. Their work appears in journals such as Cancer Biology & Therapy, Molecular Pharmacology, Blood, Molecular Cancer Therapeutics and Oncogene.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.