Natalie Burrows

1.4k citations
20 papers · 728 · h-index 14

Impact in

Papers in

    • Cancer, Hypoxia, and Metabolism 5
    • T-cell and B-cell Immunology 2
    • Complement system in diseases 1

Natalie Burrows

20 papers receiving 718 citations

Peers

Natalie Burrows
Comparison fields: 5 of 48
  • Cancer Research 110
  • Endocrinology, Diabetes and Metabolism 72
  • Molecular Biology 170
  • Pathology and Forensic Medicine 31
  • Genetics 13
Replace Junaid Abdulghani with:
Junaid Abdulghani United States
Hossein Tabatabaeian Iran
Parvin F. Peddi United States
Martin Scherzer Austria
Qing Shao China
Kateřina Bouchalová Czechia
Ursula Cesta Incani Italy
Sagarika Ray United States
Marc‐Olivier Turgeon Canada
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Citations per field
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Citations per year

Countries citing papers authored by Natalie Burrows

Since Specialization
Citations

This map shows the geographic impact of Natalie Burrows's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Natalie Burrows with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Natalie Burrows more than expected).

Fields of papers citing papers by Natalie Burrows

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Natalie Burrows. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Natalie Burrows. The network helps show where Natalie Burrows may publish in the future.

Co-authors

The 25 scholars most cited alongside Natalie Burrows, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Natalie Burrows Line = papers co-authored together Natalie Burrows links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown
#Work
1 2012151
2 200990
3 202067
4 202161
5 201154
6 201753
7 201152
8 201743
9 201634
10 202223
11 201321
12 201721
13 201617
14 201314
15 201611
16 20147
17 20214
18
Dynamic regulation of hypoxia-inducible factor-1 alpha activity is essential for normal B cell development
20202
19 20252
20 20101

About Natalie Burrows

Natalie Burrows is a scholar working on Cancer Research, Immunology, Pulmonary and Respiratory Medicine, Molecular Biology and Nuclear Energy and Engineering, having authored 20 papers that have together received 728 indexed citations. Recurring topics across this work include Cancer, Hypoxia, and Metabolism (5 papers), T-cell and B-cell Immunology (2 papers), Renal cell carcinoma treatment (2 papers), Complement system in diseases (1 paper) and Enzyme function and inhibition (1 paper). The work is most often cited by research in Cancer Research (110 citations), Endocrinology, Diabetes and Metabolism (72 citations), Molecular Biology (170 citations), Pathology and Forensic Medicine (31 citations) and Genetics (13 citations). Natalie Burrows has collaborated with scholars based in United Kingdom, Germany and Belgium. Frequent co-authors include Kaye Janine Williams, Georg E. Brabant, Patrick Henry Maxwell, Julia Resch, Brian A. Telfer, Roben G. Gieling, Jean‐Yves Winum, Andrea Scozzafava, Claudiu Trandafir Supuran and Fabrizio Carta. Their work appears in journals such as Oncotarget, BMC Cancer, Molecular Cancer Therapeutics, Nature Immunology and Radiotherapy and Oncology.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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