John A. McGrath

33.0k total citations · 2 hit papers
562 papers, 17.0k citations indexed

About

John A. McGrath is a scholar working on Cell Biology, Molecular Biology and Pathology and Forensic Medicine. According to data from OpenAlex, John A. McGrath has authored 562 papers receiving a total of 17.0k indexed citations (citations by other indexed papers that have themselves been cited), including 379 papers in Cell Biology, 202 papers in Molecular Biology and 139 papers in Pathology and Forensic Medicine. Recurrent topics in John A. McGrath's work include Skin and Cellular Biology Research (356 papers), Autoimmune Bullous Skin Diseases (127 papers) and Cell Adhesion Molecules Research (87 papers). John A. McGrath is often cited by papers focused on Skin and Cellular Biology Research (356 papers), Autoimmune Bullous Skin Diseases (127 papers) and Cell Adhesion Molecules Research (87 papers). John A. McGrath collaborates with scholars based in United Kingdom, United States and Japan. John A. McGrath's co-authors include Jouni Uitto, Robin A.J. Eady, Jemima E. Mellerio, Angela M. Christiano, Joey E. Lai‐Cheong, Akemi Ishida‐Yamamoto, James R. McMillan, Leena Bruckner‐Tuderman, E. Birgitte Lane and Irene M. Leigh and has published in prestigious journals such as Science, New England Journal of Medicine and Proceedings of the National Academy of Sciences.

In The Last Decade

John A. McGrath

539 papers receiving 16.6k citations

Hit Papers

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What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

The classification of inherited epidermolysis bullosa (EB): Report of the Third International Consensus Meeting on Diagnosis and Classification of EB

2008 598 citations Jo‐David Fine, Robin A.J. Eady et al. Journal of the American Academy of Dermatology profile →
Epidermolysis bullosa

2020 210 citations John A. McGrath, Jemima E. Mellerio et al. profile →

Author Peers

Peers are selected by citation overlap in the author's most active subfields. citations · hero ref

Author						Papers	Cites
John A. McGrath	9.6k	6.5k	4.2k	3.2k	2.4k	562	17.0k
Angela M. Christiano	8.4k 0.9×	6.7k 1.0×	3.6k 0.9×	2.6k 0.8×	4.0k 1.7×	355	18.4k
Leena Bruckner‐Tuderman	7.0k 0.7×	3.8k 0.6×	4.2k 1.0×	2.0k 0.6×	1.1k 0.5×	316	13.0k
Hiroshi Shimizu	4.4k 0.5×	3.7k 0.6×	4.3k 1.0×	1.4k 0.4×	2.9k 1.2×	538	14.2k
E. Birgitte Lane	7.4k 0.8×	5.9k 0.9×	1.2k 0.3×	1.9k 0.6×	2.0k 0.8×	222	13.1k
Peter M. Steinert	12.8k 1.3×	9.8k 1.5×	972 0.2×	1.8k 0.6×	2.8k 1.2×	226	21.6k
Robin A.J. Eady	6.5k 0.7×	3.1k 0.5×	2.9k 0.7×	1.7k 0.5×	1.0k 0.4×	189	9.4k
Roland Moll	6.1k 0.6×	5.7k 0.9×	1.1k 0.3×	1.4k 0.5×	2.1k 0.9×	147	14.7k
Jouni Uitto	16.5k 1.7×	12.0k 1.8×	7.5k 1.8×	9.1k 2.9×	5.7k 2.4×	812	37.2k
Giovanna Zambruno	3.5k 0.4×	3.6k 0.6×	2.5k 0.6×	1.1k 0.4×	1.2k 0.5×	266	10.1k
Dennis R. Roop	7.5k 0.8×	12.7k 1.9×	835 0.2×	2.6k 0.8×	2.6k 1.1×	276	21.0k

Countries citing papers authored by John A. McGrath

Since Specialization

Citations

This map shows the geographic impact of John A. McGrath's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by John A. McGrath with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites John A. McGrath more than expected).

Fields of papers citing papers by John A. McGrath

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by John A. McGrath. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by John A. McGrath. The network helps show where John A. McGrath may publish in the future.

Co-authorship network of co-authors of John A. McGrath

This figure shows the co-authorship network connecting the top 25 collaborators of John A. McGrath. A scholar is included among the top collaborators of John A. McGrath based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with John A. McGrath. John A. McGrath is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Dimairo, Munyaradzi, Rachel Glover, Diana Papaioannou, et al.. (2025). Mesenchymal intravenous stromal cell infusions in children with recessive dystrophic epidermolysis bullosa: MissionEB protocol for a randomised, double-blinded, placebo-controlled, two-centre, crossover trial with an internal phase I dose de-escalation phase and open-label extension. BMJ Open. 15(5). e089857–e089857.

Jeffs, E., Elizabeth Pillay, Alessandra Bisquera, et al.. (2024). Costs of UK Community Care for Individuals with Recessive Dystrophic Epidermolysis Bullosa: Findings of the Prospective Epidermolysis Bullosa Longitudinal Evaluation Study. SHILAP Revista de lepidopterología. 4(1). e314–e314. 2 indexed citations

McGrath, John A., et al.. (2024). Real-World Experience of Using Dupilumab and Jak Inhibitors to Manage Pruritus in Epidermolysis Bullosa Pruriginosa. SHILAP Revista de lepidopterología. 4(5). e445–e445. 3 indexed citations

Eide, Cindy, et al.. (2024). Beyond the Surface: A Narrative Review Examining the Systemic Impacts of Recessive Dystrophic Epidermolysis Bullosa. Journal of Investigative Dermatology. 144(9). 1943–1953. 6 indexed citations

Kim, Young‐Ah, et al.. (2023). Topical gene editing therapeutics using lipid nanoparticles: ‘gene creams’ for genetic skin diseases?. British Journal of Dermatology. 190(5). 617–627. 7 indexed citations

Mellerio, Jemima E., Elizabeth Pillay, Alessandra Bisquera, et al.. (2023). Itch in recessive dystrophic epidermolysis bullosa: findings of PEBLES, a prospective register study. Orphanet Journal of Rare Diseases. 18(1). 235–235. 12 indexed citations

Fassihi, Hiva, et al.. (2023). Systematic review of the clinical characteristics and natural history of solar urticaria. Journal of the American Academy of Dermatology. 89(1). 138–140. 6 indexed citations

Niebergall‐Roth, Elke, Natasha Y. Frank, Christoph Ganss, et al.. (2023). ABCB5+ mesenchymal stromal cells facilitate complete and durable wound closure in recessive dystrophic epidermolysis bullosa. Cytotherapy. 25(7). 782–788. 10 indexed citations

Lwin, Su M., et al.. (2023). Innovations in the Treatment of Dystrophic Epidermolysis Bullosa (DEB): Current Landscape and Prospects. Therapeutics and Clinical Risk Management. Volume 19. 455–473. 31 indexed citations

10.

Newby, Gregory A., et al.. (2023). Highly efficient biallelic correction of homozygous COL7A1 mutation using ABE8e adenine base editor. British Journal of Dermatology. 190(4). 583–585. 1 indexed citations

11.

Prodinger, Christine, Jemima E. Mellerio, John A. McGrath, et al.. (2021). The natural history of laryngo‐onycho‐cutaneous syndrome: A case series of six pediatric patients and literature review. Pediatric Dermatology. 38(5). 1094–1101. 5 indexed citations

12.

Ung, Chuin Ying, Alexandros Onoufriadis, Maddy Parsons, John A. McGrath, & Tanya J. Shaw. (2021). Metabolic perturbations in fibrosis disease. The International Journal of Biochemistry & Cell Biology. 139. 106073–106073. 37 indexed citations

13.

Mohamad, Janan, Arti Nanda, Mor Pavlovsky, et al.. (2020). Phenotypic suppression of acral peeling skin syndrome in a patient with autosomal recessive congenital ichthyosis. Experimental Dermatology. 29(8). 742–748. 2 indexed citations

14.

Ebens, Christen L., John A. McGrath, Douglas R. Keene, et al.. (2020). Immune tolerance of allogeneic haematopoietic cell transplantation supports donor epidermal grafting of recessive dystrophic epidermolysis bullosa chronic wounds*. British Journal of Dermatology. 184(6). 1161–1169. 13 indexed citations

15.

Di, Wei‐Li, Jemima E. Mellerio, John Harper, et al.. (2013). Phase I Study Protocol for Ex Vivo Lentiviral Gene Therapy for the Inherited Skin Disease, Netherton Syndrome. PubMed. 24(4). 182–190. 27 indexed citations

16.

Pourreyron, Céline, Julio C. Salas‐Alanís, Jasbani H.S. Dayal, et al.. (2012). Fibroblast-Derived Dermal Matrix Drives Development of Aggressive Cutaneous Squamous Cell Carcinoma in Patients with Recessive Dystrophic Epidermolysis Bullosa. Cancer Research. 72(14). 3522–3534. 103 indexed citations

17.

Tamai, Katsuto, Takehiko Yamazaki, Takenao Chino, et al.. (2011). PDGFRα-positive cells in bone marrow are mobilized by high mobility group box 1 (HMGB1) to regenerate injured epithelia. Proceedings of the National Academy of Sciences. 108(16). 6609–6614. 181 indexed citations

18.

Martins, Vera, Mei Chen, Karin J. Purdie, et al.. (2009). Increased invasive behaviour in cutaneous squamous cell carcinoma with loss of basement-membrane type VII collagen. Journal of Cell Science. 122(11). 1788–1799. 89 indexed citations

19.

McGrath, John A. & Robin A.J. Eady. (1997). Heparan sulphate proteoglycan and wound healing in skin. The Journal of Pathology. 183(3). 251–252. 1 indexed citations

20.

McGrath, John A. & Robin A.J. Eady. (1997). Heparan sulphate proteoglycan and wound healing in skin. The Journal of Pathology. 183(3). 251–252. 15 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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