Michael J. Eck
- Oncology top 0.05%
- HER2/EGFR in Cancer Research 21
- Cytokine Signaling Pathways and Interactions 13
- Pulmonary and Respiratory Medicine top 0.05%
- Lung Cancer Treatments and Mutations 31
- Cancer Research top 0.1%
- Molecular Biology top 0.1%
- Protein Kinase Regulation and GTPase Signaling 26
- Cancer therapeutics and mechanisms 22
- Immunology and Allergy top 0.2%
- Cell Adhesion Molecules Research 11
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- Cellular Mechanics and Interactions 21
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- Monoclonal and Polyclonal Antibodies Research 16
- Co-authors
- Titus J. BoggonMatthew MeyersonPasi A. JänneHeidi GreulichBruce E. JohnsonCai‐Hong YunWenqing XuStephen C. Harrison
- Journals
- Journal of Biological Chemistry (10 papers)Proceedings of the National Academy of Sciences (10 papers)Nature (9 papers)
- Partner nations
- United StatesGermanySouth Korea
In The Last Decade
Michael J. Eck
152 papers receiving 31.8k citations
Hit Papers
Peers
Comparison fields: 5 of 160
- Oncology 12.5k
- Pulmonary and Respiratory Medicine 11.2k
- Cancer Research 4.5k
- Molecular Biology 18.3k
- Immunology and Allergy 1.6k
Countries citing papers authored by Michael J. Eck
This map shows the geographic impact of Michael J. Eck's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Michael J. Eck with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Michael J. Eck more than expected).
Fields of papers citing papers by Michael J. Eck
This network shows the impact of papers produced by Michael J. Eck. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Michael J. Eck. The network helps show where Michael J. Eck may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Michael J. Eck, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2025 | 0 | |
| 2 | 2025 | 0 | |
| 3 | 2025 | 0 | |
| 4 | 2024 | 1 | |
| 5 | 2024 | 5 | |
| 6 | 2024 | 2 | |
| 7 | 2023 | 4 | |
| 8 | 2022 | 17 | |
| 9 | 2022 | 56 | |
| 10 | 2019 | 250 | |
| 11 | 2015 | 291 | |
| 12 | 2014 | 136 | |
| 13 | 2013 | 86 | |
| 14 | 2012 | 125 | |
| 15 | 2011 | 109 | |
| 16 | 2010 | 254 | |
| 17 | The T790M mutation in EGFR kinase causes drug resistance by increasing the affinity for ATPbreakdown → | 2008 | 1564 |
| 18 | Gefitinib resistance caused by a secondary mutation of the epidermal growth factor receptor | 2005 | 0 |
| 19 | EGFR Mutations in Lung Cancer: Correlation with Clinical Response to Gefitinib Therapybreakdown → | 2004 | 7539 |
| 20 | 2003 | 209 |
About Michael J. Eck
Michael J. Eck is a scholar working on Immunology and Allergy, Cell Biology and Oncology, having authored 158 papers that have together received 32.5k indexed citations. Recurring topics across this work include Lung Cancer Treatments and Mutations (31 papers), Protein Kinase Regulation and GTPase Signaling (26 papers), Cancer therapeutics and mechanisms (22 papers), Cellular Mechanics and Interactions (21 papers), HER2/EGFR in Cancer Research (21 papers), Monoclonal and Polyclonal Antibodies Research (16 papers), Cytokine Signaling Pathways and Interactions (13 papers) and Cell Adhesion Molecules Research (11 papers). The work is most often cited by research in Oncology (12.5k citations), Pulmonary and Respiratory Medicine (11.2k citations) and Cancer Research (4.5k citations). Michael J. Eck has collaborated with scholars based in United States, Germany and South Korea. Frequent co-authors include Titus J. Boggon, Matthew Meyerson, Pasi A. Jänne, Heidi Greulich, Bruce E. Johnson, Cai‐Hong Yun, Wenqing Xu, Stephen C. Harrison, Steven E. Shoelson and Stephen R. Sprang. Their work appears in journals such as Journal of Biological Chemistry, Proceedings of the National Academy of Sciences, Nature, Nature Structural & Molecular Biology and Molecular Cell.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.