David M. Livingston

55.4k total citations · 25 hit papers
251 papers, 41.4k citations indexed

About

David M. Livingston is a scholar working on Molecular Biology, Oncology and Genetics. According to data from OpenAlex, David M. Livingston has authored 251 papers receiving a total of 41.4k indexed citations (citations by other indexed papers that have themselves been cited), including 158 papers in Molecular Biology, 107 papers in Oncology and 104 papers in Genetics. Recurrent topics in David M. Livingston's work include Cancer-related Molecular Pathways (62 papers), Virus-based gene therapy research (60 papers) and DNA Repair Mechanisms (54 papers). David M. Livingston is often cited by papers focused on Cancer-related Molecular Pathways (62 papers), Virus-based gene therapy research (60 papers) and DNA Repair Mechanisms (54 papers). David M. Livingston collaborates with scholars based in United States, France and United Kingdom. David M. Livingston's co-authors include Ralph Scully, James A. DeCaprio, Richard Eckner, William G. Kaelin, Andrew L. Kung, Zoltàn Arany, Mark E. Ewen, John W. Ludlow, Shridar Ganesan and Eric Huang and has published in prestigious journals such as Nature, Science and New England Journal of Medicine.

In The Last Decade

David M. Livingston

250 papers receiving 40.1k citations

Hit Papers

Association of BRCA1 with Rad51 in Mitotic and Meiotic Cells 1989 2026 2001 2013 1997 1996 2005 1994 1998 250 500 750 1000
What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

  1. Association of BRCA1 with Rad51 in Mitotic and Meiotic Cells
    1997 1.2k citations Ralph Scully, Jean Feunteun et al. Cell profile →
  2. Activation of Hypoxia-inducible Transcription Factor Depends Primarily upon Redox-sensitive Stabilization of Its α Subunit
    1996 1.0k citations David M. Livingston et al. profile →
  3. Dicer-deficient mouse embryonic stem cells are defective in differentiation and centromeric silencing
    2005 995 citations Ronny Drapkin, David M. Livingston et al. Genes & Development profile →
  4. Molecular cloning and functional analysis of the adenovirus E1A-associated 300-kD protein (p300) reveals a protein with properties of a transcriptional adaptor.
    1994 930 citations Richard Eckner, James A. DeCaprio et al. Genes & Development profile →
  5. Gene Dosage–Dependent Embryonic Development and Proliferation Defects in Mice Lacking the Transcriptional Integrator p300
    1998 815 citations Roderick T. Bronson, David M. Livingston et al. Cell profile →
  6. The product of the retinoblastoma susceptibility gene has properties of a cell cycle regulatory element
    1989 813 citations James A. DeCaprio, John W. Ludlow et al. Cell profile →
  7. Dynamic Changes of BRCA1 Subnuclear Location and Phosphorylation State Are Initiated by DNA Damage
    1997 748 citations Ralph Scully, Jean Feunteun et al. Cell profile →
  8. Growth inhibition by TGF-β linked to suppression of retinoblastoma protein phosphorylation
    1990 710 citations Marikki Laiho, James A. DeCaprio et al. Cell profile →
  9. Role of T-bet in Commitment of T H 1 Cells Before IL-12-Dependent Selection
    2001 672 citations David M. Livingston, Tso‐Pang Yao et al. Science profile →
  10. X chromosomal abnormalities in basal-like human breast cancer
    2006 668 citations Arcangela De Nicolo, Myles Brown et al. profile →
  11. E2F-1 Functions in Mice to Promote Apoptosis and Suppress Proliferation
    1996 661 citations David M. Livingston et al. Cell profile →
  12. Deregulated transcription factor E2F-1 expression leads to S-phase entry and p53-mediated apoptosis.
    1994 658 citations David M. Livingston et al. Proceedings of the National Academy of Sciences profile →
  13. An essential role for p300/CBP in the cellular response to hypoxia
    1996 646 citations Richard Eckner, David M. Livingston et al. Proceedings of the National Academy of Sciences profile →
  14. Control of BRCA2 Cellular and Clinical Functions by a Nuclear Partner, PALB2
    2006 635 citations Bing Xia, David M. Livingston et al. Molecular Cell profile →
  15. A Complex with Chromatin Modifiers That Occupies E2F- and Myc-Responsive Genes in G 0 Cells
    2002 610 citations Hidesato Ogawa, Kei‐ichiro Ishiguro et al. Science profile →
  16. Binding and modulation of p53 by p300/CBP coactivators
    1997 592 citations James A. DeCaprio, David M. Livingston et al. Nature profile →
  17. RAP80 Targets BRCA1 to Specific Ubiquitin Structures at DNA Damage Sites
    2007 553 citations Bijan Sobhian, Genze Shao et al. Science profile →
  18. BACH1, a Novel Helicase-like Protein, Interacts Directly with BRCA1 and Contributes to Its DNA Repair Function
    2001 541 citations Sharon B. Cantor, Ronny Drapkin et al. Cell profile →
  19. Aberrant T cell differentiation in the absence of Dicer
    2005 518 citations David M. Livingston et al. profile →
  20. In search of the tumour-suppressor functions of BRCA1 and BRCA2
    2000 515 citations Ralph Scully, David M. Livingston Nature profile →
  21. Interaction between the retinoblastoma protein and the oncoprotein MDM2
    1995 512 citations David M. Livingston et al. Nature profile →
  22. Identification of cellular proteins that can interact specifically with the T/ElA-binding region of the retinoblastoma gene product
    1991 496 citations James A. DeCaprio, David M. Livingston et al. Cell profile →
  23. Suppression of tumor growth through disruption of hypoxia-inducible transcription
    2000 436 citations David M. Livingston et al. profile →
  24. Identification of a growth suppression domain within the retinoblastoma gene product.
    1992 379 citations Thomas W. Chittenden, David M. Livingston et al. Genes & Development profile →
  25. BRCA1 Recruitment to Transcriptional Pause Sites Is Required for R-Loop-Driven DNA Damage Repair
    2015 351 citations Shailja Pathania, Sarah J. Hill et al. Molecular Cell profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
David M. Livingston United States 102 30.8k 16.1k 10.6k 7.6k 3.7k 251 41.4k
Charles J. Sherr United States 96 39.2k 1.3× 30.4k 1.9× 6.0k 0.6× 6.9k 0.9× 6.6k 1.8× 271 58.0k
William G. Kaelin United States 117 35.6k 1.2× 14.8k 0.9× 6.0k 0.6× 22.6k 3.0× 3.2k 0.9× 246 50.2k
Kristian Helin Denmark 103 31.3k 1.0× 9.1k 0.6× 4.6k 0.4× 4.8k 0.6× 2.3k 0.6× 275 37.0k
David Beach United States 100 34.4k 1.1× 20.2k 1.3× 3.5k 0.3× 5.3k 0.7× 3.7k 1.0× 247 47.3k
J. Michael Bishop United States 116 33.0k 1.1× 9.5k 0.6× 11.4k 1.1× 5.1k 0.7× 5.7k 1.5× 356 48.6k
Stuart A. Aaronson United States 107 27.9k 0.9× 12.2k 0.8× 9.3k 0.9× 4.4k 0.6× 6.4k 1.7× 438 45.8k
Moshe Oren Israel 116 34.9k 1.1× 28.8k 1.8× 4.3k 0.4× 9.9k 1.3× 3.3k 0.9× 323 48.9k
Jiří Bártek Denmark 107 41.7k 1.4× 23.3k 1.4× 4.7k 0.4× 9.5k 1.2× 2.7k 0.7× 383 54.0k
Frank McCormick United States 118 42.8k 1.4× 14.4k 0.9× 7.5k 0.7× 5.6k 0.7× 4.3k 1.2× 381 57.5k
Axel Ullrich Germany 97 32.2k 1.0× 23.0k 1.4× 4.4k 0.4× 6.8k 0.9× 8.3k 2.3× 257 55.6k

Countries citing papers authored by David M. Livingston

Since Specialization
Citations

This map shows the geographic impact of David M. Livingston's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by David M. Livingston with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites David M. Livingston more than expected).

Fields of papers citing papers by David M. Livingston

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by David M. Livingston. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by David M. Livingston. The network helps show where David M. Livingston may publish in the future.

Co-authorship network of co-authors of David M. Livingston

This figure shows the co-authorship network connecting the top 25 collaborators of David M. Livingston. A scholar is included among the top collaborators of David M. Livingston based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with David M. Livingston. David M. Livingston is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Xiang, Dongxi, Ben Liu, Aina He, et al.. (2019). Inadequate DNA Damage Repair Promotes Mammary Transdifferentiation, Leading to BRCA1 Breast Cancer. Cell. 178(1). 135–151.e19. 67 indexed citations
2.
Hu, Yiduo, Sarah A. Petit, Scott B. Ficarro, et al.. (2014). PARP1-Driven Poly-ADP-Ribosylation Regulates BRCA1 Function in Homologous Recombination–Mediated DNA Repair. Cancer Discovery. 4(12). 1430–1447. 136 indexed citations
3.
Naetar, Nana, Vel Murugan, Larisa Litovchick, et al.. (2014). PP2A-Mediated Regulation of Ras Signaling in G2 Is Essential for Stable Quiescence and Normal G1 Length. Molecular Cell. 54(6). 932–945. 53 indexed citations
4.
Pathania, Shailja, Jenna Nguyen, Sarah J. Hill, et al.. (2011). BRCA1 Is Required for Postreplication Repair after UV-Induced DNA Damage. Molecular Cell. 44(2). 235–251. 88 indexed citations
5.
Jirawatnotai, Siwanon, Yiduo Hu, Wojciech Michowski, et al.. (2011). A function for cyclin D1 in DNA repair uncovered by protein interactome analyses in human cancers. Nature. 474(7350). 230–234. 255 indexed citations
6.
Hu, Yiduo, Ralph Scully, Bijan Sobhian, et al.. (2011). RAP80-directed tuning of BRCA1 homologous recombination function at ionizing radiation-induced nuclear foci. Genes & Development. 25(7). 685–700. 189 indexed citations
7.
Joukov, Vladimir, et al.. (2010). Centrosomal protein of 192 kDa (Cep192) promotes centrosome-driven spindle assembly by engaging in organelle-specific Aurora A activation. Proceedings of the National Academy of Sciences. 107(49). 21022–21027. 87 indexed citations
8.
O'Donovan, P.J. & David M. Livingston. (2010). BRCA1 and BRCA2: breast/ovarian cancer susceptibility gene products and participants in DNA double-strand break repair. Carcinogenesis. 31(6). 961–967. 211 indexed citations
9.
Sobhian, Bijan, Genze Shao, Dana R. Lilli, et al.. (2007). RAP80 Targets BRCA1 to Specific Ubiquitin Structures at DNA Damage Sites. Science. 316(5828). 1198–1202. 553 indexed citations breakdown →
10.
Cantor, Sharon B., Ronny Drapkin, Fan Zhang, et al.. (2004). The BRCA1-associated protein BACH1 is a DNA helicase targeted by clinically relevant inactivating mutations. Proceedings of the National Academy of Sciences. 101(8). 2357–2362. 193 indexed citations
11.
Ogawa, Hidesato, Kei‐ichiro Ishiguro, Stefan Gaubatz, David M. Livingston, & Yoshihiro Nakatani. (2002). A Complex with Chromatin Modifiers That Occupies E2F- and Myc-Responsive Genes in G 0 Cells. Science. 296(5570). 1132–1136. 610 indexed citations breakdown →
12.
Yang, Hong, Bart O. Williams, T. Shane Shih, et al.. (2002). Tumor Suppression by a Severely Truncated Species of Retinoblastoma Protein. Molecular and Cellular Biology. 22(9). 3103–3110. 20 indexed citations
13.
Ranganathan, Velvizhi, Walter Heine, David Ciccone, et al.. (2001). Rescue of a telomere length defect of Nijmegen breakage syndrome cells requires NBS and telomerase catalytic subunit. Current Biology. 11(12). 962–966. 94 indexed citations
14.
Gaubatz, Stefan, Geoffrey J. Lindeman, Seiichi Ishida, et al.. (2000). E2F4 and E2F5 Play an Essential Role in Pocket Protein–Mediated G1 Control. Molecular Cell. 6(3). 729–735. 229 indexed citations
15.
Kawasaki, Hiroaki, Richard Eckner, Tso‐Pang Yao, et al.. (1998). Distinct roles of the co-activators p300 and CBP in retinoic-acid-induced F9-cell differentiation. Nature. 393(6682). 284–289. 273 indexed citations
16.
Chittenden, Thomas W., David M. Livingston, & James A. DeCaprio. (1993). Cell Cycle Analysis of E2F in Primary Human T Cells Reveals Novel E2F Complexes and Biochemically Distinct Forms of Free E2F. Molecular and Cellular Biology. 13(7). 3975–3983. 88 indexed citations
17.
Laiho, Marikki, James A. DeCaprio, John W. Ludlow, David M. Livingston, & Joan Massagué. (1990). Growth inhibition by TGF-β linked to suppression of retinoblastoma protein phosphorylation. Cell. 62(1). 175–185. 710 indexed citations breakdown →
18.
Bradley, Margaret K., James D. Griffin, & David M. Livingston. (1982). Relationship of oligomerization to enzymatic and DNA-binding properties of the SV40 large T antigen. Cell. 28(1). 125–134. 108 indexed citations
19.
Livingston, David M.. (1976). Book ReviewCancer: A comprehensive treatise. Vol. 2. Etiology: Viral carcinogenesis:. New England Journal of Medicine. 295(3). 176–177. 1 indexed citations
20.
DeVita, Vincent T., Bruce A. Chabner, David M. Livingston, & Vincent T. Oliverio. (1971). Anergy and tryptophan metabolism in Hodgkin’s disease. American Journal of Clinical Nutrition. 24(7). 835–840. 13 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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