David M. Livingston

55.4k citations

251 papers · 41.4k indexed · 25 hit papers · h-index 102

Oncology top 0.02%
- Cancer-related Molecular Pathways 62
- Polyomavirus and related diseases 29
Cancer Research top 0.05%
Molecular Biology top 0.02%
- DNA Repair Mechanisms 54
- CRISPR and Genetic Engineering 35
- Ubiquitin and proteasome pathways 24
Genetics top 0.02%
- Virus-based gene therapy research 60
- BRCA gene mutations in cancer 32
Cell Biology top 0.2%
Ecology
- Bacteriophages and microbial interactions 22

Co-authors: Ralph Scully James A. DeCaprio Richard Eckner William G. Kaelin Andrew L. Kung Zoltàn Arany Mark E. Ewen John W. Ludlow
Cited by: Oncology Cancer Research Molecular Biology
Partner nations: United States France United Kingdom

In The Last Decade

David M. Livingston

250 papers receiving 40.1k citations

Hit Papers

15 papers align trajectories log scale

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

2015 BRCA1 Recruitment to Transcriptional Pause Sites Is Required for R-Loop-Driven DNA Damage Repair
2007 RAP80 Targets BRCA1 to Specific Ubiquitin Structures at DNA Damage Sites
2006 X chromosomal abnormalities in basal-like human breast cancer
2006 Control of BRCA2 Cellular and Clinical Functions by a Nuclear Partner, PALB2
2005 Dicer-deficient mouse embryonic stem cells are defective in differentiation and centromeric silencing
2005 Aberrant T cell differentiation in the absence of Dicer
2002 A Complex with Chromatin Modifiers That Occupies E2F- and Myc-Responsive Genes in G ₀ Cells
2001 Role of T-bet in Commitment of T _H 1 Cells Before IL-12-Dependent Selection
2001 BACH1, a Novel Helicase-like Protein, Interacts Directly with BRCA1 and Contributes to Its DNA Repair Function
2000 Suppression of tumor growth through disruption of hypoxia-inducible transcription
2000 In search of the tumour-suppressor functions of BRCA1 and BRCA2
1998 Gene Dosage–Dependent Embryonic Development and Proliferation Defects in Mice Lacking the Transcriptional Integrator p300
1997 Association of BRCA1 with Rad51 in Mitotic and Meiotic Cells
1997 Dynamic Changes of BRCA1 Subnuclear Location and Phosphorylation State Are Initiated by DNA Damage
1997 Binding and modulation of p53 by p300/CBP coactivators
1996 E2F-1 Functions in Mice to Promote Apoptosis and Suppress Proliferation
1996 Activation of Hypoxia-inducible Transcription Factor Depends Primarily upon Redox-sensitive Stabilization of Its α Subunit
1996 An essential role for p300/CBP in the cellular response to hypoxia
1995 Interaction between the retinoblastoma protein and the oncoprotein MDM2
1994 Molecular cloning and functional analysis of the adenovirus E1A-associated 300-kD protein (p300) reveals a protein with properties of a transcriptional adaptor.
1994 Deregulated transcription factor E2F-1 expression leads to S-phase entry and p53-mediated apoptosis.
1992 Identification of a growth suppression domain within the retinoblastoma gene product.
1991 Identification of cellular proteins that can interact specifically with the T/ElA-binding region of the retinoblastoma gene product
1990 Growth inhibition by TGF-β linked to suppression of retinoblastoma protein phosphorylation
1989 The product of the retinoblastoma susceptibility gene has properties of a cell cycle regulatory element

Peers

Countries citing papers authored by David M. Livingston

Since Specialization

Citations

This map shows the geographic impact of David M. Livingston's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by David M. Livingston with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites David M. Livingston more than expected).

Fields of papers citing papers by David M. Livingston

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by David M. Livingston. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by David M. Livingston. The network helps show where David M. Livingston may publish in the future.

Co-authorship network

The 25 scholars most cited alongside David M. Livingston, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with David M. Livingston Line = papers co-authored together David M. Livingston links everyone, so they are left out of the graph.

All Works

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20 of 20 papers shown

#	Work
1	Inadequate DNA Damage Repair Promotes Mammary Transdifferentiation, Leading to BRCA1 Breast Cancer Hua Wang,Dongxi Xiang,Ben Liu,Aina He,Helena J. Randle,Kelvin Xi Zhang,Anushka Dongre,Norman Sachs,Allison Clark,Luwei Tao,Qing Chen,Vladimir V. Botchkarev,Ying Xie,Ning Dai,Hans Clevers,Zhe Li,David M. Livingston	2019	67
2	PARP1-Driven Poly-ADP-Ribosylation Regulates BRCA1 Function in Homologous Recombination–Mediated DNA Repair Yiduo Hu,Sarah A. Petit,Scott B. Ficarro,Kimberly J. Toomire,Anyong Xie,Elgene Lim,Shiliang A. Cao,Eunyoung Park,Michael J. Eck,Ralph Scully,Myles Brown,Jarrod A. Marto,David M. Livingston	2014	136
3	PP2A-Mediated Regulation of Ras Signaling in G2 Is Essential for Stable Quiescence and Normal G1 Length Nana Naetar,Vel Murugan,Larisa Litovchick,Kelsey L Goguen,Anna Sablina,Christian Bowman-Colin,Piotr Siciński,William C. Hahn,James A. DeCaprio,David M. Livingston	2014	53
4	BRCA1 Is Required for Postreplication Repair after UV-Induced DNA Damage Shailja Pathania,Jenna Nguyen,Sarah J. Hill,Ralph Scully,Guillaume Adelmant,Jarrod A. Marto,Jean Feunteun,David M. Livingston	2011	88
5	RAP80-directed tuning of BRCA1 homologous recombination function at ionizing radiation-induced nuclear foci Yiduo Hu,Ralph Scully,Bijan Sobhian,Anyong Xie,Elena Shestakova,David M. Livingston	2011	189
6	Centrosomal protein of 192 kDa (Cep192) promotes centrosome-driven spindle assembly by engaging in organelle-specific Aurora A activation Vladimir Joukov,Arcangela De Nicolo,Alison Rodriguez,Johannes C. Walter,David M. Livingston	2010	87
7	BRCA1 and BRCA2: breast/ovarian cancer susceptibility gene products and participants in DNA double-strand break repair P.J. O'Donovan,David M. Livingston	2010	211
8	RAP80 Targets BRCA1 to Specific Ubiquitin Structures at DNA Damage Sitesbreakdown → Bijan Sobhian,Genze Shao,Dana R. Lilli,Aedín C. Culhane,Lisa A. Moreau,Bing Xia,David M. Livingston,Roger A. Greenberg	2007	553
9	Dicer-deficient mouse embryonic stem cells are defective in differentiation and centromeric silencingbreakdown → Chryssa Kanellopoulou,Stefan A. Muljo,Andrew L. Kung,Shridar Ganesan,Ronny Drapkin,Thomas Jenuwein,David M. Livingston,Klaus Rajewsky	2005	995
10	The BRCA1-associated protein BACH1 is a DNA helicase targeted by clinically relevant inactivating mutations Sharon B. Cantor,Ronny Drapkin,Fan Zhang,Yafang Lin,Juliana Han,Sushmita Pamidi,David M. Livingston	2004	193
11	A Complex with Chromatin Modifiers That Occupies E2F- and Myc-Responsive Genes in G ₀ Cellsbreakdown → Hidesato Ogawa,Kei‐ichiro Ishiguro,Stefan Gaubatz,David M. Livingston,Yoshihiro Nakatani	2002	610
12	Tumor Suppression by a Severely Truncated Species of Retinoblastoma Protein Hong Yang,Bart O. Williams,Phillip W. Hinds,T. Shane Shih,Tyler Jacks,Roderick T. Bronson,David M. Livingston	2002	20
13	Rescue of a telomere length defect of Nijmegen breakage syndrome cells requires NBS and telomerase catalytic subunit Velvizhi Ranganathan,Walter Heine,David Ciccone,K. Lenhard Rudolph,Xiaohua Wu,Sandy Chang,Mingqiu Hu,Ian M. Ahearn,David M. Livingston,Igor Resnick,Fred S. Rosen,E Seemanová,Petr Jarolı́m,Ronald A. DePinho,David T. Weaver	2001	94
14	E2F4 and E2F5 Play an Essential Role in Pocket Protein–Mediated G1 Control Stefan Gaubatz,Geoffrey J. Lindeman,Seiichi Ishida,Laszlo Jakoi,Joseph R. Nevins,David M. Livingston,Rachel E. Rempel	2000	229
15	Suppression of tumor growth through disruption of hypoxia-inducible transcriptionbreakdown → Andrew L. Kung,Stream Wang,Jeffery M. Klco,William G. Kaelin,David M. Livingston	2000	436
16	p300/MDM2 Complexes Participate in MDM2-Mediated p53 Degradation Steven R. Grossman,Marco Perez,Andrew L. Kung,M. P. Joseph,Claire Mansur,Zhi-Xiong Jim Xiao,Sushant Kumar,Peter M. Howley,David M. Livingston	1998	364
17	Cell Cycle Analysis of E2F in Primary Human T Cells Reveals Novel E2F Complexes and Biochemically Distinct Forms of Free E2F Thomas W. Chittenden,David M. Livingston,James A. DeCaprio	1993	88
18	Growth inhibition by TGF-β linked to suppression of retinoblastoma protein phosphorylationbreakdown → Marikki Laiho,James A. DeCaprio,John W. Ludlow,David M. Livingston,Joan Massagué	1990	710
19	RADIOIMMUNOASSAY OF MAMMALIAN TYPE C VIRAL PROTEINS Wade P. Parks,David M. Livingston,George J. Todaro,Raoul Ė. Benveniste,Edward M. Scolnick	1973	78
20	Anergy and tryptophan metabolism in Hodgkin’s disease Vincent T. DeVita,Bruce A. Chabner,David M. Livingston,Vincent T. Oliverio	1971	13

About David M. Livingston

David M. Livingston is a scholar working on Oncology, Genetics and Molecular Biology, having authored 251 papers that have together received 41.4k indexed citations. Recurring topics across this work include Cancer-related Molecular Pathways (62 papers), Virus-based gene therapy research (60 papers), DNA Repair Mechanisms (54 papers), CRISPR and Genetic Engineering (35 papers), BRCA gene mutations in cancer (32 papers), Polyomavirus and related diseases (29 papers), Ubiquitin and proteasome pathways (24 papers) and Bacteriophages and microbial interactions (22 papers). The work is most often cited by research in Oncology (16.1k citations), Cancer Research (7.6k citations) and Molecular Biology (30.8k citations). David M. Livingston has collaborated with scholars based in United States, France and United Kingdom. Frequent co-authors include Ralph Scully, James A. DeCaprio, Richard Eckner, William G. Kaelin, Andrew L. Kung, Zoltàn Arany, Mark E. Ewen, John W. Ludlow, Shridar Ganesan and Eric Huang.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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