Mark E. Smulson
About
Mark E. Smulson is a scholar working on Molecular Biology, Oncology and Immunology.
According to data from OpenAlex, Mark E. Smulson has authored 153 papers receiving a total of 10.8k indexed citations (citations by other indexed papers that have themselves been cited), including 125 papers in Molecular Biology, 95 papers in Oncology and 26 papers in Immunology. Recurrent topics in Mark E. Smulson's work include PARP inhibition in cancer therapy (82 papers), DNA Repair Mechanisms (49 papers) and Cell death mechanisms and regulation (29 papers). Mark E. Smulson is often cited by papers focused on PARP inhibition in cancer therapy (82 papers), DNA Repair Mechanisms (49 papers) and Cell death mechanisms and regulation (29 papers). Mark E. Smulson collaborates with scholars based in United States, Japan and France. Mark E. Smulson's co-authors include Dean S. Rosenthal, Cynthia M. Simbulan‐Rosenthal, A. Hamid Boulares, Sudha Iyer, Donald W. Nicholson, John P. Vaillancourt, Marc Labelle, Michel Gallant, Violeta Yu and Ting-Ting Yamin and has published in prestigious journals such as Nature, Science and Proceedings of the National Academy of Sciences.
In The Last Decade
Mark E. Smulson
150 papers receiving 10.3k citations
Hit Papers
Peers — A (Enhanced Table)
Peers by citation overlap · career bar shows stage (early→late) cites · hero ref
| Name | h | Career | Trend | Papers | Cites | |||||
|---|---|---|---|---|---|---|---|---|---|---|
| Mark E. Smulson United States | 50 | 7.6k | 4.0k | 1.7k | 938 | 694 | 153 | 10.8k | ||
| Serge Desnoyers Canada | 22 | 7.0k 0.9× | 3.7k 0.9× | 1.7k 1.0× | 786 0.8× | 659 0.9× | 35 | 9.3k | ||
| Richard A. Rifkind United States | 54 | 11.4k 1.5× | 2.9k 0.7× | 1.0k 0.6× | 786 0.8× | 519 0.7× | 123 | 14.3k | ||
| John A. Hickman United Kingdom | 50 | 6.9k 0.9× | 3.3k 0.8× | 930 0.5× | 1.4k 1.5× | 1.3k 1.8× | 184 | 10.8k | ||
| Robert Roskoski United States | 57 | 9.9k 1.3× | 4.1k 1.0× | 1.3k 0.8× | 1.4k 1.5× | 514 0.7× | 173 | 16.3k | ||
| Gilbert de Murcia France | 54 | 9.3k 1.2× | 9.0k 2.3× | 2.2k 1.2× | 699 0.7× | 572 0.8× | 103 | 13.1k | ||
| I. Imawati Budihardjo United States | 13 | 9.4k 1.2× | 1.8k 0.4× | 2.0k 1.1× | 1.4k 1.5× | 1.6k 2.3× | 14 | 12.1k | ||
| Reiner U. Jänicke Germany | 37 | 6.5k 0.8× | 2.0k 0.5× | 1.5k 0.9× | 1.1k 1.2× | 874 1.3× | 68 | 9.9k | ||
| Isabel Marzo Spain | 48 | 8.5k 1.1× | 2.1k 0.5× | 1.8k 1.0× | 975 1.0× | 1.2k 1.8× | 109 | 11.7k | ||
| Ingrid Herr Germany | 50 | 6.6k 0.9× | 3.0k 0.7× | 1.8k 1.0× | 2.1k 2.3× | 1.1k 1.6× | 139 | 10.2k | ||
| Christoph Borner Germany | 50 | 6.1k 0.8× | 1.3k 0.3× | 1.6k 0.9× | 715 0.8× | 1.2k 1.7× | 119 | 8.8k |
Countries citing papers authored by Mark E. Smulson
Since
Specialization
Citations
This map shows the geographic impact of Mark E. Smulson's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mark E. Smulson with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mark E. Smulson more than expected).
Fields of papers citing papers by Mark E. Smulson
Since
Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences
This network shows the impact of papers produced by Mark E. Smulson. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mark E. Smulson. The network helps show where Mark E. Smulson may publish in the future.
Co-authorship network of co-authors of Mark E. Smulson
This figure shows the co-authorship network connecting the top 25 collaborators of Mark E. Smulson. A scholar is included among the top collaborators of Mark E. Smulson based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Mark E. Smulson. Mark E. Smulson is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
1.
Errami, Youssef, Amarjit S. Naura, Hogyoung Kim, et al.. (2012). Apoptotic DNA Fragmentation May Be a Cooperative Activity between Caspase-activated Deoxyribonuclease and the Poly(ADP-ribose) Polymerase-regulated DNAS1L3, an Endoplasmic Reticulum-localized Endonuclease That Translocates to the Nucleus during Apoptosis. Journal of Biological Chemistry. 288(5). 3460–3468.
2.
Simbulan‐Rosenthal, Cynthia M., Dean S. Rosenthal, & Mark E. Smulson. (2011). Purification and Characterization of Poly(ADP-Ribosyl)ated DNA Replication/Repair Complexes. Methods in molecular biology. 780. 165–190.
3.
Wang, Tao, Cynthia M. Simbulan‐Rosenthal, Mark E. Smulson, P Boon Chock, & David C.H. Yang. (2007). Polyubiquitylation of PARP‐1 through ubiquitin K48 is modulated by activated DNA, NAD+, and dipeptides. Journal of Cellular Biochemistry. 104(1). 318–328.
4.
Chiba‐Falek, Ornit, Jeffrey A. Kowalak, Mark E. Smulson, & Robert L. Nussbaum. (2005). Regulation of α-Synuclein Expression by Poly (ADP Ribose) Polymerase-1 (PARP-1) Binding to the NACP-Rep1 Polymorphic Site Upstream of the SNCA Gene. The American Journal of Human Genetics. 76(3). 478–492.
5.
Boulares, A. Hamid, Anna J. Zoltoski, Bogdan A. Stoica, Olivier Cuvillier, & Mark E. Smulson. (2002). Acetaminophen Induces a Caspase‐Dependent and Bcl‐xLSensitive Apoptosis in Human Hepatoma Cells and Lymphocytes. Pharmacology & Toxicology. 90(1). 38–50.
6.
Boulares, A. Hamid, Anna J. Zoltoski, Francisco Contreras, et al.. (2002). Regulation of DNAS1L3 Endonuclease Activity by Poly(ADP-ribosyl)ation during Etoposide-induced Apoptosis. Journal of Biological Chemistry. 277(1). 372–378.
7.
Boulares, A. Hamid, Anna J. Zoltoski, Alexander G. Yakovlev, Ming Xu, & Mark E. Smulson. (2001). Roles of DNA Fragmentation Factor and Poly(ADP-ribose) Polymerase in an Amplification Phase of Tumor Necrosis Factor-induced Apoptosis. Journal of Biological Chemistry. 276(41). 38185–38192.
8.
Bhat, Kumar M. R., Betty Benton, Dean S. Rosenthal, Mark E. Smulson, & Rubik Ray. (2001). Role of poly(ADP-ribose) polymerase (PARP) in DNA repair in sulfur mustard-exposed normal human epidermal keratinocytes (NHEK)†‡. Journal of Applied Toxicology. 20(S1). S13–S17.
9.
Rosenthal, Dean S., et al.. (2001). Mechanisms of JP-8 Jet Fuel Cell Toxicity. II. Induction of Necrosis in Skin Fibroblasts and Keratinocytes and Modulation of Levels of Bcl-2 Family Members. Toxicology and Applied Pharmacology. 171(2). 107–116.
10.
Yakovlev, Alexander G., Geping Wang, Bogdan A. Stoica, et al.. (2000). A Role of the Ca2+/Mg2+-dependent Endonuclease in Apoptosis and Its Inhibition by Poly(ADP-ribose) Polymerase. Journal of Biological Chemistry. 275(28). 21302–21308.
11.
Rosenthal, Dean S., Cynthia M. Simbulan‐Rosenthal, Sudha Iyer, et al.. (1998). Sulfur Mustard Induces Markers of Terminal Differentiation and Apoptosis in Keratinocytes Via a Ca2+-Calmodulin and Caspase-Dependent Pathway. Journal of Investigative Dermatology. 111(1). 64–71.
12.
Rosenthal, Dean S., Ruchuang Ding, Cynthia M. Simbulan‐Rosenthal, et al.. (1997). Intact Cell Evidence for the Early Synthesis, and Subsequent Late Apopain-Mediated Suppression, of Poly(ADP-ribose) during Apoptosis. Experimental Cell Research. 232(2). 313–321.
13.
Smulson, Mark E., Veronica H. Kang, James M. Ntambi, et al.. (1995). Requirement for the Expression of Poly(ADP-ribose) Polymerase during the Early Stages of Differentiation of 3T3-L1 Preadipocytes, as Studied by Antisense RNA Induction. Journal of Biological Chemistry. 270(1). 119–127.
14.
Rosenthal, Dean S., Thomas B. Shima, Giulia Celli, Luigi M. De Luca, & Mark E. Smulson. (1995). Engineered Human Skin Model Using Poly(ADP-Ribose) Polymerase Antisense Expression Shows a Reduced Response to DNA Damage. Journal of Investigative Dermatology. 105(1). 38–43.
15.
Ding, Ruchuang & Mark E. Smulson. (1994). Depletion of nuclear poly(ADP-ribose) polymerase by antisense RNA expression: influences on genomic stability, chromatin organization, and carcinogen cytotoxicity.. PubMed. 54(17). 4627–34.
16.
Ávila, Matías A., Juan A. Velasco, Mark E. Smulson, et al.. (1994). Functional expression of human poly(ADP‐ribose) polymerase in Schizosaccharomyces pombe results in mitotic delay at G1, increased mutation rate, and sensitization to radiation. Yeast. 10(8). 1003–1017.
17.
Smulson, Mark E.. (1994). Poly(ADP-ribose) polymerase gene on chromosome 1q: Early role in differentiation linked replication; gene on human chromosome 13q: Marker of carcinogenesis. Molecular and Cellular Biochemistry. 138(1-2). 77–84.
18.
Pero, R W, G Berglund, Nelwyn T. Christie, et al.. (1993). The Malmö biomarker programme. Journal of Internal Medicine. 233(1). 69–74.
19.
Bhatia, Kishor, Albert J. Fornace, Masue Imaizumi, et al.. (1990). Expression of the poly(ADP-ribose) polymerase gene following natural and induced DNA strand breakage and effect of hyperexpression on DNA repair. Carcinogenesis. 11(1). 123–128.
20.
Hüppi, Konrad, et al.. (1989). Sequence and organization of the mouse poly (ADP-ribose) polymerase gene. Nucleic Acids Research. 17(9). 3387–3401.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.
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