Thomas Ried
- Cancer Research top 0.05%
- Cancer Genomics and Diagnostics 80
- Geriatrics and Gerontology top 0.2%
- Oncology top 0.1%
- Cancer-related Molecular Pathways 36
- Molecular Biology top 0.1%
- Genomics and Chromatin Dynamics 44
- DNA Repair Mechanisms 41
- Genetics top 0.1%
- Genomic variations and chromosomal abnormalities 112
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- Genetic factors in colorectal cancer 64
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- Chromosomal and Genetic Variations 46
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- Microtubule and mitosis dynamics 22
- Co-authors
- Evelin SchröckMichael J. DifilippantonioHesed Padilla‐NashStanislas du ManoirΒ. Michael GhadimiGert AuerAnthony Wynshaw‐BorisAndré Nussenzweig
- Partner nations
- United StatesGermanySweden
In The Last Decade
Thomas Ried
372 papers receiving 27.3k citations
Hit Papers
Peers
Comparison fields: 5 of 169
- Cancer Research 6.2k
- Geriatrics and Gerontology 1.0k
- Oncology 7.3k
- Molecular Biology 17.7k
- Genetics 6.2k
Countries citing papers authored by Thomas Ried
This map shows the geographic impact of Thomas Ried's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Thomas Ried with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Thomas Ried more than expected).
Fields of papers citing papers by Thomas Ried
This network shows the impact of papers produced by Thomas Ried. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Thomas Ried. The network helps show where Thomas Ried may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Thomas Ried, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2021 | 9 | |
| 2 | 2020 | 3 | |
| 3 | 2020 | 9 | |
| 4 | 2018 | 19 | |
| 5 | 2018 | 118 | |
| 6 | 2017 | 104 | |
| 7 | 2017 | 27 | |
| 8 | 2017 | 22 | |
| 9 | 2013 | 37 | |
| 10 | 2012 | 158 | |
| 11 | 2012 | 76 | |
| 12 | 2010 | 13 | |
| 13 | 2009 | 86 | |
| 14 | 2008 | 68 | |
| 15 | 2007 | 96 | |
| 16 | 2007 | 18 | |
| 17 | 2005 | 41 | |
| 18 | 2005 | 20 | |
| 19 | 2004 | 137 | |
| 20 | 2004 | 27 |
About Thomas Ried
Thomas Ried is a scholar working on Cancer Research, Genetics and Oncology, having authored 378 papers that have together received 27.8k indexed citations. Recurring topics across this work include Genomic variations and chromosomal abnormalities (112 papers), Cancer Genomics and Diagnostics (80 papers), Genetic factors in colorectal cancer (64 papers), Chromosomal and Genetic Variations (46 papers), Genomics and Chromatin Dynamics (44 papers), DNA Repair Mechanisms (41 papers), Cancer-related Molecular Pathways (36 papers) and Microtubule and mitosis dynamics (22 papers). The work is most often cited by research in Cancer Research (6.2k citations), Geriatrics and Gerontology (1.0k citations) and Oncology (7.3k citations). Thomas Ried has collaborated with scholars based in United States, Germany and Sweden. Frequent co-authors include Evelin Schröck, Michael J. Difilippantonio, Hesed Padilla‐Nash, Stanislas du Manoir, Β. Michael Ghadimi, Gert Auer, Anthony Wynshaw‐Boris, André Nussenzweig, Jordi Camps and Marek Liyanage. Their work appears in journals such as Genes Chromosomes and Cancer, Cancer Research, Oncogene, International Journal of Cancer and Blood.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.