John K. Eaton
- Molecular Biology top 2%
- Pulmonary and Respiratory Medicine top 0.5%
- Cancer Research top 0.5%
- Oncology top 5%
- Nutrition and Dietetics top 1%
- Co-authors
- Stuart L. SchreiberVasanthi S. ViswanathanEvgeni M. FrenkelMustafa KocakRyan D. SpanglerShannon CoyJordan RossenBoryana Petrova
- Topics
- Ferroptosis and cancer prognosis (3 papers)Mitochondrial Function and Pathology (2 papers)Trace Elements in Health (2 papers)
- Partner nations
- United StatesGermany
In The Last Decade
John K. Eaton
9 papers receiving 5.7k citations
Hit Papers
Peers
Comparison fields: 5 of 125
- Molecular Biology 3.3k
- Pulmonary and Respiratory Medicine 3.0k
- Cancer Research 2.5k
- Oncology 825
- Nutrition and Dietetics 655
Countries citing papers authored by John K. Eaton
This map shows the geographic impact of John K. Eaton's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by John K. Eaton with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites John K. Eaton more than expected).
Fields of papers citing papers by John K. Eaton
This network shows the impact of papers produced by John K. Eaton. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by John K. Eaton. The network helps show where John K. Eaton may publish in the future.
Co-authorship network of co-authors of John K. Eaton
This figure shows the co-authorship network connecting the top 25 collaborators of John K. Eaton. A scholar is included among the top collaborators of John K. Eaton based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with John K. Eaton. John K. Eaton is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | Copper induces cell death by targeting lipoylated TCA cycle proteinsbreakdown → | 2990 |
| 2 | 89 | |
| 3 | 10 | |
| 4 | Cytochrome P450 oxidoreductase contributes tophospholipid peroxidation in ferroptosisbreakdown → | 565 |
| 5 | 45 | |
| 6 | 99 | |
| 7 | A GPX4-dependent cancer cell state underlies the clear-cell morphology and confers sensitivity to ferroptosisbreakdown → | 663 |
| 8 | 97 | |
| 9 | Drug-tolerant persister cancer cells are vulnerable to GPX4 inhibitionbreakdown → | 1204 |
About John K. Eaton
John K. Eaton is a scholar working on Clinical Biochemistry, Cancer Research and Nutrition and Dietetics, having authored 9 papers that have together received 5.8k indexed citations. Recurring topics across this work include Ferroptosis and cancer prognosis (3 papers), Mitochondrial Function and Pathology (2 papers) and Trace Elements in Health (2 papers). The work is most often cited by research in Cancer Research (2.5k citations), Pulmonary and Respiratory Medicine (3.0k citations) and Molecular Biology (3.3k citations). John K. Eaton has collaborated with scholars based in United States and Germany. Frequent co-authors include Stuart L. Schreiber, Vasanthi S. Viswanathan, Evgeni M. Frenkel, Mustafa Kocak, Ryan D. Spangler, Shannon Coy, Jordan Rossen, Boryana Petrova, Sandro Santagata and Mai Abdusamad. Their work appears in journals such as Nature, Science and Cell.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.