Brooke M. Emerling
- Molecular Biology top 5%
- Cancer Research top 5%
- Physiology top 10%
- Epidemiology top 10%
- Cell Biology top 5%
- Co-authors
- Navdeep S. ChandelStéphane J. H. RicoultLeonard GuarenteFrank WeinbergBenoı̂t ViolletEric L. BellÁngel R. NebredaJuinn-Lin Liu
- Topics
- Cancer, Hypoxia, and Metabolism (11 papers)Mitochondrial Function and Pathology (5 papers)PI3K/AKT/mTOR signaling in cancer (5 papers)
- Partner nations
- United StatesSwitzerlandIsrael
In The Last Decade
Brooke M. Emerling
35 papers receiving 2.1k citations
Peers
Comparison fields: 5 of 96
- Molecular Biology 1.4k
- Cancer Research 559
- Physiology 332
- Epidemiology 312
- Cell Biology 257
Countries citing papers authored by Brooke M. Emerling
This map shows the geographic impact of Brooke M. Emerling's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Brooke M. Emerling with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Brooke M. Emerling more than expected).
Fields of papers citing papers by Brooke M. Emerling
This network shows the impact of papers produced by Brooke M. Emerling. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Brooke M. Emerling. The network helps show where Brooke M. Emerling may publish in the future.
Co-authorship network of co-authors of Brooke M. Emerling
This figure shows the co-authorship network connecting the top 25 collaborators of Brooke M. Emerling. A scholar is included among the top collaborators of Brooke M. Emerling based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Brooke M. Emerling. Brooke M. Emerling is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 5 | |
| 2 | 7 | |
| 3 | 8 | |
| 4 | 77 | |
| 5 | 5 | |
| 6 | 5 | |
| 7 | 13 | |
| 8 | 85 | |
| 9 | 48 | |
| 10 | 144 | |
| 11 | 42 | |
| 12 | 49 | |
| 13 | 16 | |
| 14 | 31 | |
| 15 | SirT3 suppresses hypoxia inducible factor 1α and tumor growth by inhibiting mitochondrial ROS production | 372 |
| 16 | 4 | |
| 17 | 278 | |
| 18 | 87 | |
| 19 | 14 | |
| 20 | 37 |
About Brooke M. Emerling
Brooke M. Emerling is a scholar working on Cancer Research, Cell Biology and Hematology, having authored 36 papers that have together received 2.1k indexed citations. Recurring topics across this work include Cancer, Hypoxia, and Metabolism (11 papers), Mitochondrial Function and Pathology (5 papers) and PI3K/AKT/mTOR signaling in cancer (5 papers). The work is most often cited by research in Geriatrics and Gerontology (238 citations), Cancer Research (559 citations) and Physiology (111 citations). Brooke M. Emerling has collaborated with scholars based in United States, Switzerland and Israel. Frequent co-authors include Navdeep S. Chandel, Stéphane J. H. Ricoult, Leonard Guarente, Frank Weinberg, Benoı̂t Viollet, Eric L. Bell, Ángel R. Nebreda, Juinn-Lin Liu, Leonidas C. Platanias and Tak W. Mak. Their work appears in journals such as Cell, Proceedings of the National Academy of Sciences and Nature reviews. Cancer.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.