Benjamin D. Hopkins
- Molecular Biology top 2%
- Oncology top 5%
- Cancer Research top 2%
- Pulmonary and Respiratory Medicine top 5%
- Physiology top 5%
- Co-authors
- Lewis C. CantleyHonyin ChiuShubha BagrodiaRobert T. AbrahamMarcus D. GoncalvesRamon ParsonsCindy HodakoskiSarah M. Mense
- Topics
- PI3K/AKT/mTOR signaling in cancer (16 papers)Metabolism, Diabetes, and Cancer (7 papers)Protein Kinase Regulation and GTPase Signaling (4 papers)
- Partner nations
- United StatesSwitzerlandUnited Kingdom
In The Last Decade
Benjamin D. Hopkins
39 papers receiving 4.5k citations
Hit Papers
Peers
Comparison fields: 5 of 130
- Molecular Biology 3.0k
- Oncology 979
- Cancer Research 940
- Pulmonary and Respiratory Medicine 576
- Physiology 485
Countries citing papers authored by Benjamin D. Hopkins
This map shows the geographic impact of Benjamin D. Hopkins's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Benjamin D. Hopkins with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Benjamin D. Hopkins more than expected).
Fields of papers citing papers by Benjamin D. Hopkins
This network shows the impact of papers produced by Benjamin D. Hopkins. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Benjamin D. Hopkins. The network helps show where Benjamin D. Hopkins may publish in the future.
Co-authorship network of co-authors of Benjamin D. Hopkins
This figure shows the co-authorship network connecting the top 25 collaborators of Benjamin D. Hopkins. A scholar is included among the top collaborators of Benjamin D. Hopkins based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Benjamin D. Hopkins. Benjamin D. Hopkins is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 0 | |
| 2 | 0 | |
| 3 | 0 | |
| 4 | 2 | |
| 5 | 56 | |
| 6 | 7 | |
| 7 | 3 | |
| 8 | 3 | |
| 9 | 5 | |
| 10 | 25 | |
| 11 | 40 | |
| 12 | 85 | |
| 13 | Suppression of insulin feedback enhances the efficacy of PI3K inhibitorsbreakdown → | 481 |
| 14 | 2 | |
| 15 | Ten Years in, Afghan Myths Live On | 0 |
| 16 | 182 | |
| 17 | 61 | |
| 18 | 44 | |
| 19 | 87 | |
| 20 | 25 |
About Benjamin D. Hopkins
Benjamin D. Hopkins is a scholar working on Cancer Research, Oncology and Molecular Biology, having authored 44 papers that have together received 4.5k indexed citations. Recurring topics across this work include PI3K/AKT/mTOR signaling in cancer (16 papers), Metabolism, Diabetes, and Cancer (7 papers) and Protein Kinase Regulation and GTPase Signaling (4 papers). The work is most often cited by research in Cancer Research (940 citations), Molecular Biology (3.0k citations) and Genetics (398 citations). Benjamin D. Hopkins has collaborated with scholars based in United States, Switzerland and United Kingdom. Frequent co-authors include Lewis C. Cantley, Honyin Chiu, Shubha Bagrodia, Robert T. Abraham, Marcus D. Goncalves, Ramon Parsons, Cindy Hodakoski, Sarah M. Mense, Douglas Barrows and Laura E. Benjamin. Their work appears in journals such as Nature, Science and New England Journal of Medicine.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.