Shigeto Sato

7.0k total citations · 3 hit papers
53 papers, 5.0k citations indexed

About

Shigeto Sato is a scholar working on Neurology, Epidemiology and Molecular Biology. According to data from OpenAlex, Shigeto Sato has authored 53 papers receiving a total of 5.0k indexed citations (citations by other indexed papers that have themselves been cited), including 40 papers in Neurology, 24 papers in Epidemiology and 16 papers in Molecular Biology. Recurrent topics in Shigeto Sato's work include Parkinson's Disease Mechanisms and Treatments (40 papers), Autophagy in Disease and Therapy (24 papers) and Mitochondrial Function and Pathology (8 papers). Shigeto Sato is often cited by papers focused on Parkinson's Disease Mechanisms and Treatments (40 papers), Autophagy in Disease and Therapy (24 papers) and Mitochondrial Function and Pathology (8 papers). Shigeto Sato collaborates with scholars based in Japan, United States and Italy. Shigeto Sato's co-authors include Nobutaka Hattori, Shinji Saiki, Keiji Tanaka, Sumihiro Kawajiri, Masaaki Komatsu, Kei Okatsu, Noriyuki Matsuda, Mayumi Kimura, Fumiaki Sato and Yu‐shin Sou and has published in prestigious journals such as Nature Communications, SHILAP Revista de lepidopterología and The Journal of Cell Biology.

In The Last Decade

Shigeto Sato

53 papers receiving 5.0k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

PINK1 stabilized by mitochondrial depolarization recruits Parkin to damaged mitochondria and activates latent Parkin for mitophagy

2010 1.5k citations Noriyuki Matsuda, Shigeto Sato et al. The Journal of Cell Biology profile →
PINK1-mediated phosphorylation of the Parkin ubiquitin-like domain primes mitochondrial translocation of Parkin and regulates mitophagy

2012 461 citations Kahori Shiba‐Fukushima, Yuzuru Imai et al. Scientific Reports profile →
PINK1 autophosphorylation upon membrane potential dissipation is essential for Parkin recruitment to damaged mitochondria

2012 415 citations Kei Okatsu, Mayumi Kimura et al. profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Shigeto Sato Japan	30	2.8k	2.6k	2.0k	897	846	53	5.0k
Fabienne C. Fiesel United States	30	2.8k 1.0×	2.4k 0.9×	2.2k 1.1×	963 1.1×	779 0.9×	50	5.3k
Wolfdieter Springer United States	29	2.9k 1.1×	2.5k 1.0×	2.0k 1.0×	984 1.1×	777 0.9×	58	5.3k
Clément Gautier France	17	3.0k 1.1×	2.8k 1.1×	2.0k 1.0×	1.1k 1.2×	918 1.1×	23	5.4k
Seok Min Jin United States	12	3.4k 1.2×	3.0k 1.2×	1.5k 0.8×	1.1k 1.2×	612 0.7×	12	5.3k
Jordi Magrané United States	27	2.2k 0.8×	1.1k 0.4×	1.4k 0.7×	1.0k 1.1×	781 0.9×	36	3.9k
Olga Corti France	34	2.1k 0.8×	1.1k 0.4×	1.7k 0.8×	816 0.9×	1.1k 1.3×	63	4.0k
Hélène Plun‐Favreau United Kingdom	29	2.0k 0.7×	1.1k 0.4×	1.6k 0.8×	798 0.9×	814 1.0×	44	3.9k
Ghazaleh Ashrafi United States	11	2.2k 0.8×	1.5k 0.6×	748 0.4×	767 0.9×	670 0.8×	16	3.6k
Qian Cai United States	35	2.5k 0.9×	1.3k 0.5×	592 0.3×	1.4k 1.6×	1.0k 1.2×	55	4.5k
Shaida A. Andrabi United States	28	3.1k 1.1×	804 0.3×	1.4k 0.7×	792 0.9×	802 0.9×	45	5.5k

Countries citing papers authored by Shigeto Sato

Since Specialization

Citations

This map shows the geographic impact of Shigeto Sato's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Shigeto Sato with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Shigeto Sato more than expected).

Fields of papers citing papers by Shigeto Sato

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Shigeto Sato. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Shigeto Sato. The network helps show where Shigeto Sato may publish in the future.

Co-authorship network of co-authors of Shigeto Sato

This figure shows the co-authorship network connecting the top 25 collaborators of Shigeto Sato. A scholar is included among the top collaborators of Shigeto Sato based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Shigeto Sato. Shigeto Sato is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

Sort: Min cites: Since: Top N: Style:

20 of 20 papers shown

Hattori, Nobutaka & Shigeto Sato. (2024). Mitochondrial dysfunction in Parkinson’s disease. Journal of Neural Transmission. 131(12). 1415–1428. 5 indexed citations

Torii, Satoru, Satoko Arakawa, Shigeto Sato, et al.. (2023). Involvement of casein kinase 1 epsilon/delta (Csnk1e/d) in the pathogenesis of familial Parkinson's disease caused by CHCHD2. EMBO Molecular Medicine. 15(9). e17451–e17451. 5 indexed citations

Kawamoto, Hiroaki, Hisamasa Imai, Makoto Shirai, et al.. (2023). Gait improvement with wearable cyborg HAL trunk unit for parkinsonian patients: five case reports. Scientific Reports. 13(1). 6962–6962. 2 indexed citations

Mano, Hiroki, Miyu Nishikawa, Shinichi Ikushiro, et al.. (2023). Functional analysis of vitamin D receptor (VDR) using adenovirus vector. The Journal of Steroid Biochemistry and Molecular Biology. 230. 106275–106275. 7 indexed citations

Cossu, Davide, Kazumasa Yokoyama, Shigeto Sato, et al.. (2021). PARKIN modifies peripheral immune response and increases neuroinflammation in active experimental autoimmune encephalomyelitis (EAE). Journal of Neuroimmunology. 359. 577694–577694. 15 indexed citations

Sato, Shigeto & Nobutaka Hattori. (2018). Dopaminergic Neuron-Specific Autophagy-Deficient Mice. Methods in molecular biology. 1759. 173–175. 9 indexed citations

Sato, Shigeto, Yuanzhe Li, & Nobutaka Hattori. (2017). Lysosomal defects in ATP13A2 and GBA associated familial Parkinson’s disease. Journal of Neural Transmission. 124(11). 1395–1400. 12 indexed citations

Shiba‐Fukushima, Kahori, Kei‐ichi Ishikawa, Tsuyoshi Inoshita, et al.. (2017). Evidence that phosphorylated ubiquitin signaling is involved in the etiology of Parkinson’s disease. Human Molecular Genetics. 26(16). 3172–3185. 43 indexed citations

Sato, Shigeto, Masato Koike, Manabu Funayama, et al.. (2016). Lysosomal Storage of Subunit c of Mitochondrial ATP Synthase in Brain-Specific Atp13a2-Deficient Mice. American Journal Of Pathology. 186(12). 3074–3082. 28 indexed citations

10.

Amo, Taku, Shigeto Sato, Shinji Saiki, et al.. (2012). Mitochondrial membrane potential decrease caused by loss of PINK1 is not due to proton leak, but to respiratory chain defects. Biochimica et Biophysica Acta (BBA) - Bioenergetics. 1817. S78–S78. 3 indexed citations

11.

Shiba‐Fukushima, Kahori, Yuzuru Imai, Yasushi Ishihama, et al.. (2012). PINK1-mediated phosphorylation of the Parkin ubiquitin-like domain primes mitochondrial translocation of Parkin and regulates mitophagy. Scientific Reports. 2(1). 1002–1002. 461 indexed citations breakdown →

12.

Saiki, Shinji, Shigeto Sato, & Nobutaka Hattori. (2011). Molecular pathogenesis of Parkinson's disease: update. Journal of Neurology Neurosurgery & Psychiatry. 83(4). 430–436. 60 indexed citations

13.

Saiki, Shinji, Yukiko Sasazawa, Yoko Imamichi, et al.. (2011). Caffeine induces apoptosis by enhancement of autophagy via PI3K/Akt/mTOR/p70S6K inhibition. Autophagy. 7(2). 176–187. 364 indexed citations

14.

Matsuda, Noriyuki, Shigeto Sato, Kahori Shiba, et al.. (2010). PINK1 stabilized by mitochondrial depolarization recruits Parkin to damaged mitochondria and activates latent Parkin for mitophagy. The Journal of Cell Biology. 189(2). 211–221. 1542 indexed citations breakdown →

15.

Okatsu, Kei, Kazuto Nakada, Hiroshi Shitara, et al.. (2010). p62/SQSTM1 cooperates with Parkin for perinuclear clustering of depolarized mitochondria. Genes to Cells. 15(8). 887–900. 328 indexed citations

16.

Kawajiri, Sumihiro, Shinji Saiki, Shigeto Sato, et al.. (2010). PINK1 is recruited to mitochondria with parkin and associates with LC3 in mitophagy. FEBS Letters. 584(6). 1073–1079. 201 indexed citations

17.

Hatano, Taku, Shinichiro Kubo, Shigeto Sato, & Nobutaka Hattori. (2009). Pathogenesis of familial Parkinson’s disease: new insights based on monogenic forms of Parkinson’s disease. Journal of Neurochemistry. 111(5). 1075–1093. 79 indexed citations

18.

Hattori, Nobutaka & Shigeto Sato. (2007). Animal models of Parkinson's disease: Similarities and differences between the disease and models. Neuropathology. 27(5). 479–483. 19 indexed citations

19.

Hattori, Nobutaka, Yutaka Machida, Shigeto Sato, et al.. (2006). Molecular mechanisms of nigral neurodegeneration in Park2 and regulation of parkin protein by other proteins. PubMed. 205–208. 5 indexed citations

20.

Sato, Shigeto, Tomoki Chiba, Eri Sakata, et al.. (2005). 14‐3‐3η is a novel regulator of parkin ubiquitin ligase. The EMBO Journal. 25(1). 211–221. 101 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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