Junji Ezaki

7.9k total citations · 2 hit papers
41 papers, 4.8k citations indexed

About

Junji Ezaki is a scholar working on Molecular Biology, Physiology and Epidemiology. According to data from OpenAlex, Junji Ezaki has authored 41 papers receiving a total of 4.8k indexed citations (citations by other indexed papers that have themselves been cited), including 23 papers in Molecular Biology, 21 papers in Physiology and 16 papers in Epidemiology. Recurrent topics in Junji Ezaki's work include Lysosomal Storage Disorders Research (20 papers), Autophagy in Disease and Therapy (14 papers) and Cellular transport and secretion (9 papers). Junji Ezaki is often cited by papers focused on Lysosomal Storage Disorders Research (20 papers), Autophagy in Disease and Therapy (14 papers) and Cellular transport and secretion (9 papers). Junji Ezaki collaborates with scholars based in Japan, Canada and United States. Junji Ezaki's co-authors include Eiki Kominami, Takashi Ueno, Masaaki Komatsu, Isei Tanida, Keiji Tanaka, Yasuo Uchiyama, Satoshi Waguri, Tomoki Chiba, Junichi Iwata and Yoshinori Ohsumi and has published in prestigious journals such as Journal of Biological Chemistry, Journal of Neuroscience and The Journal of Cell Biology.

In The Last Decade

Junji Ezaki

41 papers receiving 4.8k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Impairment of starvation-induced and constitutive autophagy in Atg7 -deficient mice

2005 1.9k citations Masaaki Komatsu, Satoshi Waguri et al. The Journal of Cell Biology profile →
Structural Basis for Sorting Mechanism of p62 in Selective Autophagy

2008 642 citations Yoshinobu Ichimura, Taichi Kumanomidou et al. Journal of Biological Chemistry profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Junji Ezaki Japan	24	3.0k	2.2k	1.3k	1.1k	522	41	4.8k
Elena Shvets Israel	18	3.5k 1.2×	2.4k 1.1×	1.5k 1.1×	583 0.5×	622 1.2×	20	5.0k
Francesco Vetrini United States	13	3.1k 1.0×	2.5k 1.2×	1.1k 0.9×	977 0.9×	885 1.7×	31	5.3k
Akiko Kuma Japan	21	4.4k 1.5×	2.7k 1.2×	1.3k 1.0×	584 0.5×	628 1.2×	28	5.9k
Satoko Arakawa Japan	27	3.1k 1.0×	2.7k 1.2×	984 0.7×	788 0.7×	438 0.8×	49	5.2k
Serhiy Pankiv Norway	15	4.3k 1.4×	2.9k 1.3×	1.5k 1.1×	711 0.7×	595 1.1×	18	5.9k
Maho Hamasaki Japan	23	3.0k 1.0×	2.5k 1.1×	1.5k 1.2×	648 0.6×	581 1.1×	41	4.9k
Eisuke Itakura Japan	24	4.0k 1.3×	2.3k 1.0×	1.9k 1.4×	698 0.7×	845 1.6×	41	5.4k
Farah H. Siddiqi United Kingdom	16	2.5k 0.8×	1.9k 0.9×	1.1k 0.8×	732 0.7×	542 1.0×	23	4.3k
Heidi Outzen Norway	6	5.0k 1.7×	3.5k 1.6×	1.6k 1.2×	718 0.7×	649 1.2×	7	6.9k
Shouqing Luo United Kingdom	30	3.6k 1.2×	3.4k 1.5×	1.3k 1.0×	800 0.7×	492 0.9×	52	6.4k

Countries citing papers authored by Junji Ezaki

Since Specialization

Citations

This map shows the geographic impact of Junji Ezaki's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Junji Ezaki with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Junji Ezaki more than expected).

Fields of papers citing papers by Junji Ezaki

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Junji Ezaki. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Junji Ezaki. The network helps show where Junji Ezaki may publish in the future.

Co-authorship network of co-authors of Junji Ezaki

This figure shows the co-authorship network connecting the top 25 collaborators of Junji Ezaki. A scholar is included among the top collaborators of Junji Ezaki based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Junji Ezaki. Junji Ezaki is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Hakozaki, Michiyuki, Hirosumi Tamura, Aki Yoshida, et al.. (2018). Establishment and Characterization of a Novel Human Clear-cell Sarcoma of Soft-tissue Cell Line, RSAR001, Derived from Pleural Effusion of a Patient with Pleural Dissemination. Anticancer Research. 38(9). 5035–5042. 2 indexed citations

Sato, Shigeto, Masato Koike, Manabu Funayama, et al.. (2016). Lysosomal Storage of Subunit c of Mitochondrial ATP Synthase in Brain-Specific Atp13a2-Deficient Mice. American Journal Of Pathology. 186(12). 3074–3082. 28 indexed citations

Okabe, Naoyuki, Junji Ezaki, Takumi Yamaura, et al.. (2015). FAM83B is a novel biomarker for diagnosis and prognosis of lung squamous cell carcinoma. International Journal of Oncology. 46(3). 999–1006. 40 indexed citations

Zheng, Dongmei, Zehua Bian, Norihiko Furuya, et al.. (2014). A treadmill exercise reactivates the signaling of the mammalian target of rapamycin (mTor) in the skeletal muscles of starved mice. Biochemical and Biophysical Research Communications. 456(1). 519–526. 13 indexed citations

Furuya, Norihiko, Shin‐ichi Ikeda, Shigeto Sato, et al.. (2014). PARK2/Parkin-mediated mitochondrial clearance contributes to proteasome activation during slow-twitch muscle atrophy via NFE2L1 nuclear translocation. Autophagy. 10(4). 631–641. 40 indexed citations

Nishina, Takashi, Sachiko Komazawa‐Sakon, Saeko Yanaka, et al.. (2012). Interleukin-11 Links Oxidative Stress and Compensatory Proliferation. Science Signaling. 5(207). ra5–ra5. 91 indexed citations

Ezaki, Junji, Naomi Matsumoto, Mitsue Takeda‐Ezaki, et al.. (2011). Liver autophagy contributes to the maintenance of blood glucose and amino acid levels. Autophagy. 7(7). 727–736. 225 indexed citations

Ushio, Hiroko, Takashi Ueno, Yuko Kojima, et al.. (2011). Crucial role for autophagy in degranulation of mast cells. Journal of Allergy and Clinical Immunology. 127(5). 1267–1276.e6. 116 indexed citations

Ueno, Takashi, Junji Ezaki, & Eiki Kominami. (2011). Metabolic contribution of hepatic autophagic proteolysis: Old wine in new bottles. Biochimica et Biophysica Acta (BBA) - Proteins and Proteomics. 1824(1). 51–58. 17 indexed citations

10.

Ezaki, Junji, Eiki Kominami, & Takashi Ueno. (2011). Peroxisome degradation in mammals. IUBMB Life. 63(11). 1001–1008. 12 indexed citations

11.

Ezaki, Junji, Masaaki Komatsu, Sadaki Yokota, Takashi Ueno, & Eiki Kominami. (2009). Chapter 14 Method for Monitoring Pexophagy in Mammalian Cells. Methods in enzymology on CD-ROM/Methods in enzymology. 452. 215–226. 7 indexed citations

12.

Ichimura, Yoshinobu, Taichi Kumanomidou, Yu‐shin Sou, et al.. (2008). Structural Basis for Sorting Mechanism of p62 in Selective Autophagy. Journal of Biological Chemistry. 283(33). 22847–22857. 642 indexed citations breakdown →

13.

Komatsu, Masaaki, Satoshi Waguri, Takashi Ueno, et al.. (2005). Impairment of starvation-induced and constitutive autophagy in Atg7 -deficient mice. The Journal of Cell Biology. 169(3). 425–434. 1941 indexed citations breakdown →

14.

Iwata, J., Junji Ezaki, Masaaki Komatsu, et al.. (2005). Excess Peroxisomes Are Degraded by Autophagic Machinery in Mammals. Journal of Biological Chemistry. 281(7). 4035–4041. 186 indexed citations

15.

Ezaki, Junji & Eiki Kominami. (2004). The Intracellular Location and Function Of Proteins Of Neuronal Ceroid Lipofuscinoses. Brain Pathology. 14(1). 77–85. 30 indexed citations

16.

Uchida, Shinichi, Junji Ezaki, Tatemitsu Rai, et al.. (2002). CLC‐3 deficiency leads to phenotypes similar to human neuronal ceroid lipofuscinosis. Genes to Cells. 7(6). 597–605. 126 indexed citations

17.

Ezaki, Junji, et al.. (1997). Decreased Lysosomal Subunit c-Degrading Activity in Fibroblasts from Patients with Late Infantile Neuronal Ceroid Lipofuscinosis. Neuropediatrics. 28(1). 53–55. 9 indexed citations

18.

Ezaki, Junji, et al.. (1996). Lysosomal Proteinosis Based on Decreased Degration of a Specific Protein, Mitochondrial ATP Synthase Subunit C. Advances in experimental medicine and biology. 389. 121–128. 1 indexed citations

19.

Ezaki, Junji, L. S. Wolfe, & Eiki Kominami. (1995). DEFECT OF PROTEOLYSIS OF MITOCHONDRIAL ATP SYNTHASE SUBUNIT C IN NEURONAL CEROID LIPOFUSCINOSIS. Gerontology. 41(2). 259–270. 6 indexed citations

20.

Ezaki, Junji, Leonhard S. Wolfe, Tomihiko Higuti, Kazumi Ishidoh, & Eiki Kominami. (1995). Specific Delay of Degradation of Mitochondrial ATP Synthase Subunit c in Late Infantile Neuronal Ceroid Lipofuscinosis (Batten Disease). Journal of Neurochemistry. 64(2). 733–741. 63 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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