Wolfdieter Springer

17.6k total citations · 1 hit paper
58 papers, 5.3k citations indexed

About

Wolfdieter Springer is a scholar working on Neurology, Epidemiology and Molecular Biology. According to data from OpenAlex, Wolfdieter Springer has authored 58 papers receiving a total of 5.3k indexed citations (citations by other indexed papers that have themselves been cited), including 36 papers in Neurology, 30 papers in Epidemiology and 24 papers in Molecular Biology. Recurrent topics in Wolfdieter Springer's work include Parkinson's Disease Mechanisms and Treatments (33 papers), Autophagy in Disease and Therapy (29 papers) and Ubiquitin and proteasome pathways (12 papers). Wolfdieter Springer is often cited by papers focused on Parkinson's Disease Mechanisms and Treatments (33 papers), Autophagy in Disease and Therapy (29 papers) and Ubiquitin and proteasome pathways (12 papers). Wolfdieter Springer collaborates with scholars based in United States, Germany and Poland. Wolfdieter Springer's co-authors include Fabienne C. Fiesel, Philipp J. Kahle, Sven Geisler, Kira M. Holmström, Oliver C. Rothfuss, Xu Hou, Thomas R. Caulfield, Jens O. Watzlawik, Stephanie Weber and Elisabeth L. Moussaud-Lamodière and has published in prestigious journals such as Cell, Proceedings of the National Academy of Sciences and Journal of Biological Chemistry.

In The Last Decade

Wolfdieter Springer

55 papers receiving 5.3k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

PINK1/Parkin-mediated mitophagy is dependent on VDAC1 and p62/SQSTM1

2010 2.3k citations Sven Geisler, Kira M. Holmström et al. Nature Cell Biology profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Wolfdieter Springer United States	29	2.9k	2.5k	2.0k	984	777	58	5.3k
Fabienne C. Fiesel United States	30	2.8k 1.0×	2.4k 1.0×	2.2k 1.1×	963 1.0×	779 1.0×	50	5.3k
Shigeto Sato Japan	30	2.8k 1.0×	2.6k 1.0×	2.0k 1.0×	897 0.9×	846 1.1×	53	5.0k
Seok Min Jin United States	12	3.4k 1.2×	3.0k 1.2×	1.5k 0.8×	1.1k 1.1×	612 0.8×	12	5.3k
Der‐Fen Suen Taiwan	14	4.1k 1.4×	3.1k 1.2×	1.4k 0.7×	999 1.0×	657 0.8×	17	6.2k
Kei Okatsu Japan	16	2.8k 1.0×	2.9k 1.1×	1.5k 0.7×	727 0.7×	440 0.6×	27	4.4k
Atsushi Tanaka Japan	15	5.0k 1.7×	4.4k 1.7×	2.2k 1.1×	1.3k 1.4×	903 1.2×	30	7.8k
Jordi Magrané United States	27	2.2k 0.7×	1.1k 0.4×	1.4k 0.7×	1.0k 1.0×	781 1.0×	36	3.9k
Ghazaleh Ashrafi United States	11	2.2k 0.8×	1.5k 0.6×	748 0.4×	767 0.8×	670 0.9×	16	3.6k
Hélène Plun‐Favreau United Kingdom	29	2.0k 0.7×	1.1k 0.4×	1.6k 0.8×	798 0.8×	814 1.0×	44	3.9k
Shaida A. Andrabi United States	28	3.1k 1.1×	804 0.3×	1.4k 0.7×	792 0.8×	802 1.0×	45	5.5k

Countries citing papers authored by Wolfdieter Springer

Since Specialization

Citations

This map shows the geographic impact of Wolfdieter Springer's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Wolfdieter Springer with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Wolfdieter Springer more than expected).

Fields of papers citing papers by Wolfdieter Springer

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Wolfdieter Springer. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Wolfdieter Springer. The network helps show where Wolfdieter Springer may publish in the future.

Co-authorship network of co-authors of Wolfdieter Springer

This figure shows the co-authorship network connecting the top 25 collaborators of Wolfdieter Springer. A scholar is included among the top collaborators of Wolfdieter Springer based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Wolfdieter Springer. Wolfdieter Springer is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Naddaf, Elie, et al.. (2025). Mitochondria-centred metabolomic map of inclusion body myositis: sex-specific alterations in central carbon metabolism. Annals of the Rheumatic Diseases. 84(8). 1375–1386.

Rasool, Shafqat, Nathalie Croteau, Simon Veyron, et al.. (2024). Identification and structural characterization of small molecule inhibitors of PINK1. Scientific Reports. 14(1). 7739–7739. 5 indexed citations

Naddaf, Elie, Thi Kim Oanh Nguyen, Jens O. Watzlawik, et al.. (2024). NLRP3 Inflammasome Activation and Altered Mitophagy Are Key Pathways in Inclusion Body Myositis. Journal of Cachexia Sarcopenia and Muscle. 16(1). e13672–e13672. 7 indexed citations

Yan, Tingxiang, Michael G. Heckman, Chia‐Chen Liu, et al.. (2024). The UFMylation pathway is impaired in Alzheimer’s disease. Molecular Neurodegeneration. 19(1). 97–97. 5 indexed citations

Siuda, Joanna, Jarosław Sławek, Andreas Puschmann, et al.. (2024). Structural and Functional Characterization of the Most Frequent Pathogenic PRKN Substitution p.R275W. Cells. 13(18). 1540–1540. 2 indexed citations

Zhu, Xingxing, Yue Wu, Yanfeng Li, et al.. (2024). The nutrient-sensing Rag-GTPase complex in B cells controls humoral immunity via TFEB/TFE3-dependent mitochondrial fitness. Nature Communications. 15(1). 10163–10163. 2 indexed citations

Watzlawik, Jens O., Xu Hou, Dominika Fričová, et al.. (2020). Sensitive ELISA-based detection method for the mitophagy marker p-S65-Ub in human cells, autopsy brain, and blood samples. Autophagy. 17(9). 2613–2628. 35 indexed citations

Park, Jae Hyeon, Jeremy D. Burgess, Fabienne C. Fiesel, et al.. (2020). Alpha-synuclein-induced mitochondrial dysfunction is mediated via a sirtuin 3-dependent pathway. Molecular Neurodegeneration. 15(1). 5–5. 136 indexed citations

Hou, Xu, Jens O. Watzlawik, Fabienne C. Fiesel, & Wolfdieter Springer. (2020). Autophagy in Parkinson's Disease. Journal of Molecular Biology. 432(8). 2651–2672. 251 indexed citations

10.

Luo, Shilin, Seong Su Kang, Xia Liu, et al.. (2019). Akt Phosphorylates NQO1 and Triggers its Degradation, Abolishing Its Antioxidative Activities in Parkinson's Disease. Journal of Neuroscience. 39(37). 7291–7305. 56 indexed citations

11.

Paulus, Aneel, Sharoon Akhtar, Alak Manna, et al.. (2017). Waldenstrom macroglobulinemia cells devoid of BTKC481S or CXCR4WHIM-like mutations acquire resistance to ibrutinib through upregulation of Bcl-2 and AKT resulting in vulnerability towards venetoclax or MK2206 treatment. Blood Cancer Journal. 7(5). e565–e565. 44 indexed citations

12.

Kim, Jaekwang, Fabienne C. Fiesel, Roman Hudec, et al.. (2016). miR-27a and miR-27b regulate autophagic clearance of damaged mitochondria by targeting PTEN-induced putative kinase 1 (PINK1). Molecular Neurodegeneration. 11(1). 55–55. 128 indexed citations

13.

Truban, Dominika, Xu Hou, Thomas R. Caulfield, Fabienne C. Fiesel, & Wolfdieter Springer. (2016). PINK1, Parkin, and Mitochondrial Quality Control: What can we Learn about Parkinson’s Disease Pathobiology?. Journal of Parkinson s Disease. 7(1). 13–29. 176 indexed citations

14.

Yue, Mei, Kelly M. Hinkle, Paul Davies, et al.. (2015). Progressive dopaminergic alterations and mitochondrial abnormalities in LRRK2 G2019S knock-in mice. Neurobiology of Disease. 78. 172–195. 180 indexed citations

15.

Fiesel, Fabienne C., Elisabeth L. Moussaud-Lamodière, Maya Ando, & Wolfdieter Springer. (2014). Select E2 enzymes differentially regulate parkin activation and mitophagy. Journal of Cell Science. 127(Pt 16). 3488–504. 47 indexed citations

16.

Shannon, Barbara, Alexandra I. Soto‐Ortolaza, Sruti Rayaprolu, et al.. (2014). Genetic variation of the retromer subunits VPS26A/B-VPS29 in Parkinson's disease. Neurobiology of Aging. 35(8). 1958.e1–1958.e2. 20 indexed citations

17.

Springer, Wolfdieter & Philipp J. Kahle. (2011). Regulation of PINK1-Parkin-mediated mitophagy. Autophagy. 7(3). 266–278. 143 indexed citations

18.

Waak, Jens, Stephanie Weber, Fabienne C. Fiesel, et al.. (2010). Parkinson’s disease-associated DJ-1 modulates innate immunity signaling in Caenorhabditis elegans. Journal of Neural Transmission. 117(5). 599–604. 30 indexed citations

19.

Geisler, Sven, Kira M. Holmström, Fabienne C. Fiesel, et al.. (2010). PINK1/Parkin-mediated mitophagy is dependent on VDAC1 and p62/SQSTM1. Nature Cell Biology. 12(2). 119–131. 2297 indexed citations breakdown →

20.

Springer, Wolfdieter & Philipp J. Kahle. (2006). Mechanisms and models of α-synuclein-related neurodegeneration. Current Neurology and Neuroscience Reports. 6(5). 432–436. 15 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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