Kei‐ichi Ishikawa

2.9k total citations
72 papers, 1.9k citations indexed

About

Kei‐ichi Ishikawa is a scholar working on Molecular Biology, Neurology and Cellular and Molecular Neuroscience. According to data from OpenAlex, Kei‐ichi Ishikawa has authored 72 papers receiving a total of 1.9k indexed citations (citations by other indexed papers that have themselves been cited), including 27 papers in Molecular Biology, 25 papers in Neurology and 20 papers in Cellular and Molecular Neuroscience. Recurrent topics in Kei‐ichi Ishikawa's work include Parkinson's Disease Mechanisms and Treatments (23 papers), Genetic Neurodegenerative Diseases (11 papers) and Pluripotent Stem Cells Research (9 papers). Kei‐ichi Ishikawa is often cited by papers focused on Parkinson's Disease Mechanisms and Treatments (23 papers), Genetic Neurodegenerative Diseases (11 papers) and Pluripotent Stem Cells Research (9 papers). Kei‐ichi Ishikawa collaborates with scholars based in Japan, United States and United Kingdom. Kei‐ichi Ishikawa's co-authors include Nobutaka Hattori, Shinji Saiki, Yoko Imamichi, Yoshio Saitō, Katsuaki Ono, Toshihiro Akaike, Wado Akamatsu, Akira Kurita, Masayuki Ishihara and Shigeto Sato and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Circulation and Journal of Neuroscience.

In The Last Decade

Kei‐ichi Ishikawa

66 papers receiving 1.9k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Kei‐ichi Ishikawa Japan 20 653 488 356 271 255 72 1.9k
Chiung‐Chyi Shen Taiwan 30 762 1.2× 634 1.3× 570 1.6× 313 1.2× 660 2.6× 152 2.9k
Wei Jin China 31 1.3k 2.0× 254 0.5× 240 0.7× 392 1.4× 282 1.1× 75 2.8k
Srinivasulu Chigurupati United States 26 1.1k 1.7× 339 0.7× 266 0.7× 442 1.6× 172 0.7× 41 3.3k
Shahla Shojaei Canada 25 1.0k 1.6× 177 0.4× 674 1.9× 181 0.7× 255 1.0× 49 2.4k
Tongyu Zhang China 25 792 1.2× 564 1.2× 257 0.7× 141 0.5× 102 0.4× 94 2.1k
Jimin Wu China 25 1.1k 1.8× 1.2k 2.4× 605 1.7× 233 0.9× 228 0.9× 62 2.9k
Vincent Bérézowski France 25 880 1.3× 235 0.5× 330 0.9× 238 0.9× 324 1.3× 49 2.5k
Yan Peng China 34 1.1k 1.7× 201 0.4× 309 0.9× 181 0.7× 742 2.9× 133 3.3k

Countries citing papers authored by Kei‐ichi Ishikawa

Since Specialization
Citations

This map shows the geographic impact of Kei‐ichi Ishikawa's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Kei‐ichi Ishikawa with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Kei‐ichi Ishikawa more than expected).

Fields of papers citing papers by Kei‐ichi Ishikawa

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Kei‐ichi Ishikawa. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Kei‐ichi Ishikawa. The network helps show where Kei‐ichi Ishikawa may publish in the future.

Co-authorship network of co-authors of Kei‐ichi Ishikawa

This figure shows the co-authorship network connecting the top 25 collaborators of Kei‐ichi Ishikawa. A scholar is included among the top collaborators of Kei‐ichi Ishikawa based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Kei‐ichi Ishikawa. Kei‐ichi Ishikawa is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Ishikawa, Kei‐ichi, Ayami Okuzumi, Hiroyo Yoshino, Nobutaka Hattori, & Wado Akamatsu. (2024). Generation of hiPSCs (JUCGRMi003-A) from a patient with Parkinson’s disease with PARK2 mutation. Stem Cell Research. 76. 103323–103323.
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Abe, Yuka, et al.. (2024). Comprehensive Gene Expression Analysis Using Human Induced Pluripotent Stem Cells Derived from Patients with Sleep Bruxism: A Preliminary In Vitro Study. International Journal of Molecular Sciences. 25(23). 13141–13141. 1 indexed citations
4.
Torii, Satoru, Satoko Arakawa, Shigeto Sato, et al.. (2023). Involvement of casein kinase 1 epsilon/delta (Csnk1e/d) in the pathogenesis of familial Parkinson's disease caused by CHCHD2. EMBO Molecular Medicine. 15(9). e17451–e17451. 5 indexed citations
5.
Ishikawa, Kei‐ichi, et al.. (2023). Generation of a control iPS cell line (JUCGRMi006-A) with no abnormalities in Parkinson's disease-related genes. Stem Cell Research. 74. 103270–103270.
6.
Ishikawa, Kei‐ichi, et al.. (2023). Generation of three clones (JUCGRMi002-A, B, C) of induced pluripotent stem cells from a Parkinson’s disease patient with SNCA duplication. Stem Cell Research. 74. 103296–103296. 1 indexed citations
7.
Nakamura, Ryota, Risa Nonaka, Genko Oyama, et al.. (2023). A defined method for differentiating human iPSCs into midbrain dopaminergic progenitors that safely restore motor deficits in Parkinson’s disease. Frontiers in Neuroscience. 17. 1202027–1202027. 10 indexed citations
8.
Kim, Jeonghyun, et al.. (2021). Uniaxially fixed mechanical boundary condition elicits cellular alignment in collagen matrix with induction of osteogenesis. Scientific Reports. 11(1). 9009–9009. 7 indexed citations
9.
Yamaguchi, Akihiro, Kei‐ichi Ishikawa, Tsuyoshi Inoshita, et al.. (2020). Identifying Therapeutic Agents for Amelioration of Mitochondrial Clearance Disorder in Neurons of Familial Parkinson Disease. Stem Cell Reports. 14(6). 1060–1075. 43 indexed citations
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Suzuki, Sadafumi, Wado Akamatsu, Takefumi Sone, et al.. (2017). Efficient induction of dopaminergic neuron differentiation from induced pluripotent stem cells reveals impaired mitophagy in PARK2 neurons. Biochemical and Biophysical Research Communications. 483(1). 88–93. 56 indexed citations
12.
Ishikawa, Kei‐ichi, Akihiro Yamaguchi, Hideyuki Okano, & Wado Akamatsu. (2017). Assessment of Mitophagy in iPS Cell-Derived Neurons. Methods in molecular biology. 1759. 59–67. 5 indexed citations
13.
Shiba‐Fukushima, Kahori, Kei‐ichi Ishikawa, Tsuyoshi Inoshita, et al.. (2017). Evidence that phosphorylated ubiquitin signaling is involved in the etiology of Parkinson’s disease. Human Molecular Genetics. 26(16). 3172–3185. 43 indexed citations
14.
Shiba, Kahori, Kei‐ichi Ishikawa, Masashi Takanashi, et al.. (2016). Evidence that phosphorylated ubiquitin signaling is involved in the etiology of Parkinson's disease. 5 indexed citations
15.
Dowaki, Shoichi, Koji Fujita, Kei‐ichi Ishikawa, et al.. (2015). Endoscopic biliary drainage as a bridging procedure to single-stage surgery for perforated choledochal cyst: a case report and review of the literature. Surgical Case Reports. 1(1). 117–117. 10 indexed citations
16.
Ishikawa, Kei‐ichi, Shinji Saiki, Norihiko Furuya, et al.. (2014). p150glued-Associated Disorders Are Caused by Activation of Intrinsic Apoptotic Pathway. PLoS ONE. 9(4). e94645–e94645. 14 indexed citations
17.
Saiki, Shinji, Yukiko Sasazawa, Yoko Imamichi, et al.. (2011). Caffeine induces apoptosis by enhancement of autophagy via PI3K/Akt/mTOR/p70S6K inhibition. Autophagy. 7(2). 176–187. 364 indexed citations
18.
Kurihara, Hiroyuki, et al.. (2008). New Classification of Anal Fistula: Clarifying Low Intersphincteric Fistula and Posterior Complex Fistula. Nihon Daicho Komonbyo Gakkai Zasshi. 61(8). 467–475. 3 indexed citations
19.
Ueno, Hideki, Hidetaka Mochizuki, Yojiro Hashiguchi, et al.. (2003). Preoperative Parameters Expanding the Indication of Sphincter Preserving Surgery in Patients With Advanced Low Rectal Cancer. Annals of Surgery. 239(1). 34–42. 73 indexed citations
20.
Noguchi, T., Kei‐ichi Ishikawa, & K. Ninagawa. (1999). Effects of terrestrial weathering on the matrix mineralogy of Colony CO3 chondrite. Institutional Repository National Institute of Polar Research (National Institute of Polar Research (Japan)). 12(12). 36–56. 6 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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