Kazuto Nakada
Impact in
- Clinical Biochemistry top 0.1%
- Metabolism and Genetic Disorders
- Aging top 2%
Papers in
-
- Mitochondrial Function and Pathology 79
- ATP Synthase and ATPases Research 40
- Muscle Physiology and Disorders 10
- RNA modifications and cancer 10
- RNA and protein synthesis mechanisms 8
- DNA Repair Mechanisms 6
-
- Metabolism and Genetic Disorders 48
- Co-authors
- Jun‐Ichi Hayashi (55 shared papers)Kaori Ishikawa (34 shared papers)Kotoyo Isobe (6 shared papers)Keizo Takenaga (10 shared papers)Hirotake Imanishi (8 shared papers)Miho Akimoto (4 shared papers)Nobuko Koshikawa (3 shared papers)Tomoko Ono (8 shared papers)
- Journals
- Biochemical and Biophysical Research Communications (12 papers)EXPERIMENTAL ANIMALS (6 papers)Mitochondrion (6 papers)Scientific Reports (5 papers)PLoS ONE (5 papers)
- Partner nations
- JapanUnited StatesFinland
In The Last Decade
Kazuto Nakada
97 papers receiving 5.1k citations
Kazuto Nakada's Hit Papers
Peers
Comparison fields: 5 of 125
- Clinical Biochemistry 1.2k
- Aging 117
- Molecular Biology 3.9k
- Cancer Research 776
- Epidemiology 802
Countries citing papers authored by Kazuto Nakada
This map shows the geographic impact of Kazuto Nakada's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Kazuto Nakada with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Kazuto Nakada more than expected).
Fields of papers citing papers by Kazuto Nakada
This network shows the impact of papers produced by Kazuto Nakada. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Kazuto Nakada. The network helps show where Kazuto Nakada may publish in the future.
Co-authors
The 25 scholars most cited alongside Kazuto Nakada, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
Showing the 20 most-cited of 99 papers — load more, or switch the sort, to bring in the rest.
| # | Work | ||
|---|---|---|---|
| 1 | ROS-Generating Mitochondrial DNA Mutations Can Regulate Tumor Cell Metastasis Hit paper breakdown → | 2008 | 1173 |
| 2 | Persistent activation of Nrf2 through p62 in hepatocellular carcinoma cells Hit paper breakdown → | 2011 | 504 |
| 3 | 2001 | 347 | |
| 4 | 2001 | 342 | |
| 5 | 2010 | 330 | |
| 6 | 2000 | 299 | |
| 7 | 2006 | 206 | |
| 8 | 2014 | 152 | |
| 9 | 2005 | 130 | |
| 10 | 2002 | 100 | |
| 11 | 2011 | 93 | |
| 12 | 2010 | 67 | |
| 13 | 2015 | 66 | |
| 14 | 2012 | 60 | |
| 15 | 2006 | 59 | |
| 16 | 2005 | 47 | |
| 17 | 2018 | 46 | |
| 18 | 2017 | 44 | |
| 19 | 2008 | 42 | |
| 20 | 2007 | 41 |
About Kazuto Nakada
Kazuto Nakada is a scholar working on Molecular Biology, Clinical Biochemistry, Cancer Research, Surgery and Cardiology and Cardiovascular Medicine, having authored 99 papers that have together received 5.2k indexed citations. Recurring topics across this work include Mitochondrial Function and Pathology (79 papers), Metabolism and Genetic Disorders (48 papers), ATP Synthase and ATPases Research (40 papers), Muscle Physiology and Disorders (10 papers), RNA modifications and cancer (10 papers), Cancer, Hypoxia, and Metabolism (9 papers), RNA and protein synthesis mechanisms (8 papers) and DNA Repair Mechanisms (6 papers). The work is most often cited by research in Clinical Biochemistry (1.2k citations), Aging (117 citations), Molecular Biology (3.9k citations), Cancer Research (776 citations) and Epidemiology (802 citations). Kazuto Nakada has collaborated with scholars based in Japan, United States and Finland. Frequent co-authors include Jun‐Ichi Hayashi, Kaori Ishikawa, Kotoyo Isobe, Keizo Takenaga, Hirotake Imanishi, Miho Akimoto, Nobuko Koshikawa, Tomoko Ono, Yoshio Honma and Aya Yamaguchi. Their work appears in journals such as Biochemical and Biophysical Research Communications, EXPERIMENTAL ANIMALS, Mitochondrion, Scientific Reports and PLoS ONE.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.