Ryuichi Amakawa

3.7k total citations · 1 hit paper
59 papers, 3.0k citations indexed

About

Ryuichi Amakawa is a scholar working on Immunology, Pathology and Forensic Medicine and Genetics. According to data from OpenAlex, Ryuichi Amakawa has authored 59 papers receiving a total of 3.0k indexed citations (citations by other indexed papers that have themselves been cited), including 31 papers in Immunology, 18 papers in Pathology and Forensic Medicine and 14 papers in Genetics. Recurrent topics in Ryuichi Amakawa's work include Lymphoma Diagnosis and Treatment (17 papers), Immunotherapy and Immune Responses (16 papers) and Immune Cell Function and Interaction (15 papers). Ryuichi Amakawa is often cited by papers focused on Lymphoma Diagnosis and Treatment (17 papers), Immunotherapy and Immune Responses (16 papers) and Immune Cell Function and Interaction (15 papers). Ryuichi Amakawa collaborates with scholars based in Japan, United States and Canada. Ryuichi Amakawa's co-authors include Shirou Fukuhara, Tomoki� Ito, Muneo Inaba, Kayo Inaba, Susumu Ikehara, Kenichirou Tajima, Andrew Wakeham, Yoshio Ozaki, Kazutaka Uehira and Thomas M. Kündig and has published in prestigious journals such as Cell, The Journal of Experimental Medicine and Blood.

In The Last Decade

Ryuichi Amakawa

56 papers receiving 3.0k citations

Hit Papers

Targeted disruption of IRF-1 or IRF-2 results in abnormal... 1993 2026 2004 2015 1993 100 200 300 400 500

Peers

Ryuichi Amakawa
Ryuichi Amakawa
Citations per year, relative to Ryuichi Amakawa Ryuichi Amakawa (= 1×) peers Pierre Garrone

Countries citing papers authored by Ryuichi Amakawa

Since Specialization
Citations

This map shows the geographic impact of Ryuichi Amakawa's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ryuichi Amakawa with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ryuichi Amakawa more than expected).

Fields of papers citing papers by Ryuichi Amakawa

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Ryuichi Amakawa. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ryuichi Amakawa. The network helps show where Ryuichi Amakawa may publish in the future.

Co-authorship network of co-authors of Ryuichi Amakawa

This figure shows the co-authorship network connecting the top 25 collaborators of Ryuichi Amakawa. A scholar is included among the top collaborators of Ryuichi Amakawa based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ryuichi Amakawa. Ryuichi Amakawa is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
2.
Miyamoto, Rie, Tomoki� Ito, Shosaku� Nomura, et al.. (2010). Inhibitor of IκB kinase activity, BAY 11-7082, interferes with interferon regulatory factor 7 nuclear translocation and type I interferon production by plasmacytoid dendritic cells. Arthritis Research & Therapy. 12(3). R87–R87. 25 indexed citations
3.
Miyamoto, Rie, Keiko Shimamoto, Makoto Ogata, et al.. (2010). Interferon-α and interleukin-12 are induced, respectively, by double-stranded DNA and single-stranded RNA in human myeloid dendritic cells. Immunology. 132(2). 165–173. 18 indexed citations
4.
Iwai, Hiroshi, et al.. (2009). Parotid mucosa‐associated lymphoid tissue lymphoma regression after Helicobacter pylori eradication. The Laryngoscope. 119(8). 1491–1494. 15 indexed citations
5.
Torii, Yoshitaro, Tomoki� Ito, Ryuichi Amakawa, et al.. (2008). Imidazoquinoline Acts as Immune Adjuvant for Functional Alteration of Thymic Stromal Lymphopoietin-Mediated Allergic T Cell Response. The Journal of Immunology. 181(8). 5340–5349. 21 indexed citations
6.
Yokoi, Takashi, Ryuichi Amakawa, H. Sugimoto, et al.. (2008). Mycobacterium bovis Bacillus Calmette-Guerin suppresses inflammatory Th2 responses by inducing functional alteration of TSLP-activated dendritic cells. International Immunology. 20(10). 1321–1329. 19 indexed citations
7.
Nakamura, Kōichi, Ryuichi Amakawa, Masashi Takebayashi, et al.. (2005). IL-4-producing CD8+ T cells may be an immunological hallmark of chronic GVHD. Bone Marrow Transplantation. 36(7). 639–647. 11 indexed citations
8.
Ito, Tomoki�, Ryuichi Amakawa, Muneo Inaba, et al.. (2004). Plasmacytoid Dendritic Cells Regulate Th Cell Responses through OX40 Ligand and Type I IFNs. The Journal of Immunology. 172(7). 4253–4259. 152 indexed citations
9.
Takebayashi, Masashi, Ryuichi Amakawa, Michihiko Miyaji, et al.. (2004). Blood dendritic cells are decreased in acute graft-versus-host disease. Bone Marrow Transplantation. 33(10). 989–996. 14 indexed citations
10.
Ito, Tomoki�, Ryuichi Amakawa, & Shirou Fukuhara. (2002). Roles of toll-like receptors in natural interferon-producing cells as sensors in immune surveillance. Human Immunology. 63(12). 1120–1125. 47 indexed citations
11.
Inoue, Hiroshi, Michihiko Miyaji, Atsushi Kosugi, et al.. (2002). Lipid rafts as the signaling scaffold for NK cell activation: tyrosine phosphorylation and association of LAT with phosphatidylinositol 3-kinase and phospholipase C-γ following CD2 stimulation. European Journal of Immunology. 32(8). 2188–2188. 46 indexed citations
12.
Ito, Tomoki�, Ryuichi Amakawa, Tsuneyasu Kaisho, et al.. (2002). Interferon-α and Interleukin-12 Are Induced Differentially by Toll-like Receptor 7 Ligands in Human Blood Dendritic Cell Subsets. The Journal of Experimental Medicine. 195(11). 1507–1512. 360 indexed citations
13.
Ito, Tomoki�, Ryuichi Amakawa, Muneo Inaba, et al.. (2001). Differential Regulation of Human Blood Dendritic Cell Subsets by IFNs. The Journal of Immunology. 166(5). 2961–2969. 259 indexed citations
14.
Uehira, Kazutaka, Ryuichi Amakawa, Tomoki� Ito, et al.. (2001). A Hodgkin’s Disease Cell Line, KM-H2, Shows Biphenotypic Features of Dendritic Cells and B Cells. International Journal of Hematology. 73(2). 236–244. 14 indexed citations
15.
Ito, Tomoki�, Muneo Inaba, Kayo Inaba, et al.. (1999). A CD1a+/CD11c+ Subset of Human Blood Dendritic Cells Is a Direct Precursor of Langerhans Cells. The Journal of Immunology. 163(3). 1409–1419. 254 indexed citations
16.
Texidó, Gemma, Heinz Jacobs, Ralf Kühn, et al.. (1996). Somatic hypermutation occurs in B cells of terminal deoxynucleotidyl transferase‐, CD23‐, interleukin‐4‐, IgD‐ and CD30‐deficient mouse mutants. European Journal of Immunology. 26(8). 1966–1969. 15 indexed citations
17.
Amakawa, Ryuichi, Anne Hakem, Thomas M. Kündig, et al.. (1996). Impaired Negative Selection of T Cells in Hodgkin's Disease Antigen CD30–Deficient Mice. Cell. 84(4). 551–562. 266 indexed citations
18.
Amakawa, Ryuichi, Jing Wu, Kazuo Ozawa, et al.. (1993). Human Jk Recombination Signal Binding Protein Gene (IGKJRB): Comparison with Its Mouse Homologue. Genomics. 17(2). 306–315. 55 indexed citations
19.
Ohno, Hitoshi, Shirou Fukuhara, Ryuichi Amakawa, et al.. (1989). Involvement of c-myc oncogene in lymphoma cell lines with no detectable chromosome rearrangement of band 8q24. Cancer Genetics and Cytogenetics. 40(1). 73–82. 2 indexed citations
20.
Tanabe, Shin‐ichi, Doi S, Ikuma Kato, et al.. (1988). Treatment of extranodal lymphoma. 35(8). 199–206. 2 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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