Michiyuki Maeda

4.3k total citations · 2 hit papers
72 papers, 3.5k citations indexed

About

Michiyuki Maeda is a scholar working on Immunology, Ecology, Evolution, Behavior and Systematics and Molecular Biology. According to data from OpenAlex, Michiyuki Maeda has authored 72 papers receiving a total of 3.5k indexed citations (citations by other indexed papers that have themselves been cited), including 54 papers in Immunology, 21 papers in Ecology, Evolution, Behavior and Systematics and 18 papers in Molecular Biology. Recurrent topics in Michiyuki Maeda's work include T-cell and Retrovirus Studies (36 papers), Vector-Borne Animal Diseases (21 papers) and Animal Disease Management and Epidemiology (16 papers). Michiyuki Maeda is often cited by papers focused on T-cell and Retrovirus Studies (36 papers), Vector-Borne Animal Diseases (21 papers) and Animal Disease Management and Epidemiology (16 papers). Michiyuki Maeda collaborates with scholars based in Japan, United States and South Korea. Michiyuki Maeda's co-authors include Takashi Uchiyama, Hiroshi Fujiwara, Shingo Fujii, Junji Yodoi, Masao Matsuoka, Kisato Nosaka, Tasuku Honjo, Akira Shimizu, Shimon Sakaguchi and Shohei Hori and has published in prestigious journals such as Nature, Proceedings of the National Academy of Sciences and Journal of Biological Chemistry.

In The Last Decade

Michiyuki Maeda

72 papers receiving 3.4k citations

Hit Papers

Crucial role of FOXP3 in the development and ... 1984 2026 1998 2012 2004 1984 200 400 600

Peers

Michiyuki Maeda
Michiyuki Maeda
Citations per year, relative to Michiyuki Maeda Michiyuki Maeda (= 1×) peers Yves Lepelletier

Countries citing papers authored by Michiyuki Maeda

Since Specialization
Citations

This map shows the geographic impact of Michiyuki Maeda's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Michiyuki Maeda with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Michiyuki Maeda more than expected).

Fields of papers citing papers by Michiyuki Maeda

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Michiyuki Maeda. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Michiyuki Maeda. The network helps show where Michiyuki Maeda may publish in the future.

Co-authorship network of co-authors of Michiyuki Maeda

This figure shows the co-authorship network connecting the top 25 collaborators of Michiyuki Maeda. A scholar is included among the top collaborators of Michiyuki Maeda based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Michiyuki Maeda. Michiyuki Maeda is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Chiba, Masahiro, Takashi Ishio, Michiyuki Maeda, et al.. (2024). Genome-wide CRISPR screen identifies MAD2L1BP and ANAPC15 as targets for brentuximab vedotin sensitivity in CD30+ peripheral T-cell lymphoma. Leukemia. 39(1). 243–247. 1 indexed citations
2.
Chiba, Masahiro, Joji Shimono, Takashi Ishio, et al.. (2023). Whole-genome CRISPR screening identifies molecular mechanisms of PD-L1 expression in adult T-cell leukemia/lymphoma. Blood. 143(14). 1379–1390. 8 indexed citations
3.
Chiba, Masahiro, Joji Shimono, Takashi Ishio, et al.. (2022). Genome-wide CRISPR screens identify CD48 defining susceptibility to NK cytotoxicity in peripheral T-cell lymphomas. Blood. 140(18). 1951–1963. 21 indexed citations
4.
Maeda, Michiyuki, Akira Nagaoka, Kenji Araki, & Kensuke Nishioka. (2021). Practical and simplified measurements for representative photovoltaic array temperatures robust to climate variations. Solar Energy. 231. 243–251. 3 indexed citations
6.
Maeda, Michiyuki, et al.. (2005). Distinct IκB kinase regulation in adult T cell leukemia and HTLV-I-transformed cells. Experimental Cell Research. 308(1). 29–40. 8 indexed citations
7.
Fujiwara, Hiroshi, Toshihiro Higuchi, Yukiyasu Sato, et al.. (2005). Regulation of human extravillous trophoblast function by membrane-bound peptidases. Biochimica et Biophysica Acta (BBA) - Proteins and Proteomics. 1751(1). 26–32. 32 indexed citations
8.
Mori, Naoki, et al.. (2004). Tax-Independent Constitutive IκB Kinase Activation in Adult T-Cell Leukemia Cells. Neoplasia. 6(3). 266–278. 69 indexed citations
9.
Hori, Toshiyuki, et al.. (2002). OX40 Signaling Renders Adult T-Cell Leukemia Cells Resistant to Fas-Induced Apoptosis. International Journal of Hematology. 76(3). 260–266. 9 indexed citations
10.
Sato, Yukiyasu, Hiroshi Fujiwara, Toshihiro Higuchi, et al.. (2002). Involvement of Dipeptidyl Peptidase IV in Extravillous Trophoblast Invasion and Differentiation. The Journal of Clinical Endocrinology & Metabolism. 87(9). 4287–4296. 59 indexed citations
11.
Tsujino, Shiho, Tadaaki Miyazaki, Atsuo Kawahara, et al.. (1999). Critical role of the membrane‐proximal, proline‐rich motif of the interleukin‐2 receptor γc chain in the Jak3‐independent signal transduction. Genes to Cells. 4(6). 363–373. 12 indexed citations
12.
Fujiwara, Hiroshi, Tetsuro Honda, Masamichi Ueda, et al.. (1997). Laminin Suppresses Progesterone Production by Human Luteinizing Granulosa Cells via Interaction with Integrinα6β11. The Journal of Clinical Endocrinology & Metabolism. 82(7). 2122–2128. 41 indexed citations
13.
Fujiwara, Hiroshi, et al.. (1996). A Differentiation Antigen of Human Large Luteal Cells in Corpora Lutea of the Menstrual Cycle and Early Pregnancy1. Biology of Reproduction. 54(6). 1173–1183. 18 indexed citations
14.
Fujiwara, Hiroshi, Michiyuki Maeda, Tetsuro Honda, et al.. (1996). Granulosa Cells Express Integrin α6: Possible Involvement of Integrin α6 in Folliculogenesis. Hormone Research. 46(1). 24–30. 14 indexed citations
15.
Fujiwara, Hiroshi, et al.. (1995). A Porcine Homolog of Human Integrin α6 is a Differentiation Antigen of Granulosa Cells1. Biology of Reproduction. 53(2). 407–417. 16 indexed citations
16.
Imai, Kimitoshi, Michiyuki Maeda, Hiroshi Fujiwara, et al.. (1992). Human Endometrial Stromal Cells and Decidual Cells Express Cluster of Differentiation (CD) 13 Antigen/Aminopeptidase N and CD1O Antigen/Neutral Endopeptidase1. Biology of Reproduction. 46(3). 328–334. 66 indexed citations
17.
Umehara, Hisanori, et al.. (1988). Enhanced production of interleukin‐2 in patients with progressive systemic sclerosis. hyperactivity of cd4‐positive t cells?. Arthritis & Rheumatism. 31(3). 401–407. 44 indexed citations
18.
Nishimura, Masataka, Ichiro Akiguchi, Masahiro Takigawa, et al.. (1988). Human T cell lines established from the cerebrospinal fluid of patients with human T lymphotropic virus type I-associated myelopathy (HAM). Journal of Neuroimmunology. 17(3). 229–236. 10 indexed citations
19.
Nishimura, Masataka, Akio Adachi, Michiyuki Maeda, et al.. (1988). Analysis of the provirus genome integrated in T cell lines established from the cerebrospinal fluid of patients with human T lymphotropic virus type I-associated myelopathy (HAM). Journal of Neuroimmunology. 20(1). 33–37. 7 indexed citations
20.
Yodoi, Junji, Yutaka Tagaya, Masafumi OKADA, et al.. (1987). Interleukin-2 Receptor-Inducing Factor(s) in Adult T Cell Leukemia. Acta Haematologica. 78(1). 56–63. 3 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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