Robert P. Brendza

1.9k total citations
17 papers, 1.3k citations indexed

About

Robert P. Brendza is a scholar working on Physiology, Molecular Biology and Neurology. According to data from OpenAlex, Robert P. Brendza has authored 17 papers receiving a total of 1.3k indexed citations (citations by other indexed papers that have themselves been cited), including 10 papers in Physiology, 8 papers in Molecular Biology and 5 papers in Neurology. Recurrent topics in Robert P. Brendza's work include Alzheimer's disease research and treatments (10 papers), Computational Drug Discovery Methods (3 papers) and Microtubule and mitosis dynamics (3 papers). Robert P. Brendza is often cited by papers focused on Alzheimer's disease research and treatments (10 papers), Computational Drug Discovery Methods (3 papers) and Microtubule and mitosis dynamics (3 papers). Robert P. Brendza collaborates with scholars based in United States, France and United Kingdom. Robert P. Brendza's co-authors include David M. Holtzman, William M. Saxton, Laura R. Serbus, Joseph B. Duffy, John R. Cirrito, John Denis Fryer, Steven M. Paul, Sheng‐Kwei Song, Joong Hee Kim and Malca Kierson and has published in prestigious journals such as Science, Journal of Clinical Investigation and Nature Medicine.

In The Last Decade

Robert P. Brendza

17 papers receiving 1.3k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Robert P. Brendza United States	14	661	494	239	200	194	17	1.3k
Tatsuhide Kunishita Japan	21	769 1.2×	677 1.4×	125 0.5×	230 1.1×	296 1.5×	50	1.9k
Gaku Sakaguchi Japan	25	869 1.3×	620 1.3×	443 1.9×	169 0.8×	439 2.3×	51	1.6k
Jon R. Backstrom United States	14	659 1.0×	446 0.9×	79 0.3×	108 0.5×	341 1.8×	20	1.3k
An Snellinx Belgium	16	1.1k 1.7×	776 1.6×	212 0.9×	298 1.5×	465 2.4×	24	2.0k
Agnieszka Krzyzanowska Sweden	19	485 0.7×	265 0.5×	105 0.4×	71 0.4×	331 1.7×	31	1.1k
Kristina R. Patterson United States	12	531 0.8×	769 1.6×	156 0.7×	228 1.1×	394 2.0×	19	1.5k
Wang Zheng China	21	721 1.1×	122 0.2×	216 0.9×	97 0.5×	343 1.8×	43	1.5k
Kazuhiko Tagawa Japan	25	1.4k 2.2×	385 0.8×	493 2.1×	134 0.7×	644 3.3×	52	2.0k
A. Défossez France	18	518 0.8×	713 1.4×	115 0.5×	222 1.1×	169 0.9×	49	1.3k
Anne Philippi France	20	1000 1.5×	248 0.5×	116 0.5×	73 0.4×	175 0.9×	33	1.9k

Countries citing papers authored by Robert P. Brendza

Since Specialization

Citations

This map shows the geographic impact of Robert P. Brendza's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Robert P. Brendza with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Robert P. Brendza more than expected).

Fields of papers citing papers by Robert P. Brendza

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Robert P. Brendza. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Robert P. Brendza. The network helps show where Robert P. Brendza may publish in the future.

Co-authorship network of co-authors of Robert P. Brendza

This figure shows the co-authorship network connecting the top 25 collaborators of Robert P. Brendza. A scholar is included among the top collaborators of Robert P. Brendza based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Robert P. Brendza. Robert P. Brendza is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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17 of 17 papers shown

1.

Brendza, Robert P., Xiaoying Gao, Kimberly L. Stark, et al.. (2022). Anti-α-synuclein c-terminal antibodies block PFF uptake and accumulation of phospho-synuclein in preclinical models of Parkinson's disease. Neurobiology of Disease. 177. 105969–105969. 7 indexed citations

2.

Brendza, Robert P., Han Lin, Kimberly L. Stark, et al.. (2021). Genetic ablation of Gpnmb does not alter synuclein-related pathology. Neurobiology of Disease. 159. 105494–105494. 9 indexed citations

3.

Domínguez, Sara L., Eugene Varfolomeev, Robert P. Brendza, et al.. (2020). Genetic inactivation of RIP1 kinase does not ameliorate disease in a mouse model of ALS. Cell Death and Differentiation. 28(3). 915–931. 24 indexed citations

4.

Xie, Luke, Γεώργιος Κούκος, Kai Barck, et al.. (2018). Micro-CT imaging and structural analysis of glomeruli in a model of Adriamycin-induced nephropathy. American Journal of Physiology-Renal Physiology. 316(1). F76–F89. 23 indexed citations

5.

Han, Byung Hee, Meng‐Liang Zhou, Robert P. Brendza, et al.. (2008). Cerebrovascular Dysfunction in Amyloid Precursor Protein Transgenic Mice: Contribution of Soluble and Insoluble Amyloid-β Peptide, Partial Restoration via γ-Secretase Inhibition. Journal of Neuroscience. 28(50). 13542–13550. 111 indexed citations

6.

Brendza, Robert P. & David M. Holtzman. (2006). Amyloid-β Immunotherapies in Mice and Men. Alzheimer Disease & Associated Disorders. 20(2). 118–123. 13 indexed citations

7.

Brendza, Robert P., Brian J. Bacskai, John R. Cirrito, et al.. (2005). Anti-Aβ antibody treatment promotes the rapid recovery of amyloid-associated neuritic dystrophy in PDAPP transgenic mice. Journal of Clinical Investigation. 115(2). 428–433. 23 indexed citations

8.

Brendza, Robert P., Brian J. Bacskai, John R. Cirrito, et al.. (2005). Anti-Aβ antibody treatment promotes the rapid recovery of amyloid-associated neuritic dystrophy in PDAPP transgenic mice. Journal of Clinical Investigation. 115(2). 428–433. 160 indexed citations

9.

Song, Sheng‐Kwei, et al.. (2004). Diffusion tensor imaging detects age-dependent white matter changes in a transgenic mouse model with amyloid deposition. Neurobiology of Disease. 15(3). 640–647. 141 indexed citations

10.

Brendza, Robert P.. (2003). Use of YFP to study amyloid-β associated neurite alterations in live brain slices. Neurobiology of Aging. 24(8). 1071–1077. 19 indexed citations

11.

Brendza, Robert P., Kelly Simmons, Daniel W. McKeel, et al.. (2003). PDAPP; YFP double transgenic mice: A tool to study amyloid‐β associated changes in axonal, dendritic, and synaptic structures. The Journal of Comparative Neurology. 456(4). 375–383. 52 indexed citations

12.

Brendza, Robert P., Kelly R. Bales, Steven M. Paul, & David M. Holtzman. (2002). Role of apoE/Aβ interactions in Alzheimer's disease: insights from transgenic mouse models. Molecular Psychiatry. 7(2). 132–135. 24 indexed citations

13.

Brendza, Robert P., Laura R. Serbus, William M. Saxton, & Joseph B. Duffy. (2002). Posterior Localization of Dynein and Dorsal-Ventral Axis Formation Depend on Kinesin in Drosophila Oocytes. Current Biology. 12(17). 1541–1545. 79 indexed citations

14.

DeMattos, Ronald B., Robert P. Brendza, John E. Heuser, et al.. (2001). Purification and characterization of astrocyte-secreted apolipoprotein E and J-containing lipoproteins from wild-type and human apoE transgenic mice. Neurochemistry International. 39(5-6). 415–425. 137 indexed citations

15.

Han, Byung Hee, Ronald B. DeMattos, Laura L. Dugan, et al.. (2001). Clusterin contributes to caspase-3–independent brain injury following neonatal hypoxia-ischemia. Nature Medicine. 7(3). 338–343. 177 indexed citations

16.

Brendza, Robert P., Laura R. Serbus, Joseph B. Duffy, & William M. Saxton. (2000). A Function for Kinesin I in the Posterior Transport of oskar mRNA and Staufen Protein. Science. 289(5487). 2120–2122. 298 indexed citations

17.

Brendza, Robert P., Kathy B. Sheehan, F. Rudolf Turner, & William M. Saxton. (2000). Clonal Tests of Conventional Kinesin Function during Cell Proliferation and Differentiation. Molecular Biology of the Cell. 11(4). 1329–1343. 21 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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