Mirjam T. Epping

2.3k total citations
16 papers, 1.7k citations indexed

About

Mirjam T. Epping is a scholar working on Molecular Biology, Immunology and Oncology. According to data from OpenAlex, Mirjam T. Epping has authored 16 papers receiving a total of 1.7k indexed citations (citations by other indexed papers that have themselves been cited), including 14 papers in Molecular Biology, 6 papers in Immunology and 5 papers in Oncology. Recurrent topics in Mirjam T. Epping's work include Retinoids in leukemia and cellular processes (7 papers), Histone Deacetylase Inhibitors Research (5 papers) and Ubiquitin and proteasome pathways (4 papers). Mirjam T. Epping is often cited by papers focused on Retinoids in leukemia and cellular processes (7 papers), Histone Deacetylase Inhibitors Research (5 papers) and Ubiquitin and proteasome pathways (4 papers). Mirjam T. Epping collaborates with scholars based in Netherlands, United States and United Kingdom. Mirjam T. Epping's co-authors include René Bernards, Liming Wang, Jonathan R. Pollack, Patrick O. Brown, Jennifer Willert, Roel Nusse, Michael J. Edel, María Inmaculada Hernández, Giuseppe Giaccone and Carlos Gil Ferreira and has published in prestigious journals such as Cell, Proceedings of the National Academy of Sciences and Blood.

In The Last Decade

Mirjam T. Epping

16 papers receiving 1.7k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Mirjam T. Epping Netherlands	15	1.3k	453	383	219	168	16	1.7k
Nilgun Tasdemir United States	15	1.1k 0.9×	429 0.9×	183 0.5×	168 0.8×	349 2.1×	26	1.6k
Jason A. Powell Australia	24	1.0k 0.8×	318 0.7×	340 0.9×	114 0.5×	169 1.0×	42	1.6k
David Sekula United States	18	850 0.7×	440 1.0×	213 0.6×	127 0.6×	163 1.0×	25	1.1k
Scott Wise United States	17	660 0.5×	293 0.6×	195 0.5×	157 0.7×	161 1.0×	43	1.2k
Céline Charvet France	18	1.0k 0.8×	443 1.0×	608 1.6×	74 0.3×	167 1.0×	26	1.7k
Mikio Tomida Japan	24	929 0.7×	685 1.5×	581 1.5×	175 0.8×	132 0.8×	62	1.8k
Francesco Trapasso Italy	26	1.5k 1.2×	375 0.8×	344 0.9×	330 1.5×	312 1.9×	74	2.0k
Melanie Braig Germany	15	1.3k 1.0×	450 1.0×	258 0.7×	74 0.3×	197 1.2×	22	1.8k
Franz Kohlhuber Germany	15	1.1k 0.9×	825 1.8×	357 0.9×	90 0.4×	281 1.7×	17	1.8k
Brian C. Grabiner United States	11	1.1k 0.9×	441 1.0×	422 1.1×	136 0.6×	472 2.8×	11	1.8k

Countries citing papers authored by Mirjam T. Epping

Since Specialization

Citations

This map shows the geographic impact of Mirjam T. Epping's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mirjam T. Epping with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mirjam T. Epping more than expected).

Fields of papers citing papers by Mirjam T. Epping

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Mirjam T. Epping. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mirjam T. Epping. The network helps show where Mirjam T. Epping may publish in the future.

Co-authorship network of co-authors of Mirjam T. Epping

This figure shows the co-authorship network connecting the top 25 collaborators of Mirjam T. Epping. A scholar is included among the top collaborators of Mirjam T. Epping based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Mirjam T. Epping. Mirjam T. Epping is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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16 of 16 papers shown

Yin, Dean R.P.P. Chan Pin, Mirjam T. Epping, René Boomars, et al.. (2024). Pre-hospital evaluation of chest pain patients using the modified HEART-score: rationale and design. Future Cardiology. 20(5-6). 241–250. 1 indexed citations

Epping, Mirjam T., Andrea Lunardi, Daphna Nachmani, et al.. (2015). TSPYL2 is an essential component of the REST/NRSF transcriptional complex for TGFβ signaling activation. Cell Death and Differentiation. 22(8). 1353–1362. 35 indexed citations

González‐Billalabeitia, Enrique, Nina Seitzer, Su Jung Song, et al.. (2014). Vulnerabilities of PTEN – TP53 -Deficient Prostate Cancers to Compound PARP–PI3K Inhibition. Cancer Discovery. 4(8). 896–904. 80 indexed citations

Dummer, Reinhard, Marc Beyer, Kenneth B. Hymes, et al.. (2012). Vorinostat combined with bexarotene for treatment of cutaneous T-cell lymphoma:in vitroand phase I clinical evidence supporting augmentation of retinoic acid receptor/retinoid X receptor activation by histone deacetylase inhibition. Leukemia & lymphoma. 53(8). 1501–1508. 51 indexed citations

Epping, Mirjam T., Lars A.T. Meijer, Oscar Krijgsman, et al.. (2010). TSPYL5 suppresses p53 levels and function by physical interaction with USP7. Nature Cell Biology. 13(1). 102–108. 103 indexed citations

Epping, Mirjam T., Lars A.T. Meijer, Johannes L. Bos, & René Bernards. (2009). UNC45A Confers Resistance to Histone Deacetylase Inhibitors and Retinoic Acid. Molecular Cancer Research. 7(11). 1861–1870. 22 indexed citations

Huang, Sidong, Jamila Laoukili, Mirjam T. Epping, et al.. (2009). ZNF423 Is Critically Required for Retinoic Acid-Induced Differentiation and Is a Marker of Neuroblastoma Outcome. Cancer Cell. 15(4). 328–340. 112 indexed citations

Fotheringham, Susan, Mirjam T. Epping, Lindsay Stimson, et al.. (2009). Genome-wide Loss-of-Function Screen Reveals an Important Role for the Proteasome in HDAC Inhibitor-Induced Apoptosis. Cancer Cell. 15(1). 57–66. 102 indexed citations

Oehler, Vivian G., Katherine A. Guthrie, Kathleen M. Sabo, et al.. (2009). The preferentially expressed antigen in melanoma (PRAME) inhibits myeloid differentiation in normal hematopoietic and leukemic progenitor cells. Blood. 114(15). 3299–3308. 65 indexed citations

10.

Epping, Mirjam T. & René Bernards. (2008). Molecular basis of the anti-cancer effects of histone deacetylase inhibitors. The International Journal of Biochemistry & Cell Biology. 41(1). 16–20. 15 indexed citations

11.

Epping, Mirjam T., A.A.M. Hart, Annuska M. Glas, Oscar Krijgsman, & René Bernards. (2008). PRAME expression and clinical outcome of breast cancer. British Journal of Cancer. 99(3). 398–403. 91 indexed citations

12.

Epping, Mirjam T., Liming Wang, Jane A. Plumb, et al.. (2007). A functional genetic screen identifies retinoic acid signaling as a target of histone deacetylase inhibitors. Proceedings of the National Academy of Sciences. 104(45). 17777–17782. 65 indexed citations

13.

Epping, Mirjam T. & René Bernards. (2006). A Causal Role for the Human Tumor Antigen Preferentially Expressed Antigen of Melanoma in Cancer. Cancer Research. 66(22). 10639–10642. 78 indexed citations

14.

Epping, Mirjam T., et al.. (2005). The Human Tumor Antigen PRAME Is a Dominant Repressor of Retinoic Acid Receptor Signaling. Cell. 122(6). 835–847. 348 indexed citations

15.

Willert, Jennifer, Mirjam T. Epping, Jonathan R. Pollack, Patrick O. Brown, & Roel Nusse. (2002). A transcriptional response to Wnt protein in human embryonic carcinoma cells. BMC Developmental Biology. 2(1). 8–8. 335 indexed citations

16.

Ferreira, Carlos Gil, Mirjam T. Epping, Frank A.E. Kruyt, & Giuseppe Giaccone. (2002). Apoptosis: target of cancer therapy.. PubMed. 8(7). 2024–34. 220 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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