Frank A.E. Kruyt
- Molecular Biology top 1%
- Oncology top 1%
- Cancer Research top 0.5%
- Immunology top 5%
- Pulmonary and Respiratory Medicine top 5%
- Co-authors
- Giuseppe GiacconeLinda E. BrökerJosé Antonio RodríguezSimone W. SpanCarlos Gil FerreiraGodefridus J. PetersJustin V. JosephMaarten L. Janmaat
- Topics
- Cell death mechanisms and regulation (45 papers)DNA Repair Mechanisms (21 papers)Cancer-related Molecular Pathways (20 papers)
- Partner nations
- NetherlandsUnited StatesJapan
In The Last Decade
Frank A.E. Kruyt
137 papers receiving 7.8k citations
Hit Papers
Peers
Comparison fields: 5 of 136
- Molecular Biology 5.7k
- Oncology 2.3k
- Cancer Research 1.7k
- Immunology 888
- Pulmonary and Respiratory Medicine 826
Countries citing papers authored by Frank A.E. Kruyt
This map shows the geographic impact of Frank A.E. Kruyt's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Frank A.E. Kruyt with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Frank A.E. Kruyt more than expected).
Fields of papers citing papers by Frank A.E. Kruyt
This network shows the impact of papers produced by Frank A.E. Kruyt. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Frank A.E. Kruyt. The network helps show where Frank A.E. Kruyt may publish in the future.
Co-authorship network of co-authors of Frank A.E. Kruyt
This figure shows the co-authorship network connecting the top 25 collaborators of Frank A.E. Kruyt. A scholar is included among the top collaborators of Frank A.E. Kruyt based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Frank A.E. Kruyt. Frank A.E. Kruyt is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 0 | |
| 2 | 32 | |
| 3 | 0 | |
| 4 | 7 | |
| 5 | 17 | |
| 6 | 10 | |
| 7 | 55 | |
| 8 | 139 | |
| 9 | 59 | |
| 10 | 134 | |
| 11 | 63 | |
| 12 | 12 | |
| 13 | 62 | |
| 14 | 14 | |
| 15 | 36 | |
| 16 | 62 | |
| 17 | 49 | |
| 18 | Regulated binding of the Fanconi anemia proteins, FANCA and FANCC - Response | 1 |
| 19 | 10 | |
| 20 | 40 |
About Frank A.E. Kruyt
Frank A.E. Kruyt is a scholar working on Cancer Research, Oncology and Molecular Biology, having authored 140 papers that have together received 7.9k indexed citations. Recurring topics across this work include Cell death mechanisms and regulation (45 papers), DNA Repair Mechanisms (21 papers) and Cancer-related Molecular Pathways (20 papers). The work is most often cited by research in Cancer Research (1.7k citations), Oncology (2.3k citations) and Molecular Biology (5.7k citations). Frank A.E. Kruyt has collaborated with scholars based in Netherlands, United States and Japan. Frequent co-authors include Giuseppe Giaccone, Linda E. Bröker, José Antonio Rodríguez, Simone W. Span, Carlos Gil Ferreira, Godefridus J. Peters, Justin V. Joseph, Maarten L. Janmaat, Steven de Jong and Hagop Youssoufian. Their work appears in journals such as Proceedings of the National Academy of Sciences, Nucleic Acids Research and Advanced Materials.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.