Matthew J. Brody

3.1k total citations · 1 hit paper
52 papers, 2.3k citations indexed

About

Matthew J. Brody is a scholar working on Molecular Biology, Cardiology and Cardiovascular Medicine and Physiology. According to data from OpenAlex, Matthew J. Brody has authored 52 papers receiving a total of 2.3k indexed citations (citations by other indexed papers that have themselves been cited), including 20 papers in Molecular Biology, 19 papers in Cardiology and Cardiovascular Medicine and 9 papers in Physiology. Recurrent topics in Matthew J. Brody's work include Cardiac Fibrosis and Remodeling (9 papers), Protein Kinase Regulation and GTPase Signaling (5 papers) and Cardiomyopathy and Myosin Studies (4 papers). Matthew J. Brody is often cited by papers focused on Cardiac Fibrosis and Remodeling (9 papers), Protein Kinase Regulation and GTPase Signaling (5 papers) and Cardiomyopathy and Myosin Studies (4 papers). Matthew J. Brody collaborates with scholars based in United States, Australia and Philippines. Matthew J. Brody's co-authors include Jeffery D. Molkentin, Donald D. Heistad, Onur Kanisicak, Jason Karch, Bryan D. Maliken, Robert N. Correll, M. N. Hart, Hadi Khalil, P. J. Kadowitz and Suh‐Chin J. Lin and has published in prestigious journals such as JAMA, Journal of Biological Chemistry and Circulation.

In The Last Decade

Matthew J. Brody

48 papers receiving 2.2k citations

Hit Papers

Genetic lineage tracing d... 2016 2026 2019 2022 2016 200 400 600

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Matthew J. Brody United States 26 1.0k 979 398 396 194 52 2.3k
Andrea Lippoldt Germany 29 1.2k 1.2× 863 0.9× 326 0.8× 339 0.9× 162 0.8× 54 3.1k
Nobutaka Koibuchi Japan 31 957 0.9× 695 0.7× 365 0.9× 406 1.0× 85 0.4× 58 2.5k
Yasutomo Fukunaga Japan 24 969 0.9× 512 0.5× 615 1.5× 292 0.7× 131 0.7× 36 2.0k
Heinz‐Gerd Zimmer Germany 30 1.1k 1.0× 1.5k 1.5× 372 0.9× 390 1.0× 85 0.4× 135 2.8k
Jens Fielitz Germany 30 1.9k 1.9× 795 0.8× 484 1.2× 349 0.9× 260 1.3× 68 3.1k
Jo El J. Schultz United States 26 1.6k 1.5× 1.3k 1.4× 261 0.7× 433 1.1× 139 0.7× 46 3.4k
Scott P. Heximer Canada 33 2.0k 2.0× 898 0.9× 452 1.1× 285 0.7× 261 1.3× 80 3.5k
Renhui Yang United States 28 1.3k 1.3× 968 1.0× 535 1.3× 319 0.8× 95 0.5× 64 2.9k
Michelle L. Nieman United States 25 2.0k 2.0× 1.2k 1.3× 267 0.7× 353 0.9× 332 1.7× 44 3.1k
Peter Razeghi United States 29 1.7k 1.6× 1.5k 1.6× 932 2.3× 504 1.3× 162 0.8× 44 3.4k

Countries citing papers authored by Matthew J. Brody

Since Specialization
Citations

This map shows the geographic impact of Matthew J. Brody's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Matthew J. Brody with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Matthew J. Brody more than expected).

Fields of papers citing papers by Matthew J. Brody

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Matthew J. Brody. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Matthew J. Brody. The network helps show where Matthew J. Brody may publish in the future.

Co-authorship network of co-authors of Matthew J. Brody

This figure shows the co-authorship network connecting the top 25 collaborators of Matthew J. Brody. A scholar is included among the top collaborators of Matthew J. Brody based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Matthew J. Brody. Matthew J. Brody is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
2.
Essandoh, Kobina, et al.. (2025). Cardiomyocyte Janus kinase 1 (JAK1) signaling is required for cardiac homeostasis and cytokine-dependent activation of STAT3. Journal of Molecular and Cellular Cardiology. 207. 13–23. 1 indexed citations
3.
Goldsmith, Robert, et al.. (2024). In Vitro Assessment of Cardiac Fibroblast Activation at Physiologic Stiffness. Current Protocols. 4(4). e1025–e1025.
4.
Essandoh, Kobina, et al.. (2024). Regulation of cardiomyocyte intracellular trafficking and signal transduction by protein palmitoylation. Biochemical Society Transactions. 52(1). 41–53. 8 indexed citations
5.
Baldwin, Tanya A., Yasuhide Kuwabara, Suh‐Chin J. Lin, et al.. (2023). Palmitoylation-dependent regulation of cardiomyocyte Rac1 signaling activity and minor effects on cardiac hypertrophy. Journal of Biological Chemistry. 299(12). 105426–105426. 15 indexed citations
6.
Essandoh, Kobina, et al.. (2023). zDHHC9 Regulates Cardiomyocyte Rab3a Activity and Atrial Natriuretic Peptide Secretion Through Palmitoylation of Rab3gap1. JACC Basic to Translational Science. 8(5). 518–542. 15 indexed citations
7.
Essandoh, Kobina, et al.. (2022). NADPH Oxidases in Diastolic Dysfunction and Heart Failure with Preserved Ejection Fraction. Antioxidants. 11(9). 1822–1822. 34 indexed citations
8.
Guo, Angela H., Mary E. Skinner, Surinder Kumar, et al.. (2022). Sirtuin 5 levels are limiting in preserving cardiac function and suppressing fibrosis in response to pressure overload. Scientific Reports. 12(1). 12258–12258. 19 indexed citations
9.
Vanhoutte, Davy, Tobias G. Schips, Kelly M. Grimes, et al.. (2021). Thbs1 induces lethal cardiac atrophy through PERK-ATF4 regulated autophagy. Nature Communications. 12(1). 3928–3928. 109 indexed citations
10.
Schips, Tobias G., Davy Vanhoutte, Robert N. Correll, et al.. (2019). Thrombospondin-3 augments injury-induced cardiomyopathy by intracellular integrin inhibition and sarcolemmal instability. Nature Communications. 10(1). 76–76. 42 indexed citations
11.
Karch, Jason, Matthew J. Brody, Onur Kanisicak, et al.. (2017). Abstract 205: Overexpression of the Mitochondrial F o F 1 ATP Synthase regulators ATP12 and ATP5J are Protective against Ischemia-Reperfusion Injury in the Heart. Circulation Research. 121(suppl_1). 1 indexed citations
12.
Kanisicak, Onur, Hadi Khalil, Malina J. Ivey, et al.. (2016). Genetic lineage tracing defines myofibroblast origin and function in the injured heart. Nature Communications. 7(1). 12260–12260. 640 indexed citations breakdown →
13.
Brody, Matthew J., Adrian C. Grimes, Timothy A. Hacker, et al.. (2015). LRRC10 is required to maintain cardiac function in response to pressure overload. American Journal of Physiology-Heart and Circulatory Physiology. 310(2). H269–H278. 22 indexed citations
14.
Hacker, Timothy A., George R. Flentke, Victoria Drake, et al.. (2009). Altered Cardiac Function and Ventricular Septal Defect in Avian Embryos Exposed to Low-Dose Trichloroethylene. Toxicological Sciences. 113(2). 444–452. 34 indexed citations
15.
Vasquez, Elisardo C., Simon J.G. Lewis, Kurt J. Varner, & Matthew J. Brody. (1991). Lesions of the rostral ventrolateral rvlm but not rostral ventromedial medulla rvmm attenuate 5 ht induced reflex tachycardia. The FASEB Journal. 5(4). 743. 7 indexed citations
16.
Brody, Matthew J.. (1983). American Assassins: The Darker Side of Politics. JAMA. 249(12). 1641–1641. 1 indexed citations
17.
DiBona, Gerald F., et al.. (1977). Arterial Pressure Development in Neonatal and Young Spontaneously Hypertensive Rats. Journal of Vascular Research. 14(5). 277–284. 72 indexed citations
18.
Synhorst, David, Romy Lauer, Donald B. Doty, & Matthew J. Brody. (1976). Hemodynamic effects of vasodilator agents in dogs with experimental ventricular septal defects.. Circulation. 54(3). 472–477. 31 indexed citations
19.
Brody, Matthew J. & P. J. Kadowitz. (1974). Prostaglandins as modulators of the autonomic nervous system.. PubMed. 33(1). 48–60. 125 indexed citations
20.
Brody, Matthew J.. (1952). A clinical evaluation of the use of decamethonium bromide (C10) in convulsive shock therapy. Psychiatric Quarterly. 26(1-4). 205–211.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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